Epstein-Barr virus DNase (BGLF5) induces genomic instability in human epithelial cells. - Wikidata - awgldk - TriplyDB

Epstein-Barr virus DNase (BGLF5) induces genomic instability in human epithelial cells.

Epstein-Barr virus DNase (BGLF5) induces genomic instability in human epithelial cells.

http://www.wikidata.org/entity/Q33760239

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Understanding the interplay between host immunity and Epstein-Barr virus in NPC patients Modulation of DNA damage and repair pathways by human tumour viruses Roles of the PI3K/Akt pathway in Epstein-Barr virus-induced cancers and therapeutic implications Crystal structure of a KSHV-SOX-DNA complex: insights into the molecular mechanisms underlying DNase activity and host shutoff Epstein-Barr virus lytic reactivation regulation and its pathogenic role in carcinogenesis The synergistic effect of chemical carcinogens enhances Epstein-Barr virus reactivation and tumor progression of nasopharyngeal carcinoma cells Transformation of the genital epithelial tract occurs early in California sea lion development Chromosomal variation in lymphoblastoid cell lines Epstein-Barr virus BALF3 mediates genomic instability and progressive malignancy in nasopharyngeal carcinoma.DNA Damage Signaling Is Induced in the Absence of Epstein-Barr Virus (EBV) Lytic DNA Replication and in Response to Expression of ZEBRA.Necrosis is increased in lymphoblastoid cell lines from children with autism compared with their non-autistic siblings under conditions of oxidative and nitrosative stress EBV reactivation as a target of luteolin to repress NPC tumorigenesis.Altered expression of the mismatch repair genes in DF-1 cells infected with the avian leukosis virus subgroup A.Inhibition of Epstein-Barr virus reactivation by the flavonoid apigenin.Effects of parental radiation exposure on developmental instability in grasshoppers.How virus persistence can initiate the tumorigenesis process.Switching of EBV cycles between latent and lytic states.Modes of infection and oncogenesis by the Epstein-Barr virus.Regulation of Epstein-Barr virus reactivation from latency.The role of Epstein-Barr virus infection in the pathogenesis of nasopharyngeal carcinoma.Epstein-Barr virus: more than 50 years old and still providing surprises.The role of oxidative stress in EBV lytic reactivation, radioresistance and the potential preventive and therapeutic implications.Epstein-Barr virus infection and nasopharyngeal carcinoma.Programmed Death-Ligand 1 Expression, Microsatellite Instability, Epstein-Barr Virus, and Human Papillomavirus in Nasopharyngeal Carcinomas of Patients from the Philippines.Induction of DNA damages upon Marek's disease virus infection: implication in viral replication and pathogenesis.Inhibition of Epstein-Barr virus reactivation in nasopharyngeal carcinoma cells by dietary sulforaphane.Epstein-Barr virus BRLF1 induces genomic instability and progressive malignancy in nasopharyngeal carcinoma cells.Pyruvate kinase M2 phosphorylates H2AX and promotes genomic instability in human tumor cells.Epstein-Barr Virus as a Promising Immunotherapeutic Target for Nasopharyngeal Carcinoma Treatment.Perspective: Contribution of Epstein-Barr virus (EBV) Reactivation to the Carcinogenicity of Nasopharyngeal Cancer Cells.The Immunomodulatory Capacity of an Epstein-Barr Virus Abortive Lytic Cycle: Potential Contribution to Viral Tumorigenesis.Therapies based on targeting Epstein-Barr virus lytic replication for EBV-associated malignancies.Integrated genomic analyses in PDX model reveal a cyclin-dependent kinase inhibitor Palbociclib as a novel candidate drug for nasopharyngeal carcinoma

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Epstein-Barr virus DNase (BGLF5) induces genomic instability in human epithelial cells.

http://www.wikidata.org/entity/Q33760239

description

2009 nî lūn-bûn

@nan

2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年学术文章

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2009年学术文章

@zh-cn

2009年学术文章

@zh-hans

2009年学术文章

@zh-my

2009年学术文章

@zh-sg

2009年學術文章

@yue

name

Epstein-Barr virus DNase (BGLF5) induces genomic instability in human epithelial cells.

@ast

Epstein-Barr virus DNase (BGLF5) induces genomic instability in human epithelial cells.

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type

label

Epstein-Barr virus DNase (BGLF5) induces genomic instability in human epithelial cells.

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Epstein-Barr virus DNase (BGLF5) induces genomic instability in human epithelial cells.

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prefLabel

Epstein-Barr virus DNase (BGLF5) induces genomic instability in human epithelial cells.

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Epstein-Barr virus DNase (BGLF5) induces genomic instability in human epithelial cells.

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Nucleic Acids Research

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Epstein-Barr virus DNase (BGLF5) induces genomic instability in human epithelial cells.

@en

P2093

Chia-Huei Lee

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Chih-Yeu Fang

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Chung-Chun Wu

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Hsin-Wei Liao

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Hsin-Ying Chuang

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Jen-Yang Chen

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Kuan-Lin Kuo

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Ming-Tsan Liu

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Pei-Wen Wang

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Sheng-Ping Chou

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P2860

The Hallmarks of Cancer

ATR kinase activation mediated by MutSalpha and MutLalpha in response to cytotoxic O6-methylguanine adducts

The role of chromosomal instability in tumor initiation.

Overexpression of human RAD51 and RAD52 reduces double-strand break-induced homologous recombination in mammalian cells

Hepatitis B virus X protein interacts with a probable cellular DNA repair protein

Human AP endonuclease suppresses DNA mismatch repair activity leading to microsatellite instability.

Epstein-Barr virus provides a new paradigm: a requirement for the immediate inhibition of apoptosis.

DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139

Genetic instabilities in human cancers

Expression in normal human tissues of five nucleotide excision repair genes measured simultaneously by multiplex reverse transcription-polymerase chain reaction

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1932-1949

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scholarly article

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10.1093/NAR/GKP1169

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6

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38

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2009-12-23T00:00:00Z

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40774095

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20034954

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Epstein–Barr virus

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2847232

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