TGF-β1 as possible link between loss of bone mineral density and chronic inflammation. - Wikidata - awgldk - TriplyDB

TGF-β1 as possible link between loss of bone mineral density and chronic inflammation.

TGF-β1 as possible link between loss of bone mineral density and chronic inflammation.

http://www.wikidata.org/entity/Q33761160

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Modeling hepatic osteodystrophy in Abcb4 deficient mice A novel library screen identifies immunosuppressors that promote osteoblast differentiation.Transforming growth factor β1 inhibits bone morphogenic protein (BMP)-2 and BMP-7 signaling via upregulation of Ski-related novel protein N (SnoN): possible mechanism for the failure of BMP therapy?5-azacytidine improves the osteogenic differentiation potential of aged human adipose-derived mesenchymal stem cells by DNA demethylation Transforming growth factor-β impairs glucocorticoid activity in the A549 lung adenocarcinoma cell line.Fluoride Regulate Osteoblastic Transforming Growth Factor-β1 Signaling by Mediating Recycling of the Type I Receptor ALK5 Congenic mice provide in vivo evidence for a genetic locus that modulates intrinsic transforming growth factor β1-mediated signaling and bone acquisition.Liraglutide attenuates the osteoblastic differentiation of MC3T3‑E1 cells by modulating AMPK/mTOR signaling Multiple analyses of large-scale genome-wide association study highlight new risk pathways in lumbar spine bone mineral density.Interleukin-10 inhibits bone resorption: a potential therapeutic strategy in periodontitis and other bone loss diseases.Comparative immunoexpression of ICAM-1, TGF-β1 and ki-67 in periapical and residual cysts.Activation of peroxisome proliferator activated receptor alpha ameliorates ethanol mediated liver fibrosis in mice.BMP9 a possible alternative drug for the recently withdrawn BMP7? New perspectives for (re-)implementation by personalized medicine.Primary human osteoblasts with reduced alkaline phosphatase and matrix mineralization baseline capacity are responsive to extremely low frequency pulsed electromagnetic field exposure - Clinical implication possible.The Effect of Exogenous Zinc Concentration on the Responsiveness of MC3T3-E1 Pre-Osteoblasts to Surface Microtopography: Part II (Differentiation)Effects of TGF-β1 on mineralization mediated by rat calvaria-derived osteogenic cells.Chronic CCl4 intoxication causes liver and bone damage similar to the human pathology of hepatic osteodystrophy: a mouse model to analyse the liver-bone axis.In vitro and in vivo evaluation of the marine sponge skeleton as a bone mimicking biomaterial.Green tea protects human osteoblasts from cigarette smoke-induced injury: possible clinical implication.Hyperactive transforming growth factor-β1 signaling potentiates skeletal defects in a neurofibromatosis type 1 mouse model.Men and Women Differ in the Biochemical Composition of Platelet-Rich Plasma.Characterisation of porous knitted titanium for replacement of intervertebral disc nucleus pulposus.Resveratrol protects primary cilia integrity of human mesenchymal stem cells from cigarette smoke to improve osteogenic differentiation in vitro.TGF-β1 impairs mechanosensation of human osteoblasts via HDAC6-mediated shortening and distortion of primary cilia.Human amniotic epithelial cells regulate osteoblast differentiation through the secretion of TGFβ1 and microRNA-34a-5p.Quercetin protects primary human osteoblasts exposed to cigarette smoke through activation of the antioxidative enzymes HO-1 and SOD-1 The Cocktail Effect of BMP-2 and TGF-β1 Loaded in Visible Light-Cured Glycol Chitosan Hydrogels for the Enhancement of Bone Formation in a Rat Tibial Defect Model Huangqi (astragalus) decoction ameliorates diabetic nephropathy via IRS1-PI3K-GLUT signaling pathway

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TGF-β1 as possible link between loss of bone mineral density and chronic inflammation.

http://www.wikidata.org/entity/Q33761160

description

2010 nî lūn-bûn

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2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

TGF-β1 as possible link between loss of bone mineral density and chronic inflammation.

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TGF-β1 as possible link between loss of bone mineral density and chronic inflammation.

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TGF-β1 as possible link between loss of bone mineral density and chronic inflammation.

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TGF-β1 as possible link between loss of bone mineral density and chronic inflammation.

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TGF-β1 as possible link between loss of bone mineral density and chronic inflammation.

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TGF-β1 as possible link between loss of bone mineral density and chronic inflammation.

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JOURNAL.PONE.0014073

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TGF-β1 as possible link between loss of bone mineral density and chronic inflammation.

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Andreas K Nussler

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Andreas Schmitt

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Helen Vester

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Johannes Baur

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Markus Neumaier

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Ulrich Stöckle

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Birth and Death of Bone Cells: Basic Regulatory Mechanisms and Implications for the Pathogenesis and Treatment of Osteoporosis 1

SB-431542 is a potent and specific inhibitor of transforming growth factor-beta superfamily type I activin receptor-like kinase (ALK) receptors ALK4, ALK5, and ALK7

Endogenous TGF-beta signaling suppresses maturation of osteoblastic mesenchymal cells

Mechanisms of TGF-beta signaling from cell membrane to the nucleus

Smad-dependent and Smad-independent pathways in TGF-beta family signalling

Transforming growth factor beta in tissue fibrosis

Hepatocyte-specific Smad7 expression attenuates TGF-beta-mediated fibrogenesis and protects against liver damage

New colorimetric cytotoxicity assay for anticancer-drug screening

Human osteoblast cells: isolation, characterization, and growth on polymers for musculoskeletal tissue engineering.

Pharmacologic inhibition of the TGF-beta type I receptor kinase has anabolic and anti-catabolic effects on bone.

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e14073

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10.1371/JOURNAL.PONE.0014073

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11

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5

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Britt Wildemann

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2010-11-22T00:00:00Z

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49651984

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2989906

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