PHLDA3 is a novel tumor suppressor of pancreatic neuroendocrine tumors. - Wikidata - awgldk - TriplyDB

PHLDA3 is a novel tumor suppressor of pancreatic neuroendocrine tumors.

PHLDA3 is a novel tumor suppressor of pancreatic neuroendocrine tumors.

http://www.wikidata.org/entity/Q33767655

about

Surgical and molecular pathology of pancreatic neoplasms Application and Dosimetric Requirements for Gallium-68-labeled Somatostatin Analogues in Targeted Radionuclide Therapy for Gastroenteropancreatic Neuroendocrine Tumors PHLDA3 impedes somatic cell reprogramming by activating Akt-GSK3β pathway.A vicious partnership between AKT and PHLDA3 to facilitate neuroendocrine tumors.Hypoxia-induced p53 modulates both apoptosis and radiosensitivity via AKT Genetics of pancreatic neuroendocrine tumors: implications for the clinic.Novel p53 target gene FUCA1 encodes a fucosidase and regulates growth and survival of cancer cells.PHLDA3 overexpression in hepatocytes by endoplasmic reticulum stress via IRE1-Xbp1s pathway expedites liver injury.A new immunohistochemistry prognostic score (IPS) for recurrence and survival in resected pancreatic neuroendocrine tumors (PanNET)Pleckstrin homology domain-containing protein PHLDB3 supports cancer growth via a negative feedback loop involving p53.The genetic classification of pancreatic neoplasia.Nuclear imaging of neuroendocrine tumors with unknown primary: why, when and how?DNA methylation profiles distinguish different subtypes of gastroenteropancreatic neuroendocrine tumors.Advances in the diagnosis and treatment of pancreatic neuroendocrine neoplasms in Japan.Ionizing radiation response of primary normal human lens epithelial cells.Germline and somatic genetic changes in multicentric tumors obtained from a patient with multiple endocrine neoplasia type 1.Targeted therapy of gastroenteropancreatic neuroendocrine tumours: preclinical strategies and future targets.Pleckstrin homology-like domain family A, member 3 (PHLDA3) deficiency improves islets engraftment through the suppression of hypoxic damage.p53 Maintains Baseline Expression of Multiple Tumor Suppressor Genes.Pancreatic Cancer: Molecular Characterization, Clonal Evolution and Cancer Stem Cells.PHLDA1, another PHLDA family protein that inhibits Akt

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P2860

PHLDA3 is a novel tumor suppressor of pancreatic neuroendocrine tumors.

http://www.wikidata.org/entity/Q33767655

description

2014 nî lūn-bûn

@nan

2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

PHLDA3 is a novel tumor suppressor of pancreatic neuroendocrine tumors.

@ast

PHLDA3 is a novel tumor suppressor of pancreatic neuroendocrine tumors.

@en

type

label

PHLDA3 is a novel tumor suppressor of pancreatic neuroendocrine tumors.

@ast

PHLDA3 is a novel tumor suppressor of pancreatic neuroendocrine tumors.

@en

prefLabel

PHLDA3 is a novel tumor suppressor of pancreatic neuroendocrine tumors.

@ast

PHLDA3 is a novel tumor suppressor of pancreatic neuroendocrine tumors.

@en

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PNAS.1319962111

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

PHLDA3 is a novel tumor suppressor of pancreatic neuroendocrine tumors.

@en

P2093

Benjamin Tycko

http://www.w3.org/2001/XMLSchema#string

Goichi Yanai

http://www.w3.org/2001/XMLSchema#string

Hideo Namiki

http://www.w3.org/2001/XMLSchema#string

Hiroko Shimizu

http://www.w3.org/2001/XMLSchema#string

Hitoshi Nakagama

http://www.w3.org/2001/XMLSchema#string

Kazuaki Shimada

http://www.w3.org/2001/XMLSchema#string

Kozue Saito

http://www.w3.org/2001/XMLSchema#string

Maiko Minegishi

http://www.w3.org/2001/XMLSchema#string

Naoaki Sakata

http://www.w3.org/2001/XMLSchema#string

Nobuyoshi Hiraoka

http://www.w3.org/2001/XMLSchema#string

P2860

PH domain-only protein PHLDA3 is a p53-regulated repressor of Akt

Placental overgrowth in mice lacking the imprinted gene Ipl

Reprimo, a new candidate mediator of the p53-mediated cell cycle arrest at the G2 phase

DAXX/ATRX, MEN1, and mTOR pathway genes are frequently altered in pancreatic neuroendocrine tumors

Islet beta cell expression of constitutively active Akt1/PKB alpha induces striking hypertrophy, hyperplasia, and hyperinsulinemia

The phosphatidylinositol 3-Kinase AKT pathway in human cancer

Cancer susceptibility polymorphism of p53 at codon 72 affects phosphorylation and degradation of p53 protein.

DNA methylation of the first exon is tightly linked to transcriptional silencing

Multiple endocrine neoplasia type 1.

Everolimus for advanced pancreatic neuroendocrine tumors.

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10.1073/PNAS.1319962111

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111

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