Up-regulation of glycolytic metabolism is required for HIF1α-driven bone formation - Wikidata - awgldk - TriplyDB

Up-regulation of glycolytic metabolism is required for HIF1α-driven bone formation

Up-regulation of glycolytic metabolism is required for HIF1α-driven bone formation

http://www.wikidata.org/entity/Q33767756

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Bone and the regulation of global energy balance Aerobic glycolysis in osteoblasts.Oxygen-sensing PHDs regulate bone homeostasis through the modulation of osteoprotegerin.HIF targets in bone remodeling and metastatic disease.New insights into the biology of osteocalcin.Conditional Deletion of Prolyl Hydroxylase Domain-Containing Protein 2 (Phd2) Gene Reveals Its Essential Role in Chondrocyte Function and Endochondral Bone Formation.Dual function of Bmpr1a signaling in restricting preosteoblast proliferation and stimulating osteoblast activity in mouse Skeletal integration of energy homeostasis: Translational implications.Parathyroid Hormone Reprograms Osteoblast Metabolism PTH Promotes Bone Anabolism by Stimulating Aerobic Glycolysis via IGF Signaling.Navigating the bone marrow niche: translational insights and cancer-driven dysfunction.Prolyl Hydroxylase Domain-Containing Protein 2 (Phd2) Regulates Chondrocyte Differentiation and Secondary Ossification in Mice.Energy Excess, Glucose Utilization, and Skeletal Remodeling: New Insights.Expanding roles of the hypoxia-response network in chronic kidney disease.Mechanisms of marrow adiposity and its implications for skeletal health.Wnt signaling and cellular metabolism in osteoblasts.Bone Cell Bioenergetics and Skeletal Energy Homeostasis.Fatty acid metabolism by the osteoblast.MicroRNA-433 Dampens Glucocorticoid Receptor Signaling, Impacting Circadian Rhythm and Osteoblastic Gene Expression.Energy Metabolism in Mesenchymal Stem Cells During Osteogenic Differentiation.Donor Treatment With a Hypoxia-Inducible Factor-1 Agonist Prevents Donation After Cardiac Death Liver Graft Injury in a Rat Isolated Perfusion Model.An essential role for IGF2 in cartilage development and glucose metabolism during postnatal long bone growth.Vhl deletion in osteoblasts boosts cellular glycolysis and improves global glucose metabolism.Glucose metabolism in bone.Energy Metabolism of the Osteoblast: Implications for Osteoporosis.Recruitment of osteogenic cells to bone formation sites during development and fracture repair.Osteocytic oxygen sensing controls bone mass through epigenetic regulation of sclerostin.Regulation of Osteoblast Metabolism by Wnt Signaling

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P2860

Up-regulation of glycolytic metabolism is required for HIF1α-driven bone formation

http://www.wikidata.org/entity/Q33767756

description

2014 nî lūn-bûn

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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի մայիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Up-regulation of glycolytic metabolism is required for HIF1α-driven bone formation

@ast

Up-regulation of glycolytic metabolism is required for HIF1α-driven bone formation

@en

type

label

Up-regulation of glycolytic metabolism is required for HIF1α-driven bone formation

@ast

Up-regulation of glycolytic metabolism is required for HIF1α-driven bone formation

@en

prefLabel

Up-regulation of glycolytic metabolism is required for HIF1α-driven bone formation

@ast

Up-regulation of glycolytic metabolism is required for HIF1α-driven bone formation

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PNAS.1324290111

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Up-regulation of glycolytic metabolism is required for HIF1α-driven bone formation

@en

P2093

Fanxin Long

http://www.w3.org/2001/XMLSchema#string

Jeffrey M Arbeit

http://www.w3.org/2001/XMLSchema#string

Jenna N Regan

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Joohyun Lim

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Kyu Sang Joeng

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Ralph V Shohet

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Yu Shi

http://www.w3.org/2001/XMLSchema#string

P2860

The tumour suppressor protein VHL targets hypoxia-inducible factors for oxygen-dependent proteolysis

Hypoxia-inducible factor 1 is a basic-helix-loop-helix-PAS heterodimer regulated by cellular O2 tension

Activation of vascular endothelial growth factor gene transcription by hypoxia-inducible factor 1

Understanding the Warburg effect: the metabolic requirements of cell proliferation

Regulation of hypoxia-inducible factor 1alpha is mediated by an O2-dependent degradation domain via the ubiquitin-proteasome pathway

Targeting of HIF-alpha to the von Hippel-Lindau ubiquitylation complex by O2-regulated prolyl hydroxylation

On the Origin of Cancer Cells

Modeling pO(2) distributions in the bone marrow hematopoietic compartment. II. Modified Kroghian models

Ihh signaling is directly required for the osteoblast lineage in the endochondral skeleton

IDH mutation impairs histone demethylation and results in a block to cell differentiation

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23

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262939775

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