The PLC/IP 3 R/PKC pathway is required for ethanol-enhanced GABA release.
about
Neuropeptide modulation of central amygdala neuroplasticity is a key mediator of alcohol dependence.Suppression of Protective Responses upon Activation of L-Type Voltage Gated Calcium Channel in Macrophages during Mycobacterium bovis BCG InfectionSynaptic effects induced by alcohol.Effects of ethanol on glycinergic synaptic currents in mouse spinal cord neurons.Ethanol-enhanced GABA release: a focus on G protein-coupled receptors.Salicylic acid modulates levels of phosphoinositide dependent-phospholipase C substrates and products to remodel the Arabidopsis suspension cell transcriptome.Voluntary Ethanol Consumption Induced by Social Isolation Reverses the Increase of α(4)/δ GABA(A) Receptor Gene Expression and Function in the Hippocampus of C57BL/6J MiceEthanol, not detectably metabolized in brain, significantly reduces brain metabolism, probably via action at specific GABA(A) receptors and has measureable metabolic effects at very low concentrations.Ethanol modulation of synaptic plasticity.Brief alcohol exposure alters transcription in astrocytes via the heat shock pathway.Ultrastructure of synapses in the mammalian brainIncreased voluntary ethanol consumption and changes in hippocampal synaptic plasticity in isolated C57BL/6J mice.Mini-Review: Effects of Ethanol on GABAA Receptor-Mediated Neurotransmission in the Cerebellar Cortex--Recent Advances.The Arabidopsis DREB2 genetic pathway is constitutively repressed by basal phosphoinositide-dependent phospholipase C coupled to diacylglycerol kinase.HSF1 transcriptional activity mediates alcohol induction of Vamp2 expression and GABA releaseElevated expression patterns and tight correlation of the PLCE1 and NF-κB signaling in Kazakh patients with esophageal carcinoma.Diacylglycerol kinases activate tobacco NADPH oxidase-dependent oxidative burst in response to cryptogein.Polyphosphoinositide metabolism and Golgi complex morphology in hippocampal neurons in primary culture is altered by chronic ethanol exposure.
P2860
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P2860
The PLC/IP 3 R/PKC pathway is required for ethanol-enhanced GABA release.
description
2010 nî lūn-bûn
@nan
2010 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի մարտին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
The PLC/IP 3 R/PKC pathway is required for ethanol-enhanced GABA release.
@ast
The PLC/IP 3 R/PKC pathway is required for ethanol-enhanced GABA release.
@en
type
label
The PLC/IP 3 R/PKC pathway is required for ethanol-enhanced GABA release.
@ast
The PLC/IP 3 R/PKC pathway is required for ethanol-enhanced GABA release.
@en
prefLabel
The PLC/IP 3 R/PKC pathway is required for ethanol-enhanced GABA release.
@ast
The PLC/IP 3 R/PKC pathway is required for ethanol-enhanced GABA release.
@en
P2093
P2860
P1433
P1476
The PLC/IP 3 R/PKC pathway is required for ethanol-enhanced GABA release
@en
P2093
George R Breese
Hugh E Criswell
M Katherine Kelm
P2860
P304
P356
10.1016/J.NEUROPHARM.2010.02.018
P577
2010-03-04T00:00:00Z