Self-limiting nature of seasonal cholera epidemics: Role of host-mediated amplification of phage. - Wikidata - awgldk - TriplyDB

Self-limiting nature of seasonal cholera epidemics: Role of host-mediated amplification of phage.

Self-limiting nature of seasonal cholera epidemics: Role of host-mediated amplification of phage.

http://www.wikidata.org/entity/Q33771191

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Environmental reservoirs and mechanisms of persistence of Vibrio cholerae Host, pathogen and the environment: the case of Macrobrachium rosenbergii, Vibrio parahaemolyticus and magnesium Statistical structure of host-phage interactions Transmission of Vibrio cholerae is antagonized by lytic phage and entry into the aquatic environment Genomic science in understanding cholera outbreaks and evolution of Vibrio cholerae as a human pathogen.Vibrio parahaemolyticus and its specific bacteriophages as an indicator in cockles (Anadara granosa) for the risk of V. parahaemolyticus infection in Southern Thailand.Distribution of genes for virulence and ecological fitness among diverse Vibrio cholerae population in a cholera endemic area: tracking the evolution of pathogenic strains.Acquisition of classical CTX prophage from Vibrio cholerae O141 by El Tor strains aided by lytic phages and chitin-induced competence.An experimental study of phage mediated bactericidal selection & emergence of the El Tor Vibrio cholerae.The burden of cholera.Prediction of epidemic cholera due to Vibrio cholerae O1 in children younger than 10 years using climate data in Bangladesh.Vibrio2005: the First International Conference on the Biology of Vibrios Short-term genome evolution of Listeria monocytogenes in a non-controlled environment Spatial clustering in the spatio-temporal dynamics of endemic cholera The cyclic AMP (cAMP)-cAMP receptor protein signaling system mediates resistance of Vibrio cholerae O1 strains to multiple environmental bacteriophages.Quorum-regulated biofilms enhance the development of conditionally viable, environmental Vibrio cholerae Nasty viruses, costly plasmids, population dynamics, and the conditions for establishing and maintaining CRISPR-mediated adaptive immunity in bacteria.Minimal genetic change in Vibrio cholerae in Mozambique over time: Multilocus variable number tandem repeat analysis and whole genome sequencing Environmental bacteriophages active on biofilms and planktonic forms of toxigenic Vibrio cholerae: Potential relevance in cholera epidemiology Back to the future: studying cholera pathogenesis using infant rabbits A bacteriophage encodes its own CRISPR/Cas adaptive response to evade host innate immunity Detection of Vibrio cholerae O1 and O139 in environmental water samples by an immunofluorescent-aggregation assay.Evolutionary consequences of intra-patient phage predation on microbial populations.Killing the killers.Biofilm formation and phenotypic variation enhance predation-driven persistence of Vibrio cholerae Bacteriophage prehistory: Is or is not Hankin, 1896, a phage reference?Phase variable O antigen biosynthetic genes control expression of the major protective antigen and bacteriophage receptor in Vibrio cholerae O1 Diagnostic limitations to accurate diagnosis of cholera.O antigen is the receptor of Vibrio cholerae serogroup O1 El Tor typing phage VP4 Evidence of a dominant lineage of Vibrio cholerae-specific lytic bacteriophages shed by cholera patients over a 10-year period in Dhaka, Bangladesh.Modeling the role of bacteriophage in the control of cholera outbreaks Transmissibility of cholera: in vivo-formed biofilms and their relationship to infectivity and persistence in the environment.Bacteria-phage coevolution as a driver of ecological and evolutionary processes in microbial communities.Seasonal cholera caused by Vibrio cholerae serogroups O1 and O139 in the coastal aquatic environment of Bangladesh.Characterization of Vibrio cholerae bacteriophages isolated from the environmental waters of the Lake Victoria region of Kenya.Prevalence and characterisation of non-cholerae Vibrio spp. in final effluents of wastewater treatment facilities in two districts of the Eastern Cape Province of South Africa: implications for public health Cholera: Environmental Reservoirs and Impact on Disease Transmission.RNA-seq analysis identifies new genes regulated by the histone-like nucleoid structuring protein (H-NS) affecting Vibrio cholerae virulence, stress response and chemotaxis.Diarrheal epidemics in Dhaka, Bangladesh, during three consecutive floods: 1988, 1998, and 2004.Identifying and analyzing bacteriophages in human fecal samples: what could we discover?

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P2860

Self-limiting nature of seasonal cholera epidemics: Role of host-mediated amplification of phage.

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Self-limiting nature of season ...... diated amplification of phage.

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Self-limiting nature of season ...... diated amplification of phage.

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A S G Faruque

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G Balakrish Nair

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John J Mekalanos

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M Johirul Islam

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Qazi Shafi Ahmad

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Shah M Faruque

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P2860

Epidemiology, genetics, and ecology of toxigenic Vibrio cholerae

Seasonal epidemics of cholera inversely correlate with the prevalence of environmental cholera phages.

Response of man to infection with Vibrio cholerae. I. Clinical, serologic, and bacteriologic responses to a known inoculum.

Tropism switching in Bordetella bacteriophage defines a family of diversity-generating retroelements.

Lysogenic conversion by a filamentous phage encoding cholera toxin.

Pathogenicity islands and phages in Vibrio cholerae evolution.

A new type of conjugative transposon encodes resistance to sulfamethoxazole, trimethoprim, and streptomycin in Vibrio cholerae O139.

Detection of Vibrio cholerae O1 in the aquatic environment by fluorescent-monoclonal antibody and culture methods.

Molecular epidemiology of toxigenic Vibrio cholerae in Bangladesh studied by numerical analysis of rRNA gene restriction patterns.

Active surveillance for Vibrio cholerae O1 and vibriophages in sewage water as a potential tool to predict cholera outbreaks.

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