Normal myeloid development requires both the glutamine-rich transactivation domain and the PEST region of transcription factor PU.1 but not the potent acidic transactivation domain.
about
Isoforms of the Ets transcription factor NERF/ELF-2 physically interact with AML1 and mediate opposing effects on AML1-mediated transcription of the B cell-specific blk geneAKT induces transcriptional activity of PU.1 through phosphorylation-mediated modifications within its transactivation domainThe Q-rich subdomain of the human Ah receptor transactivation domain is required for dioxin-mediated transcriptional activityPU.1, interferon regulatory factor (IRF) 2, and the interferon consensus sequence-binding protein (ICSBP/IRF8) cooperate to activate NF1 transcription in differentiating myeloid cellsSpi-B can functionally replace PU.1 in myeloid but not lymphoid developmentBSAP can repress enhancer activity by targeting PU.1 function.Evolution of hematopoiesis: Three members of the PU.1 transcription factor family in a cartilaginous fish, Raja eglanteria.Role of Erk1/2 signaling in the regulation of neutrophil versus monocyte development in response to G-CSF and M-CSF.Gcn5 is required for PU.1-dependent IL-9 induction in Th9 cells.A gamete-specific, sex-limited homeodomain protein in ChlamydomonasMutations of the transcription factor PU.1 are not associated with acute lymphoblastic leukaemia.Analysis of concentration-dependent functions of PU.1 in hematopoiesis using mouse models.PU.1 binding to ets motifs within the equine infectious anemia virus long terminal repeat (LTR) enhancer: regulation of LTR activity and virus replication in macrophages.Mi2beta shows chromatin enzyme specificity by erasing a DNase I-hypersensitive site established by ACF.Blood pressure homeostasis is maintained by a P311-TGF-β axisc-Jun is a JNK-independent coactivator of the PU.1 transcription factor.E2A Antagonizes PU.1 Activity through Inhibition of DNA Binding.Role of Ets Proteins in Development, Differentiation, and Function of T-Cell Subsets.PU.1 regulates the expression of the vav proto-oncogene.In vivo complex formation of PU.1 with HDAC1 associated with PU.1-mediated transcriptional repression.Isolation and characterization of casein kinase I from Dictyostelium discoideum.From totipotency to T in a dish.
P2860
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P2860
Normal myeloid development requires both the glutamine-rich transactivation domain and the PEST region of transcription factor PU.1 but not the potent acidic transactivation domain.
description
1998 nî lūn-bûn
@nan
1998 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Normal myeloid development req ...... acidic transactivation domain.
@ast
Normal myeloid development req ...... acidic transactivation domain.
@en
type
label
Normal myeloid development req ...... acidic transactivation domain.
@ast
Normal myeloid development req ...... acidic transactivation domain.
@en
prefLabel
Normal myeloid development req ...... acidic transactivation domain.
@ast
Normal myeloid development req ...... acidic transactivation domain.
@en
P2093
P2860
P356
P1476
Normal myeloid development req ...... acidic transactivation domain.
@en
P2093
J M Perkel
J M Pongubala
M L Atchison
R C Fisher
P2860
P304
P356
10.1128/MCB.18.7.4347
P407
P577
1998-07-01T00:00:00Z