Implication of N-acetylglucosaminyltransferases III and V in cancer: gene regulation and signaling mechanism.
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Engagement of I-branching {beta}-1, 6-N-acetylglucosaminyltransferase 2 in breast cancer metastasis and TGF-{beta} signalingInnate immunity and transcription of MGAT-III and Toll-like receptors in Alzheimer's disease patients are improved by bisdemethoxycurcuminRegulation of differentiation- and proliferation-inducers on Lewis antigens, alpha-fucosyltransferase and metastatic potential in hepatocarcinoma cellsThe recognition of N-glycans by the lectin ArtinM mediates cell death of a human myeloid leukemia cell lineEvolutional and clinical implications of the epigenetic regulation of protein glycosylation.Metastatic transcriptional pattern revealed by gene expression profiling in primary colorectal carcinoma.Rapid analysis of amino sugars by microchip electrophoresis with laser-induced fluorescence detection.Immune defects in Alzheimer's disease: new medications development.Immunoglobulins are the major glycoproteins involved in the modifications of total serum N-glycome in cirrhotic patients.Glycosylation of liver acute-phase proteins in pancreatic cancer and chronic pancreatitis.Down-regulation of hydrogen peroxide-induced PKC delta activation in N-acetylglucosaminyltransferase III-transfected HeLaS3 cells.Loss and recovery of Mgat3 and GnT-III Mediated E-cadherin N-glycosylation is a mechanism involved in epithelial-mesenchymal-epithelial transitions.Suppression of beta 1,3galactosyltransferase beta 3Gal-T5 in cancer cells reduces sialyl-Lewis a and enhances poly N-acetyllactosamines and sialyl-Lewis x on O-glycans.Wisteria floribunda agglutinin-binding glycan expression is decreased in endometriomata.Function characterization of a glyco-engineered anti-EGFR monoclonal antibody cetuximab in vitro.Reversibility of membrane N-glycome of HeLa cells upon treatment with epigenetic inhibitors.Cell surface glycan alterations in epithelial mesenchymal transition process of Huh7 hepatocellular carcinoma cell.Hexosamine-Induced TGF-β Signaling and Osteogenic Differentiation of Dental Pulp Stem Cells Are Dependent on N-Acetylglucosaminyltransferase V.Overexpression and β-1,6-N-acetylglucosaminylation-initiated aberrant glycosylation of TIMP-1: a "double whammy" strategy in colon cancer progressionGlycoprotein disease markers and single protein-omicsNegative expression of N-acetylglucosaminyltransferase V in oral squamous cell carcinoma correlates with poor prognosis.Lentiviral vector-mediated shRNA against AIMP2-DX2 suppresses lung cancer cell growth through blocking glucose uptake.Aberrant glycosylation as biomarker for cancer: focus on CD43.Involvement of aberrant glycosylation in thyroid cancer.Integrated approach toward the discovery of glyco-biomarkers of inflammation-related diseases.Cancer intelligence acquired (CIA): tumor glycosylation and sialylation codes dismantling antitumor defense.Hepatic aberrant glycosylation by N-acetylglucosaminyltransferase V accelerates HDL assembly.A glycoproteomic approach to identify novel glycomarkers for cancer stem cells.N-acetylglucosaminyltransferase V modulates radiosensitivity and migration of small cell lung cancer through epithelial-mesenchymal transition.Expression of N-acetylglucosaminyltransferase V in the subserosal layer correlates with postsurgical survival of pathological tumor stage 2 carcinoma of the gallbladder.Functional roles of glycogene and N-glycan in multidrug resistance of human breast cancer cells.Lewis y antigen is expressed in oral squamous cell carcinoma cell lines and tissues, but disappears in the invasive regions leading to the enhanced malignant properties irrespective of sialyl-Lewis x.Modulation of CD147-induced matrix metalloproteinase activity: role of CD147 N-glycosylation.Core fucosylation of E-cadherin enhances cell-cell adhesion in human colon carcinoma WiDr cells.Alpha1,3 Fucosyltransferase-VII modifies the susceptibility of apoptosis induced by ultraviolet and retinoic acid in human hepatocarcinoma cells.Introduction of bisecting GlcNAc in N-glycans of adenylyl cyclase III enhances its activity.Down regulation of N-acetylglucosaminyltransferase V facilitates all-transretinoic acid to induce apoptosis of human hepatocarcinoma cells.Glycomic/glycoproteomic analysis by liquid chromatography/mass spectrometry: analysis of glycan structural alteration in cells.Regulation of metastasis-suppressive gene Nm23-H1 on glycosyl-transferases involved in the synthesis of sialyl Lewis antigens.Loss of Barx1 promotes hepatocellular carcinoma metastasis through up-regulating MGAT5 and MMP9 expression and indicates poor prognosis.
P2860
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P2860
Implication of N-acetylglucosaminyltransferases III and V in cancer: gene regulation and signaling mechanism.
description
1999 nî lūn-bûn
@nan
1999 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Implication of N-acetylglucosa ...... ation and signaling mechanism.
@ast
Implication of N-acetylglucosa ...... ation and signaling mechanism.
@en
type
label
Implication of N-acetylglucosa ...... ation and signaling mechanism.
@ast
Implication of N-acetylglucosa ...... ation and signaling mechanism.
@en
prefLabel
Implication of N-acetylglucosa ...... ation and signaling mechanism.
@ast
Implication of N-acetylglucosa ...... ation and signaling mechanism.
@en
P2093
P1476
Implication of N-acetylglucosa ...... ation and signaling mechanism.
@en
P2093
P304
P356
10.1016/S0925-4439(99)00066-6
P407
P577
1999-10-01T00:00:00Z