Skeletal muscle NADPH oxidase is increased and triggers stretch-induced damage in the mdx mouse.
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SIRT1: A Novel Target for the Treatment of Muscular DystrophiesAerobic Exercise and Pharmacological Therapies for Skeletal Myopathy in Heart Failure: Similarities and DifferencesCritical Role of Intracellular RyR1 Calcium Release Channels in Skeletal Muscle Function and DiseaseTaurine: the appeal of a safe amino acid for skeletal muscle disordersNox family NADPH oxidases in mechano-transduction: mechanisms and consequencesBang-bang model for regulation of local blood flowRedox Mechanism of Reactive Oxygen Species in ExerciseAge-related alterations in the sarcolemmal environment are attenuated by lifelong caloric restriction and voluntary exerciseMicrotubules underlie dysfunction in duchenne muscular dystrophy.Src-dependent impairment of autophagy by oxidative stress in a mouse model of Duchenne muscular dystrophyRedox state and mitochondrial respiratory chain function in skeletal muscle of LGMD2A patients.Electrical stimulation influences satellite cell proliferation and apoptosis in unloading-induced muscle atrophy in miceAngiotensin II modulates mouse skeletal muscle resting conductance to chloride and potassium ions and calcium homeostasis via the AT1 receptor and NADPH oxidaseDiapocynin, a dimer of the NADPH oxidase inhibitor apocynin, reduces ROS production and prevents force loss in eccentrically contracting dystrophic muscleLoss of nNOS inhibits compensatory muscle hypertrophy and exacerbates inflammation and eccentric contraction-induced damage in mdx mice.Nifedipine treatment reduces resting calcium concentration, oxidative and apoptotic gene expression, and improves muscle function in dystrophic mdx mice.Altered ROS production, NF-κB activation and interleukin-6 gene expression induced by electrical stimulation in dystrophic mdx skeletal muscle cellsQuantitative evaluation of the beneficial effects in the mdx mouse of epigallocatechin gallate, an antioxidant polyphenol from green tea.Epigenetic Regulatory Effect of Exercise on Glutathione Peroxidase 1 Expression in the Skeletal Muscle of Severely Dyslipidemic Mice.Mechanisms of Cachexia in Chronic Disease StatesNOX2-dependent ROS is required for HDAC5 nuclear efflux and contributes to HDAC4 nuclear efflux during intense repetitive activity of fast skeletal muscle fibersA new therapeutic effect of simvastatin revealed by functional improvement in muscular dystrophy.Emerging drugs for Duchenne muscular dystrophyCrosstalk between RyR2 oxidation and phosphorylation contributes to cardiac dysfunction in mice with Duchenne muscular dystrophy.Absence of Dystrophin Disrupts Skeletal Muscle Signaling: Roles of Ca2+, Reactive Oxygen Species, and Nitric Oxide in the Development of Muscular Dystrophy.Studies of mitochondrial and nonmitochondrial sources implicate nicotinamide adenine dinucleotide phosphate oxidase(s) in the increased skeletal muscle superoxide generation that occurs during contractile activityLow Po₂ conditions induce reactive oxygen species formation during contractions in single skeletal muscle fibersMolecular mechanisms and signaling pathways of angiotensin II-induced muscle wasting: potential therapeutic targets for cardiac cachexiaWasting mechanisms in muscular dystrophy.Role of dystroglycan in limiting contraction-induced injury to the sarcomeric cytoskeleton of mature skeletal muscle.Diaphragm Abnormalities in Patients with End-Stage Heart Failure: NADPH Oxidase Upregulation and Protein Oxidation.X-ROS signaling in the heart and skeletal muscle: stretch-dependent local ROS regulates [Ca²⁺]i.EUK-134 ameliorates nNOSμ translocation and skeletal muscle fiber atrophy during short-term mechanical unloading.Exacerbation of pathology by oxidative stress in respiratory and locomotor muscles with Duchenne muscular dystrophy.Angiotensin II, oxidative stress and skeletal muscle wasting.Contribution of oxidative stress to pathology in diaphragm and limb muscles with Duchenne muscular dystrophy.Oxidative stress in muscular dystrophy: from generic evidence to specific sources and targets.Drug Discovery of Therapies for Duchenne Muscular Dystrophy.Redox homeostasis and age-related deficits in neuromuscular integrity and function.Regulation of NADPH oxidases in skeletal muscle.
P2860
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P2860
Skeletal muscle NADPH oxidase is increased and triggers stretch-induced damage in the mdx mouse.
description
2010 nî lūn-bûn
@nan
2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Skeletal muscle NADPH oxidase ...... duced damage in the mdx mouse.
@ast
Skeletal muscle NADPH oxidase ...... duced damage in the mdx mouse.
@en
type
label
Skeletal muscle NADPH oxidase ...... duced damage in the mdx mouse.
@ast
Skeletal muscle NADPH oxidase ...... duced damage in the mdx mouse.
@en
prefLabel
Skeletal muscle NADPH oxidase ...... duced damage in the mdx mouse.
@ast
Skeletal muscle NADPH oxidase ...... duced damage in the mdx mouse.
@en
P2860
P1433
P1476
Skeletal muscle NADPH oxidase ...... duced damage in the mdx mouse.
@en
P2093
Nicholas P Whitehead
Stanley C Froehner
P2860
P304
P356
10.1371/JOURNAL.PONE.0015354
P407
P577
2010-12-20T00:00:00Z