Fra-1 induces morphological transformation and increases in vitro invasiveness and motility of epithelioid adenocarcinoma cells.
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C-Fos expression is a molecular predictor of progression and survival in epithelial ovarian carcinomaWound healing and inflammation genes revealed by array analysis of 'macrophageless' PU.1 null miceRegulatory pathways controlling the endovascular invasive trophoblast cell lineageTumor suppressor p53 protein is a new target for the metastasis-associated Mts1/S100A4 protein: functional consequences of their interactionElevated protein expression of cyclin D1 and Fra-1 but decreased expression of c-Myc in human colorectal adenocarcinomas overexpressing beta-cateninFOSL1 controls the assembly of endothelial cells into capillary tubes by direct repression of αv and β3 integrin transcriptionFra-1 promotes growth and survival in RAS-transformed thyroid cells by controlling cyclin A transcriptionDiversity of gene expression in adenocarcinoma of the lungAP-1 differentially expressed proteins Krp1 and fibronectin cooperatively enhance Rho-ROCK-independent mesenchymal invasion by altering the function, localization, and activity of nondifferentially expressed proteins.Small molecule inhibitors targeting activator protein 1 (AP-1).c-FOS suppresses ovarian cancer progression by changing adhesionRegulation of a multigenic invasion programme by the transcription factor, AP-1: re-expression of a down-regulated gene, TSC-36, inhibits invasion.An association of a simultaneous nuclear and cytoplasmic localization of Fra-1 with breast malignancy.FRA-1 protein overexpression is a feature of hyperplastic and neoplastic breast disordersModeling metastasis in the mouse.Emerging roles of ATF2 and the dynamic AP1 network in cancer.Characterization of Sp1, AP-1, CBF and KRC binding sites and minisatellite DNA as functional elements of the metastasis-associated mts1/S100A4 gene intronic enhancer.Dolichol-phosphate-mannose-3 (DPM3)/prostin-1 is a novel phospholipase C-gamma regulated gene negatively associated with prostate tumor invasion.Blockade of AP1 transactivation abrogates the abnormal expression of breast cancer-specific gene 1 in breast cancer cells.Transactivation of Fra-1 and consequent activation of AP-1 occur extracellular signal-regulated kinase dependently.Old players with a newly defined function: Fra-1 and c-Fos support growth of human malignant breast tumors by activating membrane biogenesis at the cytoplasm.A network of immediate early gene products propagates subtle differences in mitogen-activated protein kinase signal amplitude and duration.Role of Fos-related antigen 1 in the progression and prognosis of ductal breast carcinoma.Identify lymphatic metastasis-associated genes in mouse hepatocarcinoma cell lines using gene chipFRA-1 as a driver of tumour heterogeneity: a nexus between oncogenes and embryonic signalling pathways in cancer.Transcription factor Fos-related antigen 1 is an effective target for a breast cancer vaccine.Aberrantly expressed Fra-1 by IL-6/STAT3 transactivation promotes colorectal cancer aggressiveness through epithelial-mesenchymal transition.FOSL1 is integral to establishing the maternal-fetal interfaceGene therapy for carcinoma of the breast.Pharmacology and anti-tumor activity of RWJ67657, a novel inhibitor of p38 mitogen activated protein kinaseInvasion of normal human fibroblasts induced by v-Fos is independent of proliferation, immortalization, and the tumor suppressors p16INK4a and p53.Osteogenic capillaries orchestrate growth plate-independent ossification of the malleus.Identification of a pharmacologically tractable Fra-1/ADORA2B axis promoting breast cancer metastasis.Polo-like kinase 1 induces epithelial-to-mesenchymal transition and promotes epithelial cell motility by activating CRAF/ERK signaling.The PKCθ pathway participates in the aberrant accumulation of Fra-1 protein in invasive ER-negative breast cancer cells.The Bromodomain BET Inhibitor JQ1 Suppresses Tumor Angiogenesis in Models of Childhood SarcomaA novel combination: ranpirnase and rosiglitazone induce a synergistic apoptotic effect by down-regulating Fra-1 and Survivin in cancer cells.Inhibition of AP-1 transcriptional activity blocks the migration, invasion, and experimental metastasis of murine osteosarcoma.Oral DNA vaccines target the tumor vasculature and microenvironment and suppress tumor growth and metastasis.Selective activator protein-1 inhibitor T-5224 prevents lymph node metastasis in an oral cancer model
P2860
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P2860
Fra-1 induces morphological transformation and increases in vitro invasiveness and motility of epithelioid adenocarcinoma cells.
description
1998 nî lūn-bûn
@nan
1998 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Fra-1 induces morphological tr ...... thelioid adenocarcinoma cells.
@ast
Fra-1 induces morphological tr ...... thelioid adenocarcinoma cells.
@en
type
label
Fra-1 induces morphological tr ...... thelioid adenocarcinoma cells.
@ast
Fra-1 induces morphological tr ...... thelioid adenocarcinoma cells.
@en
prefLabel
Fra-1 induces morphological tr ...... thelioid adenocarcinoma cells.
@ast
Fra-1 induces morphological tr ...... thelioid adenocarcinoma cells.
@en
P2093
P2860
P356
P1476
Fra-1 induces morphological tr ...... thelioid adenocarcinoma cells.
@en
P2093
D Kramerov
E Lukanidin
E Tulchinsky
M Grigorian
O Kustikova
P2860
P304
P356
10.1128/MCB.18.12.7095
P407
P577
1998-12-01T00:00:00Z