Metabolic syndrome reduces the contribution of K+ channels to ischemic coronary vasodilation - Wikidata - awgldk - TriplyDB

Metabolic syndrome reduces the contribution of K+ channels to ischemic coronary vasodilation

Metabolic syndrome reduces the contribution of K+ channels to ischemic coronary vasodilation

http://www.wikidata.org/entity/Q33784407

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Effect of Soluble Epoxide Hydrolase on the Modulation of Coronary Reactive Hyperemia: Role of Oxylipins and PPARγ Deletion of soluble epoxide hydrolase enhances coronary reactive hyperemia in isolated mouse heart: role of oxylipins and PPARγ Vascular Endothelial Over-Expression of Human Soluble Epoxide Hydrolase (Tie2-sEH Tr) Attenuates Coronary Reactive Hyperemia in Mice: Role of Oxylipins and ω-Hydroxylases Heart of the matter: coronary dysfunction in metabolic syndrome.Contribution of electromechanical coupling between Kv and Ca v1.2 channels to coronary dysfunction in obesity.Metabolic syndrome and coronary artery disease in Ossabaw compared with Yucatan swine.Modeling cardiac arrest and resuscitation in the domestic pig Ca2+ regulatory mechanisms of exercise protection against coronary artery disease in metabolic syndrome and diabetes.Contribution of voltage-dependent K⁺ channels to metabolic control of coronary blood flow.Impaired coronary collateral growth: miR-shaken neutrophils caught in the act.Large conductance, calcium- and voltage-gated potassium (BK) channels: regulation by cholesterol.Daming capsule restores endothelial dysfunction induced by high-fat diet Penitrem A as a tool for understanding the role of large conductance Ca(2+)/voltage-sensitive K(+) channels in vascular function.Vascular endothelial overexpression of human CYP2J2 (Tie2-CYP2J2 Tr) modulates cardiac oxylipin profiles and enhances coronary reactive hyperemia in mice.Exercise training-enhanced, endothelium-dependent dilation mediated by altered regulation of BK(Ca) channels in collateral-dependent porcine coronary arterioles A1 adenosine receptor negatively modulates coronary reactive hyperemia via counteracting A2A-mediated H2O2 production and KATP opening in isolated mouse hearts Contribution of hydrogen sulfide to the control of coronary blood flow.Role of ion channels in coronary microcirculation: a review of the literature.Sudden cardiac death and obesity.Cardiovascular consequences of metabolic syndrome.Contribution of voltage-dependent K+ and Ca2+ channels to coronary pressure-flow autoregulation.Contribution of adenosine A(2A) and A(2B) receptors to ischemic coronary dilation: role of K(V) and K(ATP) channels.Contribution of IKCa channels to the control of coronary blood flow.Reduced coronary reactive hyperemia in mice was reversed by the soluble epoxide hydrolase inhibitor (t-AUCB): Role of adenosine A2A receptor and plasma oxylipins.Regulation of Coronary Blood Flow.The Coronary Microcirculation in Health and Disease

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Metabolic syndrome reduces the contribution of K+ channels to ischemic coronary vasodilation

http://www.wikidata.org/entity/Q33784407

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2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Metabolic syndrome reduces the ...... ischemic coronary vasodilation

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AJPHEART.00888.2009

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American Journal of Physiology - Heart and Circulatory Physiology

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Metabolic syndrome reduces the ...... ischemic coronary vasodilation

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P2093

Gregory A Payne

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Gregory M Dick

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Ian N Bratz

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Johnathan D Tune

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Léna Borbouse

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Michael Sturek

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Mouhamad Alloosh

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Zachary C Berwick

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Zachary P Neeb

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P2860

Contribution of BK(Ca) channels to local metabolic coronary vasodilation: Effects of metabolic syndrome.

Diabetes and cardiovascular disease: a statement for healthcare professionals from the American Heart Association.

Ion channels and vascular tone.

Mediators of coronary reactive hyperaemia in isolated mouse heart.

Altered mechanism of adenosine-induced coronary arteriolar dilation in early-stage metabolic syndrome.

The influence of obesity on calf blood flow and vascular reactivity in older adults.

The link between abdominal obesity, metabolic syndrome and cardiovascular disease.

Effects of exercise training and hypercholesterolemia on adenosine activation of voltage-dependent K+ channels in coronary arterioles.

Role of K+ ATP channels and adenosine in the regulation of coronary blood flow during exercise with normal and restricted coronary blood flow.

Impaired function of coronary BK(Ca) channels in metabolic syndrome

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10.1152/AJPHEART.00888.2009

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2010-01-29T00:00:00Z

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20118408

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metabolic syndrome

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