Loss of caveolin-1 accelerates neurodegeneration and aging. - Wikidata - awgldk - TriplyDB

Loss of caveolin-1 accelerates neurodegeneration and aging.

Loss of caveolin-1 accelerates neurodegeneration and aging.

http://www.wikidata.org/entity/Q33784520

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Cholesterol homeostasis: a key to prevent or slow down neurodegeneration Overnutrition Determines LPS Regulation of Mycotoxin Induced Neurotoxicity in Neurodegenerative Diseases Cholesterol in the Pathogenesis of Alzheimer's, Parkinson's Diseases and Autism: Link to Synaptic Dysfunction Nerve Growth Factor Signaling from Membrane Microdomains to the Nucleus: Differential Regulation by Caveolins.Impact of the loss of caveolin-1 on lung mass and cholesterol metabolism in mice with and without the lysosomal cholesterol transporter, Niemann-Pick type C1.Mitochondria-localized caveolin in adaptation to cellular stress and injury.Long-term atorvastatin treatment leads to alterations in behavior, cognition, and hippocampal biochemistry.Dysfunctional survival-signaling and stress-intolerance in aged murine and human myocardium.Signaling epicenters: the role of caveolae and caveolins in volatile anesthetic induced cardiac protection.Sarcolemmal cholesterol and caveolin-3 dependence of cardiac function, ischemic tolerance, and opioidergic cardioprotection Neuronal differentiation dictates estrogen-dependent survival and ERK1/2 kinetic by means of caveolin-1.Neuron-targeted caveolin-1 protein enhances signaling and promotes arborization of primary neurons.Caveolin-1, cellular senescence and age-related diseases.Role of the Insulin-Like Growth Factor Type 1 Receptor in the Pathogenesis of Diabetic Encephalopathy.Caveolin-1 and accelerated host aging in the breast tumor microenvironment: chemoprevention with rapamycin, an mTOR inhibitor and anti-aging drug Caveolins: targeting pro-survival signaling in the heart and brain.Loss of parkin promotes lipid rafts-dependent endocytosis through accumulating caveolin-1: implications for Parkinson's disease Caveolin and caveolae in age associated cardiovascular disease Breast Cancer beyond the Age of Mutation.The plasma membrane as a capacitor for energy and metabolism.Rosiglitazone ameliorates diffuse axonal injury by reducing loss of tau and up-regulating caveolin-1 expression.Citrulline diet supplementation improves specific age-related raft changes in wild-type rodent hippocampus.CLN3 loss disturbs membrane microdomain properties and protein transport in brain endothelial cells.Interaction of membrane/lipid rafts with the cytoskeleton: impact on signaling and function: membrane/lipid rafts, mediators of cytoskeletal arrangement and cell signaling.Versatile Functions of Caveolin-1 in Aging-related Diseases.Cav-1 deletion impaired hematopoietic stem cell function.Traumatic brain injury enhances neuroinflammation and lesion volume in caveolin deficient mice From embryonic development to human diseases: The functional role of caveolae/caveolin.The less-often-traveled surface of stem cells: caveolin-1 and caveolae in stem cells, tissue repair and regeneration.Caveolae: molecular insights and therapeutic targets for stroke.Mitochondrial cholesterol: a connection between caveolin, metabolism, and disease.ROR1 sustains caveolae and survival signalling as a scaffold of cavin-1 and caveolin-1 Sarcolemmal dependence of cardiac protection and stress-resistance: roles in aged or diseased hearts.Caveolins and caveolae in ocular physiology and pathophysiology.Caveolin proteins: a molecular insight into disease.Caveolin isoform switching as a molecular, structural, and metabolic regulator of microglia.Characterizing the "POAGome": A bioinformatics-driven approach to primary open-angle glaucoma.MicroRNA-181 promotes synaptogenesis and attenuates axonal outgrowth in cortical neurons.Caveolin-1 deficiency induces premature senescence with mitochondrial dysfunction.High glucose induces formation of tau hyperphosphorylation via Cav-1-mTOR pathway: A potential molecular mechanism for diabetes-induced cognitive dysfunction.

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Loss of caveolin-1 accelerates neurodegeneration and aging.

http://www.wikidata.org/entity/Q33784520

description

2010 nî lūn-bûn

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2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Loss of caveolin-1 accelerates neurodegeneration and aging.

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Loss of caveolin-1 accelerates neurodegeneration and aging.

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type

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Loss of caveolin-1 accelerates neurodegeneration and aging.

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Loss of caveolin-1 accelerates neurodegeneration and aging.

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Loss of caveolin-1 accelerates neurodegeneration and aging.

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Loss of caveolin-1 accelerates neurodegeneration and aging.

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Loss of caveolin-1 accelerates neurodegeneration and aging.

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Atsushi Miyanohara

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Brian P Head

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Mathivadhani Panneerselvam

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P2860

Alzheimer Disease in the US Population

The role of seladin-1/DHCR24 in cholesterol biosynthesis, APP processing and Abeta generation in vivo.

Actin in dendritic spines: connecting dynamics to function

p120 catenin regulates dendritic spine and synapse development through Rho-family GTPases and cadherins

Role of beta-catenin in synaptic vesicle localization and presynaptic assembly

Caveolae, caveolin and caveolin-rich membrane domains: a signalling hypothesis

Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium

Quantification of synapse formation and maintenance in vivo in the absence of synaptic release

Caveolin-1 deficiency increases cerebral ischemic injury

Development and Regulation of Dendritic Spine Synapses

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