P0-deficient knockout mice as tools to understand pathomechanisms in Charcot-Marie-Tooth 1B and P0-related Déjérine-Sottas syndrome.
about
Proteolipid protein cannot replace P0 protein as the major structural protein of peripheral nervous system myelin.GlcNAc6ST-1 regulates sulfation of N-glycans and myelination in the peripheral nervous systemMpzR98C arrests Schwann cell development in a mouse model of early-onset Charcot-Marie-Tooth disease type 1B.Pathology of a mouse mutation in peripheral myelin protein P0 is characteristic of a severe and early onset form of human Charcot-Marie-Tooth type 1B disorder.GPR56/ADGRG1 regulates development and maintenance of peripheral myelin.Functional recovery of regenerating motor axons is delayed in mice heterozygously deficient for the myelin protein P(0) gene.
P2860
P0-deficient knockout mice as tools to understand pathomechanisms in Charcot-Marie-Tooth 1B and P0-related Déjérine-Sottas syndrome.
description
1999 nî lūn-bûn
@nan
1999 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
P0-deficient knockout mice as ...... ated Déjérine-Sottas syndrome.
@ast
P0-deficient knockout mice as ...... ated Déjérine-Sottas syndrome.
@en
type
label
P0-deficient knockout mice as ...... ated Déjérine-Sottas syndrome.
@ast
P0-deficient knockout mice as ...... ated Déjérine-Sottas syndrome.
@en
prefLabel
P0-deficient knockout mice as ...... ated Déjérine-Sottas syndrome.
@ast
P0-deficient knockout mice as ...... ated Déjérine-Sottas syndrome.
@en
P2860
P1476
P0-deficient knockout mice as ...... lated Déjérine-Sottas syndrome
@en
P2093
P2860
P304
P356
10.1111/J.1749-6632.1999.TB08589.X
P407
P577
1999-09-01T00:00:00Z