TNF alpha and IL-1 beta are separate targets in chronic arthritis.
about
Early response genes induced in chondrocytes stimulated with the inflammatory cytokine interleukin-1betaRANKL inhibition by osteoprotegerin prevents bone loss without affecting local or systemic inflammation parameters in two rat arthritis models: comparison with anti-TNFalpha or anti-IL-1 therapiesSynovial fluid concentrations and relative potency of interleukin-1 alpha and beta in cartilage and meniscus degradationDynamic loading enhances integrative meniscal repair in the presence of interleukin-1.Interleukin-1, tumor necrosis factor-alpha, and transforming growth factor-beta 1 and integrative meniscal repair: influences on meniscal cell proliferation and migration.Interleukin-1 and tumor necrosis factor alpha inhibit repair of the porcine meniscus in vitro.Chondrogenic differentiation of human subchondral progenitor cells is affected by synovial fluid from donors with osteoarthritis or rheumatoid arthritisIFN-γ-deficient mice develop IL-1-dependent cutaneous and musculoskeletal inflammation during experimental brucellosis.Cytokine preconditioning of engineered cartilage provides protection against interleukin-1 insult.The effects of adipokines on cartilage and meniscus catabolismInduction of arthritis by high mobility group box chromosomal protein 1 is independent of tumour necrosis factor signalling.Inhibition of matrix metalloproteinases enhances in vitro repair of the meniscusIntegrative repair of the meniscus: lessons from in vitro studies.Inhibition of integrative repair of the meniscus following acute exposure to interleukin-1 in vitroNew developments in osteoarthritis. Posttraumatic osteoarthritis: pathogenesis and pharmacological treatment options.Blocking of interleukin-17 during reactivation of experimental arthritis prevents joint inflammation and bone erosion by decreasing RANKL and interleukin-1.Factors Expressed in an Animal Model of Anteroinferior Glenohumeral Instability.The effect of platelet-rich plasma formulations and blood products on human synoviocytes: implications for intra-articular injury and therapy.
P2860
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P2860
TNF alpha and IL-1 beta are separate targets in chronic arthritis.
description
1999 nî lūn-bûn
@nan
1999 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
name
TNF alpha and IL-1 beta are separate targets in chronic arthritis.
@ast
TNF alpha and IL-1 beta are separate targets in chronic arthritis.
@en
type
label
TNF alpha and IL-1 beta are separate targets in chronic arthritis.
@ast
TNF alpha and IL-1 beta are separate targets in chronic arthritis.
@en
prefLabel
TNF alpha and IL-1 beta are separate targets in chronic arthritis.
@ast
TNF alpha and IL-1 beta are separate targets in chronic arthritis.
@en
P2093
P1476
TNF alpha and IL-1 beta are separate targets in chronic arthritis
@en
P2093
F A van de Loo
L A Joosten
W B van den Berg
P304
P433
6 Suppl 18
P577
1999-11-01T00:00:00Z