Mechanism for prevention of alcohol-induced liver injury by dietary methyl donors. - Wikidata - awgldk - TriplyDB

Mechanism for prevention of alcohol-induced liver injury by dietary methyl donors.

Mechanism for prevention of alcohol-induced liver injury by dietary methyl donors.

http://www.wikidata.org/entity/Q33792440

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Relationships among alcoholic liver disease, antioxidants, and antioxidant enzymes Rapid Elimination of Blood Alcohol Using Erythrocytes: Mathematical Modeling and In Vitro Study.Methyl-donor supplementation in obese mice prevents the progression of NAFLD, activates AMPK and decreases acyl-carnitine levels.Betaine supplementation in maternal diet modulates the epigenetic regulation of hepatic gluconeogenic genes in neonatal piglets.Alcoholic liver disease patients treated with S-adenosyl-L-methionine: an in-depth look at liver morphologic data comparing pre and post treatment liver biopsies.Redox signaling and the innate immune system in alcoholic liver disease.Betaine feeding prevents the blood alcohol cycle in rats fed alcohol continuously for 1 month using the rat intragastric tube feeding model.Betaine treatment attenuates chronic ethanol-induced hepatic steatosis and alterations to the mitochondrial respiratory chain proteome Multiscale analysis of the murine intestine for modeling human diseases.S-adenosylmethionine decreases the peak blood alcohol levels 3 h after an acute bolus of ethanol by inducing alcohol metabolizing enzymes in the liver.Methylthioadenosine (MTA) Regulates Liver Cells Proteome and Methylproteome: Implications in Liver Biology and Disease Mitochondria and Oxidative Stress in the Cardiorenal Metabolic Syndrome.Choline and methionine differentially alter methyl carbon metabolism in bovine neonatal hepatocytes.Trichloroethylene: Mechanistic, epidemiologic and other supporting evidence of carcinogenic hazard.A mouse model of alcoholic liver fibrosis-associated acute kidney injury identifies key molecular pathways.Oxidative stress and paraoxonase activity in experimental selenosis: effects of betaine administration.Essential amino acid supplementation decreases liver damage induced by chronic ethanol consumption in rats.

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Mechanism for prevention of alcohol-induced liver injury by dietary methyl donors.

http://www.wikidata.org/entity/Q33792440

description

2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Mechanism for prevention of alcohol-induced liver injury by dietary methyl donors.

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Mechanism for prevention of alcohol-induced liver injury by dietary methyl donors.

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Mechanism for prevention of alcohol-induced liver injury by dietary methyl donors.

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Mechanism for prevention of alcohol-induced liver injury by dietary methyl donors.

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Mechanism for prevention of alcohol-induced liver injury by dietary methyl donors.

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Mechanism for prevention of alcohol-induced liver injury by dietary methyl donors.

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Toxicological Sciences

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Mechanism for prevention of alcohol-induced liver injury by dietary methyl donors.

@en

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Blair U Bradford

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Christine L Powell

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Christopher Patrick Craig

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Dennis R Koop

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Igor P Pogribny

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Ivan Rusyn

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Lisa Bleyle

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Masato Tsuchiya

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Stepan Melnyk

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Takeki Uehara

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P2860

Cloning and characterization of PIMT, a protein with a methyltransferase domain, which interacts with and enhances nuclear receptor coactivator PRIP function

Methionine deficiency and hepatic injury in a dietary steatohepatitis model

Hyperhomocysteinemia in chronic alcoholism: correlation with folate, vitamin B-12, and vitamin B-6 status

Steatohepatitis: a tale of two "hits"?

Development of an intragastric enteral model in the mouse: studies of alcohol-induced liver disease using knockout technology.

Genotypic and phenotypic characterization of a putative tumor susceptibility gene, GNMT, in liver cancer.

Effects of oral S-adenosyl-L-methionine on hepatic glutathione in patients with liver disease.

Methionine adenosyltransferase 1A knockout mice are predisposed to liver injury and exhibit increased expression of genes involved in proliferation

Alcohol-induced S-adenosylhomocysteine accumulation in the liver sensitizes to TNF hepatotoxicity: possible involvement of mitochondrial S-adenosylmethionine transport.

CYP2E1 and oxidative liver injury by alcohol.

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131-139

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scholarly article

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10.1093/TOXSCI/KFQ031

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1

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115

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David W. Threadgill

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2010-01-29T00:00:00Z

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41191245

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20118189

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2855354

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