IL-4 signaling drives a unique arginase+/IL-1β+ microglia phenotype and recruits macrophages to the inflammatory CNS: consequences of age-related deficits in IL-4Rα after traumatic spinal cord injury.

IL-4 signaling drives a unique arginase+/IL-1β+ microglia phenotype and recruits macrophages to the inflammatory CNS: consequences of age-related deficits in IL-4Rα after traumatic spinal cord injury.

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http://www.wikidata.org/entity/Q33801629

Q33801629

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Cellular and Molecular Mechanisms Underpinning Macrophage Activation during Remyelination Targeting innate immunity for neurodegenerative disorders of the central nervous system Role of microglial m1/m2 polarization in relapse and remission of psychiatric disorders and diseases Microglial priming and enhanced reactivity to secondary insult in aging, and traumatic CNS injury, and neurodegenerative disease Neuroimmune and Neuropathic Responses of Spinal Cord and Dorsal Root Ganglia in Middle Age Azithromycin drives alternative macrophage activation and improves recovery and tissue sparing in contusion spinal cord injury Mechanisms of long-term cognitive dysfunction of sepsis: from blood-borne leukocytes to glial cells Regulatory T cells in central nervous system injury: a double-edged sword Interactions of innate and adaptive immunity in brain development and function.Age decreases macrophage IL-10 expression: Implications for functional recovery and tissue repair in spinal cord injury Tissue macrophages: heterogeneity and functions.Proteomic anaysis of aged microglia: shifts in transcription, bioenergetics, and nutrient response.Low-level laser facilitates alternatively activated macrophage/microglia polarization and promotes functional recovery after crush spinal cord injury in rats.The Pathophysiological Role of Microglia in Dynamic Surveillance, Phagocytosis and Structural Remodeling of the Developing CNS.CD93 and GIPC expression and localization during central nervous system inflammation.IL-4 Knock Out Mice Display Anxiety-Like Behavior Call Off the Dog(ma): M1/M2 Polarization Is Concurrent following Traumatic Brain Injury.Microglia Transcriptome Changes in a Model of Depressive Behavior after Immune Challenge.Differential Transcriptome Networks between IDO1-Knockout and Wild-Type Mice in Brain Microglia and Macrophages.Microglia in the TBI brain: The good, the bad, and the dysregulated.Sequential activation of microglia and astrocyte cytokine expression precedes increased Iba-1 or GFAP immunoreactivity following systemic immune challenge.Cyclic AMP is a key regulator of M1 to M2a phenotypic conversion of microglia in the presence of Th2 cytokines Lipoxin A4 inhibits microglial activation and reduces neuroinflammation and neuropathic pain after spinal cord hemisection.The immunomodulator decoy receptor 3 improves locomotor functional recovery after spinal cord injury.The Alarmin HMGB1 Mediates Age-Induced Neuroinflammatory Priming Evidence for an Age-Dependent Decline in Axon Regeneration in the Adult Mammalian Central Nervous System.Predictive screening of M1 and M2 macrophages reveals the immunomodulatory effectiveness of post spinal cord injury azithromycin treatment Neuroinflammation as Fuel for Axonal Regeneration in the Injured Vertebrate Central Nervous System.TIR-Domain-Containing Adapter-Inducing Interferon-β (TRIF) Is Essential for MPTP-Induced Dopaminergic Neuroprotection via Microglial Cell M1/M2 Modulation.Microglia and neuroprotection.Microglial brain region-dependent diversity and selective regional sensitivities to aging.A Comparative Study of Three Different Types of Stem Cells for Treatment of Rat Spinal Cord Injury.Microglia in central nervous system repair after injury.Immunosenescence of microglia and macrophages: impact on the ageing central nervous system.Characterization of inflammatory markers and transcriptome profiles of differentially activated embryonic stem cell-derived microglia.Tissue Engineering Approaches to Modulate the Inflammatory Milieu following Spinal Cord Injury.Microglia Function in the Normal Brain.Microglia and Monocyte-Derived Macrophages in Stroke Microglia Priming with Aging and Stress.The age factor in axonal repair after spinal cord injury: A focus on neuron-intrinsic mechanisms.

P2860

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P2860

IL-4 signaling drives a unique arginase+/IL-1β+ microglia phenotype and recruits macrophages to the inflammatory CNS: consequences of age-related deficits in IL-4Rα after traumatic spinal cord injury.

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