TNFA deletion alters apoptosis as well as caspase 3 and 4 expression during otitis media. - Wikidata - awgldk - TriplyDB

TNFA deletion alters apoptosis as well as caspase 3 and 4 expression during otitis media.

TNFA deletion alters apoptosis as well as caspase 3 and 4 expression during otitis media.

http://www.wikidata.org/entity/Q33804517

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Panel 4: Recent advances in otitis media in molecular biology, biochemistry, genetics, and animal models.Panel 8: Complications and sequelae.Panel 3: Recent advances in anatomy, pathology, and cell biology in relation to otitis media pathogenesis.Ecrg4 attenuates the inflammatory proliferative response of mucosal epithelial cells to infection The inflammasome adaptor ASC contributes to multiple innate immune processes in the resolution of otitis media.Peptides actively transported across the tympanic membrane: Functional and structural properties Panel 3: Genetics and Precision Medicine of Otitis Media.Repurposing L-Menthol for Systems Medicine and Cancer Therapeutics? L-Menthol Induces Apoptosis through Caspase 10 and by Suppressing HSP90 Cytokine responses in the common cold and otitis media.What have we learned from murine models of otitis media?Innate Immunity: Orchestrating Inflammation and Resolution of Otitis Media.

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TNFA deletion alters apoptosis as well as caspase 3 and 4 expression during otitis media.

http://www.wikidata.org/entity/Q33804517

description

2011 nî lūn-bûn

@nan

2011 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հունվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

TNFA deletion alters apoptosis as well as caspase 3 and 4 expression during otitis media.

@ast

TNFA deletion alters apoptosis as well as caspase 3 and 4 expression during otitis media.

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type

label

TNFA deletion alters apoptosis as well as caspase 3 and 4 expression during otitis media.

@ast

TNFA deletion alters apoptosis as well as caspase 3 and 4 expression during otitis media.

@en

prefLabel

TNFA deletion alters apoptosis as well as caspase 3 and 4 expression during otitis media.

@ast

TNFA deletion alters apoptosis as well as caspase 3 and 4 expression during otitis media.

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TNFA deletion alters apoptosis as well as caspase 3 and 4 expression during otitis media.

@en

P2093

Allen F Ryan

http://www.w3.org/2001/XMLSchema#string

Anke Leichtle

http://www.w3.org/2001/XMLSchema#string

David Broide

http://www.w3.org/2001/XMLSchema#string

Holger Sudhoff

http://www.w3.org/2001/XMLSchema#string

Jacob Husseman

http://www.w3.org/2001/XMLSchema#string

Joerg Ebmeyer

http://www.w3.org/2001/XMLSchema#string

Kwang Pak

http://www.w3.org/2001/XMLSchema#string

Michelle Hernandez

http://www.w3.org/2001/XMLSchema#string

Stephen I Wasserman

http://www.w3.org/2001/XMLSchema#string

Umay Ebmeyer

http://www.w3.org/2001/XMLSchema#string

P2860

Identification of a new member of the tumor necrosis factor family and its receptor, a human ortholog of mouse GITR

BAFF, a novel ligand of the tumor necrosis factor family, stimulates B cell growth

TAJ, a novel member of the tumor necrosis factor receptor family, activates the c-Jun N-terminal kinase pathway and mediates caspase-independent cell death

A novel TNF receptor family member binds TWEAK and is implicated in angiogenesis

LIGHT, a member of the tumor necrosis factor ligand superfamily, prevents tumor necrosis factor-alpha-mediated human primary hepatocyte apoptosis, but not Fas-mediated apoptosis

Identification and characterization of a new member of the TNF family that induces apoptosis

Identification of a human OX-40 ligand, a costimulator of CD4+ T cells with homology to tumor necrosis factor

Molecular and biological characterization of human 4-1BB and its ligand

Induction of apoptosis by Apo-2 ligand, a new member of the tumor necrosis factor cytokine family

LIGHT, a novel ligand for lymphotoxin beta receptor and TR2/HVEM induces apoptosis and suppresses in vivo tumor formation via gene transfer

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