Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging. - Wikidata - awgldk - TriplyDB

Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging.

Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging.

http://www.wikidata.org/entity/Q33806916

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Fatty old hearts: role of cardiac lipotoxicity in age-related cardiomyopathy The Dual Function of Reactive Oxygen/Nitrogen Species in Bioenergetics and Cell Death: The Role of ATP Synthase Biochemical Genetic Pathways that Modulate Aging in Multiple Species Running forward: new frontiers in endurance exercise biology Sepsis-induced Cardiac Mitochondrial Damage and Potential Therapeutic Interventions in the Elderly Axis of ageing: telomeres, p53 and mitochondria Genetics and epigenetics of aging and longevity Somatic mutations in aging, cancer and neurodegeneration Mitochondria and cardiovascular aging Mitochondrial oxidative stress in aging and healthspan Mitochondrial inverted repeats strongly correlate with lifespan: mtDNA inversions and aging Oxidative stress is not a major contributor to somatic mitochondrial DNA mutations Reduced expression of MYC increases longevity and enhances healthspan.Cardiomyocyte-specific expression of lamin a improves cardiac function in Lmna-/- mice dJun and Vri/dNFIL3 are major regulators of cardiac aging in Drosophila Quality control systems in cardiac aging Growth differentiation factor 11 is a circulating factor that reverses age-related cardiac hypertrophy Interplay between oxidant species and energy metabolism Mitochondrial Metabolism in Aging Heart Mitochondrial dysfunction in cardiac aging Mitochondria and oxidative stress in heart aging Regulation of signal transduction by reactive oxygen species in the cardiovascular system Paradoxical Roles of Antioxidant Enzymes: Basic Mechanisms and Health Implications Staying young at heart: autophagy and adaptation to cardiac aging 17β-Estradiol, aging, inflammation, and the stress response in the female heart A novel intracellular isoform of matrix metalloproteinase-2 induced by oxidative stress activates innate immunity Mitochondrial genome instability and ROS enhance intestinal tumorigenesis in APC(Min/+) mice Immune-inflammatory dysregulation modulates the incidence of progressive fibrosis and diastolic stiffness in the aging heart Disruption of protein kinase A in mice enhances healthy aging.Ectopic catalase expression in mitochondria by adeno-associated virus enhances exercise performance in mice.Disruption of the mGsta4 gene increases life span of C57BL mice Arginine metabolism by macrophages promotes cardiac and muscle fibrosis in mdx muscular dystrophy.Update on the oxidative stress theory of aging: does oxidative stress play a role in aging or healthy aging?Mitohormesis: Promoting Health and Lifespan by Increased Levels of Reactive Oxygen Species (ROS).Altered proteome turnover and remodeling by short-term caloric restriction or rapamycin rejuvenate the aging heart.Monoamine Oxidases, Oxidative Stress, and Altered Mitochondrial Dynamics in Cardiac Ageing Monoamine oxidases as sources of oxidants in the heart The mitochondrial unfolded protein response and increased longevity: cause, consequence, or correlation?SIRT3 deficiency exacerbates ischemia-reperfusion injury: implication for aged hearts.Cardiac aging in mice and humans: the role of mitochondrial oxidative stress

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P2860

Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging.

http://www.wikidata.org/entity/Q33806916

description

2009 nî lūn-bûn

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2009 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2009 թվականի մայիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging.

@ast

Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging.

@en

type

label

Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging.

@ast

Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging.

@en

prefLabel

Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging.

@ast

Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging.

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Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging

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Daniela M Tomazela

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George M Martin

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Katherine Gollahon

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Lawrence A Loeb

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Marc Vermulst

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Mary J Emond

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Warren C Ladiges

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P2860

The transcription factor GATA4 is activated by extracellular signal-regulated kinase 1- and 2-mediated phosphorylation of serine 105 in cardiomyocytes

Mitochondria, oxidants, and aging

Extension of murine life span by overexpression of catalase targeted to mitochondria

Regulation of cardiac hypertrophy by intracellular signalling pathways

Suppression of reactive oxygen species and neurodegeneration by the PGC-1 transcriptional coactivators

Abnormalities of calcium cycling in the hypertrophied and failing heart.

Measuring population health: a review of indicators.

New concepts in diastolic dysfunction and diastolic heart failure: Part II: causal mechanisms and treatment.

Arterial and cardiac aging: major shareholders in cardiovascular disease enterprises: Part II: the aging heart in health: links to heart disease.

Arterial and cardiac aging: major shareholders in cardiovascular disease enterprises: Part III: cellular and molecular clues to heart and arterial aging.

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Luis F. Santana

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19451351

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