The macrophage in HIV-1 infection: from activation to deactivation? - Wikidata - awgldk - TriplyDB

The macrophage in HIV-1 infection: from activation to deactivation?

The macrophage in HIV-1 infection: from activation to deactivation?

http://www.wikidata.org/entity/Q33810779

about

The macrophage: a therapeutic target in HIV-1 infection HIV-associated neuropathogenesis: a systems biology perspective for modeling and therapy Antiviral regulation in porcine monocytic cells at different activation states Nuclear exportin receptor CAS regulates the NPI-1-mediated nuclear import of HIV-1 Vpr Discordance in CD4+T-cell levels and viral loads with co-occurrence of elevated peripheral TNF-α and IL-4 in newly diagnosed HIV-TB co-infected cases Interaction between innate immunity and porcine reproductive and respiratory syndrome virus.Cellular activation and intracellular HCV load in peripheral blood monocytes isolated from HCV monoinfected and HIV-HCV coinfected patients Macrophage polarization in inflammatory diseases.Functional proteomic analysis for regulatory T cell surveillance of the HIV-1-infected macrophage.The CSF Immune Response in HIV-1-Associated Cryptococcal Meningitis: Macrophage Activation, Correlates of Disease Severity, and Effect of Antiretroviral Therapy Innate immune recognition and activation during HIV infection.Triggering TLR2, -3, -4, -5, and -8 reinforces the restrictive nature of M1- and M2-polarized macrophages to HIV.RIG-I-mediated antiviral signaling is inhibited in HIV-1 infection by a protease-mediated sequestration of RIG-I.Hepatitis C virus core protein enhances HIV-1 replication in human macrophages through TLR2, JNK, and MEK1/2-dependent upregulation of TNF-α and IL-6.Mycobacterial antigen driven activation of CD14++CD16- monocytes is a predictor of tuberculosis-associated immune reconstitution inflammatory syndrome Macrophages and their relevance in Human Immunodeficiency Virus Type I infection.HIV-1 phylogenetic analysis shows HIV-1 transits through the meninges to brain and peripheral tissues.HIV-1 infection inhibits cytokine production in human thymic macrophages.Multiple low-dose challenges in a rhesus macaque AIDS vaccine trial result in an evolving host response that affects protective outcome Optimal structural design of mannosylated nanocarriers for macrophage targeting.Registered bioimaging of nanomaterials for diagnostic and therapeutic monitoring.Macrophage Resistance to HIV-1 Infection Is Enhanced by the Neuropeptides VIP and PACAP.Drug induced increases in CNS dopamine alter monocyte, macrophage and T cell functions: implications for HAND.Mechanisms of alcohol-mediated hepatotoxicity in human-immunodeficiency-virus-infected patients.Pulsed stable isotope labeling of amino acids in cell culture uncovers the dynamic interactions between HIV-1 and the monocyte-derived macrophage.The effect of HAART-induced HIV suppression on circulating markers of inflammation and immune activation.Methamphetamine and HIV-1 gp120 effects on lipopolysaccharide stimulated matrix metalloproteinase-9 production by human monocyte-derived macrophages.Genome-wide analysis of antiviral signature genes in porcine macrophages at different activation statuses.SHP-1-dependent macrophage differentiation exacerbates virus-induced myositis Eradication of HIV-1 from the macrophage reservoir: an uncertain goal?Viral determinants of HIV-1 macrophage tropism.Association of IL-10-promoter genetic variants with the rate of CD4 T-cell loss, IL-10 plasma levels, and breadth of cytotoxic T-cell lymphocyte response during chronic HIV-1 infection.HIV Blocks Interferon Induction in Human Dendritic Cells and Macrophages by Dysregulation of TBK1 Macrophage Polarization in Virus-Host Interactions Morphine and galectin-1 modulate HIV-1 infection of human monocyte-derived macrophages.Macrophage polarization: convergence point targeted by mycobacterium tuberculosis and HIV.Low prevalence of CCR5-Δ32, CCR2-64I and SDF1-3'A alleles in the Baiga and Gond tribes of Central India.Alterations in P-Glycoprotein Expression and Function Between Macrophage Subsets.Porphyromonas gingivalis-mediated signaling through TLR4 mediates persistent HIV infection of primary macrophages.Nanoparticle based galectin-1 gene silencing, implications in methamphetamine regulation of HIV-1 infection in monocyte derived macrophages

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P2860

The macrophage in HIV-1 infection: from activation to deactivation?

http://www.wikidata.org/entity/Q33810779

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

The macrophage in HIV-1 infection: from activation to deactivation?

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The macrophage in HIV-1 infection: from activation to deactivation?

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type

label

The macrophage in HIV-1 infection: from activation to deactivation?

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The macrophage in HIV-1 infection: from activation to deactivation?

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prefLabel

The macrophage in HIV-1 infection: from activation to deactivation?

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The macrophage in HIV-1 infection: from activation to deactivation?

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The macrophage in HIV-1 infection: from activation to deactivation?

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Audrey Varin

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Georges Herbein

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Creative Commons Attribution 2.0 Generic

P2860

The interferon response inhibits HIV particle production by induction of TRIM22

Blockade of chemokine-induced signalling inhibits CCR5-dependent HIV infection in vitro without blocking gp120/CCR5 interaction

TNF-alpha inhibits HIV-1 replication in peripheral blood monocytes and alveolar macrophages by inducing the production of RANTES and decreasing C-C chemokine receptor 5 (CCR5) expression

Retrovirally mediated IFN-beta transduction of macrophages induces resistance to HIV, correlated with up-regulation of RANTES production and down-regulation of C-C chemokine receptor-5 expression

HIV-1 Entry Cofactor: Functional cDNA Cloning of a Seven-Transmembrane, G Protein-Coupled Receptor

CD4+ T cell depletion during all stages of HIV disease occurs predominantly in the gastrointestinal tract

Primary HIV-1 infection is associated with preferential depletion of CD4+ T lymphocytes from effector sites in the gastrointestinal tract

Identification of a major co-receptor for primary isolates of HIV-1

Interleukin-10 and the interleukin-10 receptor

Monocyte chemotactic protein-2 activates CCR5 and blocks CD4/CCR5-mediated HIV-1 entry/replication

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scholarly article

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10.1186/1742-4690-7-33

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7

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Georges Herbein

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2010-04-09T00:00:00Z

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43097717

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1008565962

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20380696

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hemic and immune systems

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2859752

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