Fibrinolysis greater than 3% is the critical value for initiation of antifibrinolytic therapy.
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Viscoelastic Methods of Blood Clotting Assessment - A Multidisciplinary ReviewCRASH-2 Study of Tranexamic Acid to Treat Bleeding in Trauma Patients: A Controversy Fueled by Science and Social MediaPoint of care and factor concentrate-based coagulation algorithmsAn International Normalized Ratio-Based Definition of Acute Traumatic Coagulopathy Is Associated With Mortality, Venous Thromboembolism, and Multiple Organ Failure After InjuryApplying results from clinical trials: tranexamic acid in trauma patientsEffect of thromboelastography (TEG®) and rotational thromboelastometry (ROTEM®) on diagnosis of coagulopathy, transfusion guidance and mortality in trauma: descriptive systematic reviewFibrinolytic shutdown: fascinating theory but randomized controlled trial data are needed.Taking the Blood Bank to the Field: The Design and Rationale of the Prehospital Air Medical Plasma (PAMPer) TrialTranexamic acid for treatment and prophylaxis of bleeding and hyperfibrinolysis.Goal-directed Hemostatic Resuscitation of Trauma-induced Coagulopathy: A Pragmatic Randomized Clinical Trial Comparing a Viscoelastic Assay to Conventional Coagulation AssaysTrauma-Induced Coagulopathy: An Institution's 35 Year Perspective on Practice and Research.TEG®- or ROTEM®-based individualized goal-directed coagulation algorithms: don't wait--act now!Hyperfibrinolysis, physiologic fibrinolysis, and fibrinolysis shutdown: the spectrum of postinjury fibrinolysis and relevance to antifibrinolytic therapyHemolysis exacerbates hyperfibrinolysis, whereas platelolysis shuts down fibrinolysis: evolving concepts of the spectrum of fibrinolysis in response to severe injury.Plasma is the physiologic buffer of tissue plasminogen activator-mediated fibrinolysis: rationale for plasma-first resuscitation after life-threatening hemorrhage.Postinjury fibrinolysis shutdown: Rationale for selective tranexamic acidPolymorphisms on PAI-1 and ACE genes in association with fibrinolytic bleeding after on-pump cardiac surgery.Thromboelastography: A Practice Summary for Nurse Practitioners Treating HemorrhageDesign of the Study of Tranexamic Acid during Air Medical Prehospital Transport (STAAMP) Trial: Addressing the Knowledge Gaps.The "Death Diamond": Rapid thrombelastography identifies lethal hyperfibrinolysis.Overwhelming tPA release, not PAI-1 degradation, is responsible for hyperfibrinolysis in severely injured trauma patients.Acute Fibrinolysis Shutdown after Injury Occurs Frequently and Increases Mortality: A Multicenter Evaluation of 2,540 Severely Injured Patients.Viscoelastic measurements of platelet function, not fibrinogen function, predicts sensitivity to tissue-type plasminogen activator in trauma patients.Thromboelastographic Evaluation of Dogs with Acute Liver DiseasePost-translational oxidative modification of fibrinogen is associated with coagulopathy after traumatic injury.Targeted Thromboelastographic (TEG) Blood Component and Pharmacologic Hemostatic Therapy in Traumatic and Acquired Coagulopathy.The challenge in management of hemorrhagic shock in trauma.State of the art: massive transfusion.Global assays of hemostasis.Management of traumatic haemorrhage--the European perspective.Tranexamic acid is associated with increased mortality in patients with physiological fibrinolysis.Hemoglobin-based oxygen carriers promote systemic hyperfibrinolysis that is both dependent and independent of plasmin.Management of Trauma-Induced Coagulopathy with Thrombelastography.Rapid thrombelastography thresholds for goal-directed resuscitation of patients at risk for massive transfusion.Tranexamic Acid Use in Prehospital Uncontrolled Hemorrhage.Heterogeneous phase fibrinolysis rates by damped oscillation rheometry.Effect of solvent/detergent-treated pooled plasma on fibrinolysis in reconstituted whole blood.Decreasing the Use of Damage Control Laparotomy in Trauma: A Quality Improvement Project.[Uncritical use of tranexamic acid in trauma patients : Do no further harm!][Early viscoelasticity-based coagulation therapy for severely injured bleeding patients: Report of the consensus group on the consensus conference 2014 for formulation of S2k guidelines].
P2860
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P2860
Fibrinolysis greater than 3% is the critical value for initiation of antifibrinolytic therapy.
description
2013 nî lūn-bûn
@nan
2013 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Fibrinolysis greater than 3% i ...... n of antifibrinolytic therapy.
@ast
Fibrinolysis greater than 3% i ...... n of antifibrinolytic therapy.
@en
type
label
Fibrinolysis greater than 3% i ...... n of antifibrinolytic therapy.
@ast
Fibrinolysis greater than 3% i ...... n of antifibrinolytic therapy.
@en
prefLabel
Fibrinolysis greater than 3% i ...... n of antifibrinolytic therapy.
@ast
Fibrinolysis greater than 3% i ...... n of antifibrinolytic therapy.
@en
P2093
P2860
P1476
Fibrinolysis greater than 3% i ...... on of antifibrinolytic therapy
@en
P2093
Angela Sauaia
Arsen Ghasabyan
Christopher C Silliman
Christopher R Ramos
Ernest E Moore
John R Stringham
Michael P Chapman
Theresa L Chin
P2860
P304
961-7; discussion 967
P356
10.1097/TA.0B013E3182AA9C9F
P577
2013-12-01T00:00:00Z