Differential Alteration in Expression of Striatal GABAAR Subunits in Mouse Models of Huntington's Disease - Wikidata - awgldk - TriplyDB

Differential Alteration in Expression of Striatal GABAAR Subunits in Mouse Models of Huntington's Disease

Differential Alteration in Expression of Striatal GABAAR Subunits in Mouse Models of Huntington's Disease

http://www.wikidata.org/entity/Q33813021

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Striatal Direct and Indirect Pathway Output Structures are Differentially Altered in Mouse Models of Huntington's Disease.The adjustment of γ-aminobutyric acidA tonic subunits in Huntington's disease: from transcription to translation to synaptic levels into the neostriatum.The Tiny for the Biggest Answers in Huntington's Disease

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Differential Alteration in Expression of Striatal GABAAR Subunits in Mouse Models of Huntington's Disease

http://www.wikidata.org/entity/Q33813021

description

2017 nî lūn-bûn

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2017 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2017 թվականի հունիսին հրատարակված գիտական հոդված

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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name

Differential Alteration in Exp ...... Models of Huntington's Disease

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Differential Alteration in Exp ...... Models of Huntington's Disease

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Differential Alteration in Exp ...... Models of Huntington's Disease

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Differential Alteration in Exp ...... Models of Huntington's Disease

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Differential Alteration in Exp ...... Models of Huntington's Disease

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Differential Alteration in Exp ...... Models of Huntington's Disease

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FNMOL.2017.00198

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Frontiers in molecular neuroscience

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Differential Alteration in Exp ...... Models of Huntington's Disease

@en

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Christophe Halgand

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Frédérique Masmejean

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Gilles Courtand

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Laura Cardoit

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Margot Tertrais

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Maurice Garret

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Thierry Leste-Lasserre

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Yoon H Cho

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Zhuowei Du

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P2860

GABRB3 mutation, G32R, associated with childhood absence epilepsy alters α1β3γ2L γ-aminobutyric acid type A (GABAA) receptor expression and channel gating

GABA A receptors: subtypes provide diversity of function and pharmacology

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Possible involvement of self-defense mechanisms in the preferential vulnerability of the striatum in Huntington's disease

Identification of a new genomic hot spot of evolutionary diversification of protein function

Correlations of behavioral deficits with brain pathology assessed through longitudinal MRI and histopathology in the R6/1 mouse model of Huntington's disease

The MIQE guidelines: minimum information for publication of quantitative real-time PCR experiments

GABAergic circuits mediate the reinforcement-related signals of striatal cholinergic interneurons

Neuroligin 2 drives postsynaptic assembly at perisomatic inhibitory synapses through gephyrin and collybistin

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198

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scholarly article

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10.3389/FNMOL.2017.00198

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10

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2017-06-20T00:00:00Z

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28676743

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corpus striatum

Huntington disease

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5476702

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