Phenotypic consequences of rearranging the P, M, and G genes of vesicular stomatitis virus.
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Vesicular stomatitis virus as a flexible platform for oncolytic virotherapy against cancerEvaluating Replication-Defective Vesicular Stomatitis Virus as a Vaccine VehicleIn Vitro and In Vivo Attenuation of Vesicular Stomatitis Virus (VSV) by Phosphoprotein Deletion.Image-guided modeling of virus growth and spread.Second-site mutations selected in transcriptional regulatory sequences compensate for engineered mutations in the vesicular stomatitis virus nucleocapsid proteinA temperature sensitive VSV identifies L protein residues that affect transcription but not replication.Selection for gene junction sequences important for VSV transcriptionModel-based design of growth-attenuated virusesComputational fitness landscape for all gene-order permutations of an RNA virus.Attenuation of vesicular stomatitis virus encephalitis through microRNA targeting.Neurovirulence and immunogenicity of attenuated recombinant vesicular stomatitis viruses in nonhuman primates.Rearrangement of the genes of vesicular stomatitis virus eliminates clinical disease in the natural host: new strategy for vaccine development.A novel, live-attenuated vesicular stomatitis virus vector displaying conformationally intact, functional HIV-1 envelope trimers that elicits potent cellular and humoral responses in mice.Gene order rearrangement of the M gene in the rabies virus leads to slower replication.Overexpression of the rabies virus glycoprotein results in enhancement of apoptosis and antiviral immune response.Identification of an upstream sequence element required for vesicular stomatitis virus mRNA transcriptionAdding genes to the RNA genome of vesicular stomatitis virus: positional effects on stability of expression.Coronaviruses maintain viability despite dramatic rearrangements of the strictly conserved genome organizationGenetically engineered vesicular stomatitis virus in gene therapy: application for treatment of malignant disease.Recombinant respiratory syncytial virus with the G and F genes shifted to the promoter-proximal positionsSecond-generation rabies virus-based vaccine vectors expressing human immunodeficiency virus type 1 gag have greatly reduced pathogenicity but are highly immunogenicVesicular stomatitis virus as a vector to deliver virus-like particles of human norovirus: a new vaccine candidate against an important noncultivable virus.Synergistic attenuation of vesicular stomatitis virus by combination of specific G gene truncations and N gene translocations.Neurovirulence properties of recombinant vesicular stomatitis virus vectors in non-human primatesLevel of gene expression is a major determinant of protein evolution in the viral order Mononegavirales.Vaccination With a Highly Attenuated Recombinant Vesicular Stomatitis Virus Vector Protects Against Challenge With a Lethal Dose of Ebola Virus.Evolutionary reversion of live viral vaccines: Can genetic engineering subdue it?Vaccines to prevent severe acute respiratory syndrome coronavirus-induced disease.Computation, prediction, and experimental tests of fitness for bacteriophage T7 mutants with permuted genomes.Generation of a More Immunogenic Measles Vaccine by Increasing Its Hemagglutinin Expression.Immunogenicity of self tumor associated proteins is enhanced through protein truncation.Fitness analyses of vesicular stomatitis strains with rearranged genomes reveal replicative disadvantages.Identification of viral genomic elements responsible for rabies virus neuroinvasiveness.Phenotypic Consequences In vivo and In vitro of Rearranging the P Gene of RABV HEP-Flury.Unique safety issues associated with virus-vectored vaccines: Potential for and theoretical consequences of recombination with wild type virus strains.The viral transcription group determines the HLA class I cellular immune response against human respiratory syncytial virus.Recombinant rinderpest vaccines expressing membrane-anchored proteins as genetic markers: evidence of exclusion of marker protein from the virus envelope.Moving the glycoprotein gene of vesicular stomatitis virus to promoter-proximal positions accelerates and enhances the protective immune responseMany nonmammalian cells exhibit postentry blocks to transduction by gammaretroviruses pseudotyped with various viral envelopes, including vesicular stomatitis virus G glycoprotein.Host switching in Lyssavirus history from the Chiroptera to the Carnivora orders.
P2860
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P2860
Phenotypic consequences of rearranging the P, M, and G genes of vesicular stomatitis virus.
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Phenotypic consequences of rea ...... of vesicular stomatitis virus.
@ast
Phenotypic consequences of rea ...... of vesicular stomatitis virus.
@en
type
label
Phenotypic consequences of rea ...... of vesicular stomatitis virus.
@ast
Phenotypic consequences of rea ...... of vesicular stomatitis virus.
@en
prefLabel
Phenotypic consequences of rea ...... of vesicular stomatitis virus.
@ast
Phenotypic consequences of rea ...... of vesicular stomatitis virus.
@en
P2093
P2860
P1433
P1476
Phenotypic consequences of rea ...... of vesicular stomatitis virus.
@en
P2093
B Flanagan
C R Pringle
V P Perepelitsa
P2860
P304
P577
1999-06-01T00:00:00Z