Epistasis between COMT and MTHFR in maternal-fetal dyads increases risk for preeclampsia.
about
Associations between maternal genotypes and metabolites implicated in congenital heart defectsMalignant cancer and invasive placentation: A case for positive pleiotropy between endometrial and malignancy phenotypesA Comparison of 2-Methoxyestradiol Value in Women with Severe Preeclampsia Versus Normotensive Pregnancy.DNA methylation is altered in maternal blood vessels of women with preeclampsia.Polymorphisms in catechol-O-methyltransferase modify treatment effects of aspirin on risk of cardiovascular disease.2-methoxyestradiol plasma levels are associated with clinical severity indices and biomarkers of preeclampsia.Catechol-O-methyltransferase (COMT) single nucleotide polymorphisms and haplotypes are not major risk factors for polycystic ovary syndrome.Genetic variation throughout the folate metabolic pathway influences negative symptom severity in schizophreniaProlactin receptor gene polymorphisms are associated with gestational diabetesFetal and maternal genes' influence on gestational age in a quantitative genetic analysis of 244,000 Swedish births.Metabolic pathways involved in 2-methoxyestradiol synthesis and their role in preeclampsiaLevels of key enzymes of methionine-homocysteine metabolism in preeclampsiaA Novel ERAP2 Haplotype Structure in a Chilean Population: Implications for ERAP2 Protein Expression and Preeclampsia Risk.The contribution of genetic and environmental factors to the duration of pregnancy.Protective Role of Maternal P.VAL158MET Catechol-O-Methyltransferase Polymorphism against Early-Onset Preeclampsia and its Complications.Methylenetetrahydrofolate reductase gene C677T, A1298C polymorphisms and pre-eclampsia risk: a meta-analysis.Genetic variants in pre-eclampsia: a meta-analysis.Association of oxidative stress-related genes with idiopathic recurrent miscarriage.Association between COMT Val158Met polymorphism and preeclampsia in the Chinese Han population.The polymorphisms of methionine synthase (MTR) and methionine synthase reductase (MTRR) genes in pathogenesis of preeclampsia.Epistasis among eNOS, MMP-9 and VEGF maternal genotypes in hypertensive disorders of pregnancy.2-Methoxyoestradiol levels and placental catechol-O-methyltransferase expression in patients with late-onset preeclampsia
P2860
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P2860
Epistasis between COMT and MTHFR in maternal-fetal dyads increases risk for preeclampsia.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Epistasis between COMT and MTHFR in maternal-fetal dyads increases risk for preeclampsia.
@ast
Epistasis between COMT and MTHFR in maternal-fetal dyads increases risk for preeclampsia.
@en
type
label
Epistasis between COMT and MTHFR in maternal-fetal dyads increases risk for preeclampsia.
@ast
Epistasis between COMT and MTHFR in maternal-fetal dyads increases risk for preeclampsia.
@en
prefLabel
Epistasis between COMT and MTHFR in maternal-fetal dyads increases risk for preeclampsia.
@ast
Epistasis between COMT and MTHFR in maternal-fetal dyads increases risk for preeclampsia.
@en
P2093
P2860
P1433
P1476
Epistasis between COMT and MTHFR in maternal-fetal dyads increases risk for preeclampsia.
@en
P2093
Jerome F Strauss
Juan P Kusanovic
Lindon J Eaves
Lori D Hill
Timothy P York
P2860
P304
P356
10.1371/JOURNAL.PONE.0016681
P407
P577
2011-01-31T00:00:00Z