Loss of Nkx3.1 expression in bacterial prostatitis: a potential link between inflammation and neoplasia. - Wikidata - awgldk - TriplyDB

Loss of Nkx3.1 expression in bacterial prostatitis: a potential link between inflammation and neoplasia.

Loss of Nkx3.1 expression in bacterial prostatitis: a potential link between inflammation and neoplasia.

http://www.wikidata.org/entity/Q33816335

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Immune Infiltration and Prostate Cancer Loss of androgen receptor-dependent growth suppression by prostate cancer cells can occur independently from acquiring oncogenic addiction to androgen receptor signaling Prostate cancer and inflammation: the evidence.Molecular genetics of prostate cancer: new prospects for old challenges A functional variant in NKX3.1 associated with prostate cancer risk in the Selenium and Vitamin E Cancer Prevention Trial (SELECT).Prostate epithelial stem and progenitor cells.Antioxidant treatment promotes prostate epithelial proliferation in Nkx3.1 mutant mice.Chronic prostatic infection and inflammation by Propionibacterium acnes in a rat prostate infection model.Th1-Th17 cells contribute to the development of uropathogenic Escherichia coli-induced chronic pelvic pain Persistent inflammation leads to proliferative neoplasia and loss of smooth muscle cells in a prostate tumor model TNFα-mediated loss of β-catenin/E-cadherin association and subsequent increase in cell migration is partially restored by NKX3.1 expression in prostate cells Acute bacterial inflammation of the mouse prostate.Bacterial Prostatitis Enhances 2-Amino-1-Methyl-6-Phenylimidazo[4,5-b]Pyridine (PhIP)-Induced Cancer at Multiple Sites Characterization of autoimmune inflammation induced prostate stem cell expansion.Development of animal models underlining mechanistic connections between prostate inflammation and cancer Food Habits, Lifestyle Factors, and Risk of Prostate Cancer in Central Argentina: A Case Control Study Involving Self-Motivated Health Behavior Modifications after Diagnosis.Acupuncture and Traditional Chinese Medicine for the management of a 35-year-old man with chronic prostatitis with chronic pelvic pain syndrome.Loss of the NKX3.1 tumorsuppressor promotes the TMPRSS2-ERG fusion gene expression in prostate cancer.Prostatic inflammation enhances basal-to-luminal differentiation and accelerates initiation of prostate cancer with a basal cell origin.Dietary habits and prostate cancer prevention: a review of observational studies by focusing on South America.Regulating NKX3.1 stability and function: Post-translational modifications and structural determinants.A mouse model of chronic prostatic inflammation using a human prostate cancer-derived isolate of Propionibacterium acnes.NKX3.1 Suppresses TMPRSS2-ERG Gene Rearrangement and Mediates Repair of Androgen Receptor-Induced DNA Damage Functional activation of ATM by the prostate cancer suppressor NKX3.1.Inflammation-mediated abrogation of androgen signaling: an in vitro model of prostate cell inflammation.A human prostatic bacterial isolate alters the prostatic microenvironment and accelerates prostate cancer progression.Non-Cell-Autonomous Regulation of Prostate Epithelial Homeostasis by Androgen Receptor.Inflammation contributes to NKX3.1 loss and augments DNA damage but does not alter the DNA damage response via increased SIRT1 expression.Nkx3.1 controls the DNA repair response in the mouse prostate.Low CD38 Identifies Progenitor-like Inflammation-Associated Luminal Cells that Can Initiate Human Prostate Cancer and Predict Poor Outcome.The inflammatory microenvironment and microbiome in prostate cancer development.The presence of modified nucleosides in extracellular fluids leads to the specific incorporation of 5-chlorocytidine into RNA and modulates the transcription and translation.NK3 homeobox 1 (NKX3.1) up-regulates forkhead box O1 expression in hepatocellular carcinoma and thereby suppresses tumor proliferation and invasion.Inflammatory mechanisms and oxidative stress in prostatitis: the possible role of antioxidant therapy

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P2860

Loss of Nkx3.1 expression in bacterial prostatitis: a potential link between inflammation and neoplasia.

http://www.wikidata.org/entity/Q33816335

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2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Loss of Nkx3.1 expression in b ...... en inflammation and neoplasia.

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Loss of Nkx3.1 expression in b ...... en inflammation and neoplasia.

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Loss of Nkx3.1 expression in b ...... en inflammation and neoplasia.

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AJPATH.2010.080747

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The American Journal of Pathology

P1476

Loss of Nkx3.1 expression in b ...... en inflammation and neoplasia.

@en

P2093

Angelo M De Marzo

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Bora Gurel

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Charles J Bieberich

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Jessica L Hicks

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Laura N Mutton

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May Khalili

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P2860

p63 is a prostate basal cell marker and is required for prostate development

Inflammation and cancer: back to Virchow?

A molecular analysis of prokaryotic and viral DNA sequences in prostate tissue from patients with prostate cancer indicates the presence of multiple and diverse microorganisms

Tissue-specific expression of murine Nkx3.1 in the male urogenital system

p63 is the molecular switch for initiation of an epithelial stratification program

Targeted disruption of the Nkx3.1 gene in mice results in morphogenetic defects of minor salivary glands: parallels to glandular duct morphogenesis in prostate

Roles for Nkx3.1 in prostate development and cancer

Nkx3.1, a murine homolog of Ddrosophila bagpipe, regulates epithelial ductal branching and proliferation of the prostate and palatine glands

Cancer statistics, 2007

Ulcerative colitis and colon cancer: biology and surveillance.

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2259-2268

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10.2353/AJPATH.2010.080747

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2861091

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