3xTgAD mice exhibit altered behavior and elevated Aβ after chronic mild social stress
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Sleep, circadian rhythms, and the pathogenesis of Alzheimer diseaseAltered hypothalamic protein expression in a rat model of Huntington's diseaseSevere Cognitive Dysfunction and Occupational Extremely Low Frequency Magnetic Field Exposure among Elderly Mexican AmericansPosttraumatic stress disorder-like induction elevates β-amyloid levels, which directly activates corticotropin-releasing factor neurons to exacerbate stress responses.Central CRF system perturbation in an Alzheimer's disease knockin mouse model.A ketone ester diet exhibits anxiolytic and cognition-sparing properties, and lessens amyloid and tau pathologies in a mouse model of Alzheimer's diseaseEnvironmental novelty exacerbates stress hormones and Aβ pathology in an Alzheimer's model.Targeting psychologic stress signaling pathways in Alzheimer's disease.Recruiting adaptive cellular stress responses for successful brain ageing.Meditation and vacation effects have an impact on disease-associated molecular phenotypes.The stress response neuropeptide CRF increases amyloid-β production by regulating γ-secretase activityProlonged running, not fluoxetine treatment, increases neurogenesis, but does not alter neuropathology, in the 3xTg mouse model of Alzheimer's disease3xTg-AD mice exhibit an activated central stress axis during early-stage pathology.Pharmacological targeting of CSF1R inhibits microglial proliferation and prevents the progression of Alzheimer's-like pathology.Short-term modern life-like stress exacerbates Aβ-pathology and synapse loss in 3xTg-AD miceChronic mild sleep restriction accentuates contextual memory impairments, and accumulations of cortical Aβ and pTau in a mouse model of Alzheimer's disease.The involvement of homocysteine in stress-induced Aβ precursor protein misprocessing and related cognitive decline in rats.Hyaluronan synthesis by developing cortical neurons in vitro.Targeting Neuroinflammation to Treat Alzheimer's Disease.Actions of Brain-Derived Neurotrophic Factor and Glucocorticoid Stress in Neurogenesis.Exercise and BDNF reduce Aβ production by enhancing α-secretase processing of APP.An Intranasal Formulation of Erythropoietin (Neuro-EPO) Prevents Memory Deficits and Amyloid Toxicity in the APPSwe Transgenic Mouse Model of Alzheimer's Disease.Alzheimer's disease and modern lifestyle: what is the role of stress?The relationship between stress and Alzheimer's disease.Chronic stress as a risk factor for Alzheimer's disease: Roles of microglia-mediated synaptic remodeling, inflammation, and oxidative stress.Amyloid pathology fingerprint differentiates post-traumatic stress disorder and traumatic brain injury.Brain Physiology and Pathophysiology in Mental StressCentral Role of Glucocorticoid Receptors in Alzheimer's Disease and Depression
P2860
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P2860
3xTgAD mice exhibit altered behavior and elevated Aβ after chronic mild social stress
description
2011 nî lūn-bûn
@nan
2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
3xTgAD mice exhibit altered behavior and elevated Aβ after chronic mild social stress
@ast
3xTgAD mice exhibit altered behavior and elevated Aβ after chronic mild social stress
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type
label
3xTgAD mice exhibit altered behavior and elevated Aβ after chronic mild social stress
@ast
3xTgAD mice exhibit altered behavior and elevated Aβ after chronic mild social stress
@en
prefLabel
3xTgAD mice exhibit altered behavior and elevated Aβ after chronic mild social stress
@ast
3xTgAD mice exhibit altered behavior and elevated Aβ after chronic mild social stress
@en
P2093
P2860
P1476
3xTgAD mice exhibit altered behavior and elevated Aβ after chronic mild social stress
@en
P2093
Bronwen Martin
Mohamed R Mughal
Nathan Herdener
Sarah J Texel
Sarah M Rothman
Wei-Na Cong
P2860
P304
P356
10.1016/J.NEUROBIOLAGING.2011.07.005
P577
2011-08-19T00:00:00Z