Alteration of gene expression by alcohol exposure at early neurulation. - Wikidata - awgldk - TriplyDB

Alteration of gene expression by alcohol exposure at early neurulation.

Alteration of gene expression by alcohol exposure at early neurulation.

http://www.wikidata.org/entity/Q33825808

about

Long-term genomic and epigenomic dysregulation as a consequence of prenatal alcohol exposure: a model for fetal alcohol spectrum disorders.Fetal alcohol syndrome, chemo-biology and OMICS: ethanol effects on vitamin metabolism during neurodevelopment as measured by systems biology analysis.Implications of genomic signatures in the differential vulnerability to fetal alcohol exposure in C57BL/6 and DBA/2 mice Ethanol alters the balance of Sox2, Oct4, and Nanog expression in distinct subpopulations during differentiation of embryonic stem cells.Dissecting FASD through the global transcriptome CB1-receptor knockout neonatal mice are protected against ethanol-induced impairments of DNMT1, DNMT3A, and DNA methylation.Prenatal alcohol exposure modifies glucocorticoid receptor subcellular distribution in the medial prefrontal cortex and impairs frontal cortex-dependent learning.Early maternal alcohol consumption alters hippocampal DNA methylation, gene expression and volume in a mouse model.Dose-dependent alcohol-induced alterations in chromatin structure persist beyond the window of exposure and correlate with fetal alcohol syndrome birth defects.Choline supplementation and DNA methylation in the hippocampus and prefrontal cortex of rats exposed to alcohol during development Gene expression changes in C57BL/6J and DBA/2J mice following prenatal alcohol exposure.DNA methylation program in developing hippocampus and its alteration by alcohol.Genome-wide significant association signals in IPO11-HTR1A region specific for alcohol and nicotine codependence.Neurodevelopmental alcohol exposure elicits long-term changes to gene expression that alter distinct molecular pathways dependent on timing of exposure.Prenatal alcohol exposure alters steady-state and activated gene expression in the adult rat brain.G9a-mediated histone methylation regulates ethanol-induced neurodegeneration in the neonatal mouse brain.Alcohol-induced morphological deficits in the development of octavolateral organs of the zebrafish (Danio rerio).Alcohol exposure during development: Impact on the epigenome Epigenetics and lifestyle.Stress-response pathways are altered in the hippocampus of chronic alcoholics.CITED2 mutation and methylation in children with congenital heart disease Eye-specific gene expression following embryonic ethanol exposure in zebrafish: roles for heat shock factor 1.Ethanol induced acetylation of histone at G9a exon1 and G9a-mediated histone H3 dimethylation leads to neurodegeneration in neonatal mice.Meta-Analysis of Gene Expression Patterns in Animal Models of Prenatal Alcohol Exposure Suggests Role for Protein Synthesis Inhibition and Chromatin Remodeling.DNA methylation program during development.DNA Methylation program in normal and alcohol-induced thinning cortex.Epigenetics studies of fetal alcohol spectrum disorder: where are we now?PCDHB14- and GABRB1-like nervous system developmental genes are altered during early neuronal differentiation of NCCIT cells treated with ethanol.On the relationships in rhesus macaques between chronic ethanol consumption and the brain transcriptome.Ethanol exposure alters protein expression in a mouse model of fetal alcohol spectrum disorders A comparison of gene expression responses in rat whole embryo culture and in vivo: time-dependent retinoic acid-induced teratogenic response.Neuronal development genes are key elements mediating the reinforcing effects of methamphetamine, amphetamine, and methylphenidate.PM2.5 induces embryonic growth retardation: Potential involvement of ROS-MAPKs-apoptosis and G0/G1 arrest pathways.Angiogenesis is repressed by ethanol exposure during chick embryonic development.Transcriptional profiling of human neural precursors post alcohol exposure reveals impaired neurogenesis via dysregulation of ERK signaling and miR-145.Ethanol-related alterations in gene expression patterns in the developing murine hippocampus.Delivering alcohol neurotoxicity into nucleus, when clusterin meets BclXL: a commentary.Disconnect between alcohol-induced alterations in chromatin structure and gene transcription in a mouse embryonic stem cell model of exposure.

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P2860

Alteration of gene expression by alcohol exposure at early neurulation.

http://www.wikidata.org/entity/Q33825808

description

2011 nî lūn-bûn

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2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի փետրվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Alteration of gene expression by alcohol exposure at early neurulation.

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Alteration of gene expression by alcohol exposure at early neurulation.

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type

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Alteration of gene expression by alcohol exposure at early neurulation.

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Alteration of gene expression by alcohol exposure at early neurulation.

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Alteration of gene expression by alcohol exposure at early neurulation.

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Alteration of gene expression by alcohol exposure at early neurulation.

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Alteration of gene expression by alcohol exposure at early neurulation.

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Feng C Zhou

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Lang Li

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Qianqian Zhao

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Tiebing Liang

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Yunlong Liu

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P2860

Gene set enrichment analysis: a knowledge-based approach for interpreting genome-wide expression profiles.

Reproducibility of oligonucleotide arrays using small samples

Effects of filtering by Present call on analysis of microarray experiments

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

The language of covalent histone modifications

Transforming growth factor beta1 and ethanol affect transcription and translation of genes and proteins for cell adhesion molecules in B104 neuroblastoma cells

Neurogenin 2 is required for the development of ventral midbrain dopaminergic neurons

PGC-1alpha-responsive genes involved in oxidative phosphorylation are coordinately downregulated in human diabetes

Histone modifications in transcriptional regulation

Neurogenin 1 null mutant ears develop fewer, morphologically normal hair cells in smaller sensory epithelia devoid of innervation.

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Howard J Edenberg

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