YopJ-promoted cytotoxicity and systemic colonization are associated with high levels of murine interleukin-18, gamma interferon, and neutrophils in a live vaccine model of Yersinia pseudotuberculosis infection

YopJ-promoted cytotoxicity and systemic colonization are associated with high levels of murine interleukin-18, gamma interferon, and neutrophils in a live vaccine model of Yersinia pseudotuberculosis infection

Item
http://www.wikidata.org/entity/Q33826059
Yersinia type III effectors perturb host innate immune responsesCaspase-8 and RIP kinases regulate bacteria-induced innate immune responses and cell death.Effector CD8+ T cells are generated in response to an immunodominant epitope in type III effector YopE during primary Yersinia pseudotuberculosis infectionLcrV delivered via type III secretion system of live attenuated Yersinia pseudotuberculosis enhances immunogenicity against pneumonic plague.Mathematical relationship between cytokine concentrations and pathogen levels during infection.YopP-expressing variant of Y. pestis activates a potent innate immune response affording cross-protection against yersiniosis and tularemia [corrected].Superantigenic Yersinia pseudotuberculosis induces the expression of granzymes and perforin by CD4+ T cellsA protective epitope in type III effector YopE is a major CD8 T cell antigen during primary infection with Yersinia pseudotuberculosis.Type III secretion system-dependent translocation of ectopically expressed Yop effectors into macrophages by intracellular Yersinia pseudotuberculosis.CCR2+ Inflammatory Dendritic Cells and Translocation of Antigen by Type III Secretion Are Required for the Exceptionally Large CD8+ T Cell Response to the Protective YopE69-77 Epitope during Yersinia Infection.Uncovering an Important Role for YopJ in the Inhibition of Caspase-1 in Activated Macrophages and Promoting Yersinia pseudotuberculosis Virulence.The cytotoxic necrotizing factor of Yersinia pseudotuberculosis (CNFY) enhances inflammation and Yop delivery during infection by activation of Rho GTPases.Anti-Pneumococcal Vaccine-Induced Cellular Immune Responses in Post-Traumatic Splenectomized Individuals.RIPK1-dependent apoptosis bypasses pathogen blockade of innate signaling to promote immune defense.CCR2+ inflammatory monocytes are recruited to Yersinia pyogranulomas and dictate adaptive responses at the expense of innate immunity during oral infection.All Are Not Created Equal: Phenotypic Adaptation to Distinct Niches Within Mammalian Tissues
P2860

YopJ-promoted cytotoxicity and systemic colonization are associated with high levels of murine interleukin-18, gamma interferon, and neutrophils in a live vaccine model of Yersinia pseudotuberculosis infection

Item
http://www.wikidata.org/entity/Q33826059
description
2010 nî lūn-bûn
@nan
2010 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի մարտին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
YopJ-promoted cytotoxicity and ...... a pseudotuberculosis infection
@ast
YopJ-promoted cytotoxicity and ...... a pseudotuberculosis infection
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type
label
YopJ-promoted cytotoxicity and ...... a pseudotuberculosis infection
@ast
YopJ-promoted cytotoxicity and ...... a pseudotuberculosis infection
@en
prefLabel
YopJ-promoted cytotoxicity and ...... a pseudotuberculosis infection
@ast
YopJ-promoted cytotoxicity and ...... a pseudotuberculosis infection
@en
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P698
P921
P932
P356
P1433
P1476
YopJ-promoted cytotoxicity and ...... a pseudotuberculosis infection
@en
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P698
P921
P932