Quantitative mass spectrometry-based proteomics reveals the dynamic range of primary mouse astrocyte protein secretion - Wikidata - awgldk - TriplyDB

Quantitative mass spectrometry-based proteomics reveals the dynamic range of primary mouse astrocyte protein secretion

Quantitative mass spectrometry-based proteomics reveals the dynamic range of primary mouse astrocyte protein secretion

http://www.wikidata.org/entity/Q33835688

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Sample preparation techniques for the untargeted LC-MS-based discovery of peptides in complex biological matrices Proteome-wide lysine acetylation in cortical astrocytes and alterations that occur during infection with brain parasite Toxoplasma gondii Recent advances in quantitative neuroproteomics SILAC-based proteomics of human primary endothelial cell morphogenesis unveils tumor angiogenic markers Chemical analysis of single cells.A role for cathepsin Z in neuroinflammation provides mechanistic support for an epigenetic risk factor in multiple sclerosis Developmental alcohol exposure leads to a persistent change on astrocyte secretome.Secretome profiling with antibody microarrays.Adipokines: a treasure trove for the discovery of biomarkers for metabolic disorders.Application of proteomics to cerebrovascular disease.Activation of C6 glioblastoma cell ceruloplasmin expression by neighboring human brain endothelia-derived interleukins in an in vitro blood-brain barrier model system.Iron transport across the blood-brain barrier: development, neurovascular regulation and cerebral amyloid angiopathy.Progress in mining the human proteome for disease applications Glial cell ceruloplasmin and hepcidin differentially regulate iron efflux from brain microvascular endothelial cells.Extracellular vimentin interacts with insulin-like growth factor 1 receptor to promote axonal growth.Monitoring Astrocytic Proteome Dynamics by Cell Type-Specific Protein Labeling.Peptidomic analyses of mouse astrocytic cell lines and rat primary cultured astrocytes.Quantitative proteomics reveals altered expression of extracellular matrix related proteins of human primary dermal fibroblasts in response to sulfated hyaluronan and collagen applied as artificial extracellular matrix.GFAP expression as an indicator of disease severity in mouse models of Alexander disease.Structural Basis for Nucleotide Hydrolysis by the Acid Sphingomyelinase-like Phosphodiesterase SMPDL3A.Cand1 promotes assembly of new SCF complexes through dynamic exchange of F box proteins.Mutant copper-zinc superoxide dismutase (SOD1) induces protein secretion pathway alterations and exosome release in astrocytes: implications for disease spreading and motor neuron pathology in amyotrophic lateral sclerosis Mutant SOD1-expressing astrocytes release toxic factors that trigger motoneuron death by inducing hyperexcitability Ferroportin and exocytoplasmic ferroxidase activity are required for brain microvascular endothelial cell iron efflux Extracellular vimentin is a novel axonal growth facilitator for functional recovery in spinal cord-injured mice Mapping of the secretome of primary isolates of mammalian cells, stem cells and derived cell lines.Glia-based biomarkers and their functional role in the CNS.Proteome of brain glia: the molecular basis of diverse glial phenotypes.Astrocyte regulation of CNS inflammation and remyelination.Characterization of a SILAC method for proteomic analysis of primary rat microglia.Proteomic analysis of mouse astrocytes and their secretome by a combination of FASP and StageTip-based, high pH, reversed-phase fractionation.An iTRAQ based quantitative proteomic strategy to explore novel secreted proteins in metastatic hepatocellular carcinoma cell lines.Traumatically injured astrocytes release a proteomic signature modulated by STAT3-dependent cell survival.A novel compound, denosomin, ameliorates spinal cord injury via axonal growth associated with astrocyte-secreted vimentin.Crosstalk between glial and glioblastoma cells triggers the "go-or-grow" phenotype of tumor cells.Nitric oxide counteracts the hyperoxia-induced proliferation and proinflammatory responses of mouse astrocytes.Astrocytes locally translate transcripts in their peripheral processes.Haptoglobin increases the vulnerability of CD163-expressing neurons to hemoglobin.Ceruloplasmin replacement therapy ameliorates neurological symptoms in a preclinical model of aceruloplasminemia.De Novo and Uninterrupted SILAC Labeling of Primary Microglia.

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P2860

Quantitative mass spectrometry-based proteomics reveals the dynamic range of primary mouse astrocyte protein secretion

http://www.wikidata.org/entity/Q33835688

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Quantitative mass spectrometry ...... se astrocyte protein secretion

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Quantitative mass spectrometry ...... se astrocyte protein secretion

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type

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Quantitative mass spectrometry ...... se astrocyte protein secretion

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Quantitative mass spectrometry ...... se astrocyte protein secretion

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Quantitative mass spectrometry ...... se astrocyte protein secretion

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Quantitative mass spectrometry ...... se astrocyte protein secretion

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Quantitative mass spectrometry ...... se astrocyte protein secretion

@en

P2093

Adrian Mak

http://www.w3.org/2001/XMLSchema#string

Harry Ischiropoulos

http://www.w3.org/2001/XMLSchema#string

Lynn Spruce

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Steven H Seeholzer

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Todd M Greco

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P2860

Accurate quantitation of protein expression and site-specific phosphorylation

Improved prediction of signal peptides: SignalP 3.0

Stable isotope labeling by amino acids in cell culture, SILAC, as a simple and accurate approach to expression proteomics

Predicting subcellular localization of proteins based on their N-terminal amino acid sequence

Empirical statistical model to estimate the accuracy of peptide identifications made by MS/MS and database search

Feature-based prediction of non-classical and leaderless protein secretion

Mass spectrometry-based proteomics turns quantitative

Astrocytes expressing ALS-linked mutated SOD1 release factors selectively toxic to motor neurons

Deficiency of ABCA1 impairs apolipoprotein E metabolism in brain

Thrombospondins are astrocyte-secreted proteins that promote CNS synaptogenesis

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2764-2774

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