The cell wall components peptidoglycan and lipoteichoic acid from Staphylococcus aureus act in synergy to cause shock and multiple organ failure
about
Lipoteichoic acid (LTA) of Streptococcus pneumoniae and Staphylococcus aureus activates immune cells via Toll-like receptor (TLR)-2, lipopolysaccharide-binding protein (LBP), and CD14, whereas TLR-4 and MD-2 are not involvedCD14 is a cell-activating receptor for bacterial peptidoglycanNatural pathogens of laboratory mice, rats, and rabbits and their effects on research.Recognition of Staphylococcus aureus by the innate immune system.Chemokines are the main proinflammatory mediators in human monocytes activated by Staphylococcus aureus, peptidoglycan, and endotoxinPeptidoglycan and lipoteichoic acid modify monocyte phenotype in human whole bloodTherapeutic targeting of NOD1 receptorsMonoacyl lipoteichoic acid from pneumococci stimulates human cells but not mouse cellsBlood-brain barrier invasion by group B Streptococcus depends upon proper cell-surface anchoring of lipoteichoic acid.Peptidoglycan and lipoteichoic acid from Staphylococcus aureus induce tumor necrosis factor alpha, interleukin 6 (IL-6), and IL-10 production in both T cells and monocytes in a human whole blood model.Cell-type-specific crosstalk between p38 MAPK and Rho signaling in lung micro- and macrovascular barrier dysfunction induced by Staphylococcus aureus-derived pathogens.The impact of Staphylococcus aureus-associated molecular patterns on staphylococcal superantigen-induced toxic shock syndrome and pneumonia.Pathogenesis of gram-positive bacterial endophthalmitis.Bacterial endophthalmitis: epidemiology, therapeutics, and bacterium-host interactions.Regulation of heme oxygenase-1 gene by peptidoglycan involves the interaction of Elk-1 and C/EBPalpha to increase expression.Anchors away: contribution of a glycolipid anchor to bacterial invasion of host cells.The role of antimicrobial peptides in animal defenses.Differential host inflammatory responses to viable versus antibiotic-killed bacteria in experimental microbial sepsis.Capsular polysaccharide and O-specific antigen divergently modulate pulmonary neutrophil influx in an Escherichia coli model of gram-negative pneumonitis in rats.Commercial preparations of lipoteichoic acid contain endotoxin that contributes to activation of mouse macrophages in vitroUpregulation of toll-like receptor 2 gene expression in macrophage response to peptidoglycan and high concentration of lipopolysaccharide is involved in NF-kappa b activation.Differential tumor necrosis factor alpha expression and release from peritoneal mouse macrophages in vitro in response to proliferating gram-positive versus gram-negative bacteria.Enzyme degradation and proinflammatory activity in arthritogenic and nonarthritogenic Eubacterium aerofaciens cell wallsWhat are the microbial components implicated in the pathogenesis of sepsis? Report on a symposium.Membrane TLR signaling mechanisms in the gastrointestinal tract during sepsis.Nitric oxide and septic shock. From bench to bedsideIndividuality in gut microbiota composition is a complex polygenic trait shaped by multiple environmental and host genetic factorsThe Cell Wall Teichuronic Acid Synthetase (TUAS) Is an Enzyme Complex Located in the Cytoplasmic Membrane of Micrococcus luteusModulation of release of proinflammatory bacterial compounds by antibacterials: potential impact on course of inflammation and outcome in sepsis and meningitis.Structure-function relationship of cytokine induction by lipoteichoic acid from Staphylococcus aureus.Mechanisms of tissue injury in sepsis: contrasts between gram positive and gram negative infection.Reconstituted high-density lipoprotein modulates activation of human leukocytes.Endogenous versus exogenous glucocorticoid responses to experimental bacterial sepsis.Igniting the fire: Staphylococcus aureus virulence factors in the pathogenesis of sepsis.Strain differences in alveolar neutrophil infiltration and macrophage phenotypes in an acute lung inflammation modelReceptors, mediators, and mechanisms involved in bacterial sepsis and septic shock.Lipoteichoic acid and interleukin 1 stimulate synergistically production of hepatocyte growth factor (scatter factor) in human gingival fibroblasts in culture.Influence of lipoteichoic acid structure on recognition by the macrophage scavenger receptorA continuum of anionic charge: structures and functions of D-alanyl-teichoic acids in gram-positive bacteriaAtrial natriuretic peptide protects against Staphylococcus aureus-induced lung injury and endothelial barrier dysfunction
P2860
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P2860
The cell wall components peptidoglycan and lipoteichoic acid from Staphylococcus aureus act in synergy to cause shock and multiple organ failure
description
1995 nî lūn-bûn
@nan
1995 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
The cell wall components pepti ...... ock and multiple organ failure
@ast
The cell wall components pepti ...... ock and multiple organ failure
@en
type
label
The cell wall components pepti ...... ock and multiple organ failure
@ast
The cell wall components pepti ...... ock and multiple organ failure
@en
prefLabel
The cell wall components pepti ...... ock and multiple organ failure
@ast
The cell wall components pepti ...... ock and multiple organ failure
@en
P2093
P2860
P356
P1476
The cell wall components pepti ...... ock and multiple organ failure
@en
P2093
De Kimpe SJ
Kengatharan M
Thiemermann C
P2860
P304
10359-10363
P356
10.1073/PNAS.92.22.10359
P407
P577
1995-10-01T00:00:00Z