DNA damage and reactive nitrogen species are barriers to Vibrio cholerae colonization of the infant mouse intestine.
about
Characterization of the RpoN regulon reveals differential regulation of T6SS and new flagellar operons in Vibrio cholerae O37 strain V52.The yin and yang of bacterial resilience in the human gut microbiota.The highly conserved bacterial RNase YbeY is essential in Vibrio cholerae, playing a critical role in virulence, stress regulation, and RNA processingEfficient responses to host and bacterial signals during Vibrio cholerae colonizationMononuclear-macrophages but not neutrophils act as major infiltrating anti-leptospiral phagocytes during leptospirosis.RecO and RecR are necessary for RecA loading in response to DNA damage and replication fork stress.Either non-homologous ends joining or homologous recombination is required to repair double-strand breaks in the genome of macrophage-internalized Mycobacterium tuberculosisMetR-regulated Vibrio cholerae metabolism is required for virulence.Development of genomic resources for a thraustochytrid pathogen and investigation of temperature influences on gene expression.CTXφ Replication Depends on the Histone-Like HU Protein and the UvrD HelicaseThe NorR regulon is critical for Vibrio cholerae resistance to nitric oxide and sustained colonization of the intestines.The extracytoplasmic function sigma factor σS protects against both intracellular and extracytoplasmic stresses in Staphylococcus aureus.Residues in the N-terminal domain of MutL required for mismatch repair in Bacillus subtilis.Mechanisms of Inactivation by High-Voltage Atmospheric Cold Plasma Differ for Escherichia coli and Staphylococcus aureusThe LonA Protease Regulates Biofilm Formation, Motility, Virulence, and the Type VI Secretion System in Vibrio choleraeStaying Alive: Vibrio cholerae's Cycle of Environmental Survival, Transmission, and DisseminationRecD2 helicase limits replication fork stress in Bacillus subtilis.Fitness factors in vibrios: a mini-review.Nitric oxide-sensing H-NOX proteins govern bacterial communal behavior.Nitric Oxide Regulation of H-NOX Signaling Pathways in Bacteria.Nitric oxide modulates bacterial biofilm formation through a multicomponent cyclic-di-GMP signaling networkDnaN clamp zones provide a platform for spatiotemporal coupling of mismatch detection to DNA replication.Structural and Functional Characterization of the FadR Regulatory Protein from Vibrio alginolyticus.Trapping and visualizing intermediate steps in the mismatch repair pathway in vivo.Cyclic di-GMP Positively Regulates DNA Repair in Vibrio cholerae.Hypermutation-induced in vivo oxidative stress resistance enhances Vibrio cholerae host adaptation
P2860
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P2860
DNA damage and reactive nitrogen species are barriers to Vibrio cholerae colonization of the infant mouse intestine.
description
2011 nî lūn-bûn
@nan
2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
DNA damage and reactive nitrog ...... of the infant mouse intestine.
@ast
DNA damage and reactive nitrog ...... of the infant mouse intestine.
@en
type
label
DNA damage and reactive nitrog ...... of the infant mouse intestine.
@ast
DNA damage and reactive nitrog ...... of the infant mouse intestine.
@en
prefLabel
DNA damage and reactive nitrog ...... of the infant mouse intestine.
@ast
DNA damage and reactive nitrog ...... of the infant mouse intestine.
@en
P2093
P2860
P1433
P1476
DNA damage and reactive nitrog ...... of the infant mouse intestine.
@en
P2093
Bryan W Davies
John J Mekalanos
Lyle A Simmons
Nicole M Dupes
Ryan W Bogard
Tyler A I Gerstenfeld
P2860
P304
P356
10.1371/JOURNAL.PPAT.1001295
P577
2011-02-17T00:00:00Z