Optimal bone strength and mineralization requires the type 2 iodothyronine deiodinase in osteoblasts. - Wikidata - awgldk - TriplyDB

Optimal bone strength and mineralization requires the type 2 iodothyronine deiodinase in osteoblasts.

Optimal bone strength and mineralization requires the type 2 iodothyronine deiodinase in osteoblasts.

http://www.wikidata.org/entity/Q33841948

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Rapid-throughput skeletal phenotyping of 100 knockout mice identifies 9 new genes that determine bone strength Role and Mechanisms of Actions of Thyroid Hormone on the Skeletal Development Abnormal motor phenotype at adult stages in mice lacking type 2 deiodinase Rapid phenotyping of knockout mice to identify genetic determinants of bone strength Thyroid hormone receptors control developmental maturation of the middle ear and the size of the ossicular bones Differences in hypothalamic type 2 deiodinase ubiquitination explain localized sensitivity to thyroxine.Mice with a targeted deletion of the type 2 deiodinase are insulin resistant and susceptible to diet induced obesity.Thyroid hormone receptor α mutation causes a severe and thyroxine-resistant skeletal dysplasia in female mice.Selenium. Role of the essential metalloid in health Minireview: cracking the metabolic code for thyroid hormone signaling A Modest Protective Effect of Thyrotropin against Bone Loss Is Associated with Plasma Triiodothyronine Levels.Bone Indices in Thyroidectomized Patients on Long-Term Substitution Therapy with Levothyroxine Assessed by DXA and HR-pQCT.Thyrostimulin Regulates Osteoblastic Bone Formation During Early Skeletal Development.Coordination of hypothalamic and pituitary T3 production regulates TSH expression.Role of Thyroid Hormones in Skeletal Development and Bone Maintenance.Physiological role and regulation of iodothyronine deiodinases: a 2011 update.Thyroid hormone replacement therapy: three 'simple' questions, complex answers.Thyroid hormone actions in cartilage and bone.American Thyroid Association Guide to investigating thyroid hormone economy and action in rodent and cell models.Potential of human fetal chorionic stem cells for the treatment of osteogenesis imperfecta Type 2 deiodinase at the crossroads of thyroid hormone action Role of the type 2 iodothyronine deiodinase (D2) in the control of thyroid hormone signaling Insights from human genetic studies into the pathways involved in osteoarthritis.The multiple effects of thyroid disorders on bone and mineral metabolism.Emerging role of thyroid hormone metabolites.Physiological Bone Remodeling: Systemic Regulation and Growth Factor Involvement.Triiodothyronine stimulates glucose transport in bone cells.Somatostatin Analogue Treatment of a TSH-Secreting Adenoma Presenting With Accelerated Bone Metabolism and a Pericardial Effusion: A Case Report.Adult mice lacking the type 2 iodothyronine deiodinase have increased subchondral bone but normal articular cartilage.Bone Mineral Content and Density.Strong induction of iodothyronine deiodinases by chemotherapeutic selenocompounds.The effect of forced exercise on knee joints in Dio2(-/-) mice: type II iodothyronine deiodinase-deficient mice are less prone to develop OA-like cartilage damage upon excessive mechanical stress.Thyroid hormone receptor β mediates thyroid hormone effects on bone remodeling and bone mass.Significant deterioration in nanomechanical quality occurs through incomplete extrafibrillar mineralization in rachitic bone: evidence from in-situ synchrotron X-ray scattering and backscattered electron imaging.Selenoprotein P is the essential selenium transporter for bones.An Essential Physiological Role for MCT8 in Bone in Male Mice.Noncanonical thyroid hormone signaling mediates cardiometabolic effects in vivo.Hypothalamus-Pituitary-Thyroid Axis.Selenium-Related Transcriptional Regulation of Gene Expression

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Optimal bone strength and mineralization requires the type 2 iodothyronine deiodinase in osteoblasts.

http://www.wikidata.org/entity/Q33841948

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2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年论文

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Optimal bone strength and mine ...... ine deiodinase in osteoblasts.

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Optimal bone strength and mine ...... ine deiodinase in osteoblasts.

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Optimal bone strength and mine ...... ine deiodinase in osteoblasts.

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Optimal bone strength and mine ...... ine deiodinase in osteoblasts.

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Optimal bone strength and mine ...... ine deiodinase in osteoblasts.

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Optimal bone strength and mine ...... ine deiodinase in osteoblasts.

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PNAS.0911346107

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Proceedings of the National Academy of Sciences of the United States of America

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Optimal bone strength and mine ...... ine deiodinase in osteoblasts.

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Alan Boyde

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Donald L St Germain

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Graham R Williams

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Holly Evans

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J H Duncan Bassett

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Marta Archanco

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Michelle A Lawson

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Peter Croucher

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Peter G T Howell

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Richard H Bassett

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P2860

The Hedgehog-inducible ubiquitin ligase subunit WSB-1 modulates thyroid hormone activation and PTHrP secretion in the developing growth plate

Deiodinases: implications of the local control of thyroid hormone action

Targeted disruption of the type 2 selenodeiodinase gene (DIO2) results in a phenotype of pituitary resistance to T4

Impairment of the selenoenzyme type I iodothyronine deiodinase in C3H/He mice

Self-reported diseases and the risk of non-vertebral fractures: the Tromsø study.

Physiological and genetic analyses of inbred mouse strains with a type I iodothyronine 5' deiodinase deficiency.

Effects of thyroid hormones on bone and mineral metabolism.

The thyroid and the skeleton.

Thyroxine is the serum factor that regulates morphogenesis of columnar cartilage from isolated chondrocytes in chemically defined medium.

Induction of proliferation or hypertrophy of chondrocytes in serum-free culture: the role of insulin-like growth factor-I, insulin, or thyroxine.

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