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Basic science of pulmonary arterial hypertension for clinicians: new concepts and experimental therapies

Basic science of pulmonary arterial hypertension for clinicians: new concepts and experimental therapies

http://www.wikidata.org/entity/Q33849207

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Slow receptor dissociation kinetics differentiate macitentan from other endothelin receptor antagonists in pulmonary arterial smooth muscle cells Endothelin receptor antagonists for pulmonary arterial hypertension Mechanisms of Vascular Smooth Muscle Contraction and the Basis for Pharmacologic Treatment of Smooth Muscle Disorders Interdisciplinary networks for the treatment of childhood pulmonary vascular disease: what pulmonary hypertension doctors can learn from pediatric oncologists The Robyn Barst Memorial Lecture: Differences between the fetal, newborn, and adult pulmonary circulations: relevance for age-specific therapies (2013 Grover Conference series)Metabolic dysfunction in pulmonary hypertension: the expanding relevance of the Warburg effect Right ventricular failure and pathobiology in patients with congenital heart disease - implications for long-term follow-up MicroRNA-143 Activation Regulates Smooth Muscle and Endothelial Cell Crosstalk in Pulmonary Arterial Hypertension Ambrisentan Cardiomyocyte-specific overexpression of HEXIM1 prevents right ventricular hypertrophy in hypoxia-induced pulmonary hypertension in mice Megakaryocytic leukemia 1 (MKL1) regulates hypoxia induced pulmonary hypertension in rats The emperor's new clothes: PDE5 and the heart PGC1α-mediated mitofusin-2 deficiency in female rats and humans with pulmonary arterial hypertension miR-21/DDAH1 pathway regulates pulmonary vascular responses to hypoxia.Dynamin-related protein 1-mediated mitochondrial mitotic fission permits hyperproliferation of vascular smooth muscle cells and offers a novel therapeutic target in pulmonary hypertension Role of phosphodiesterase-5 inhibitors in heart failure: emerging data and concepts.Pulmonary arterial hypertension: the clinical syndrome.Emerging Metabolic Therapies in Pulmonary Arterial Hypertension.Role of Transient Receptor Potential Channels in Heart Transplantation: A Potential Novel Therapeutic Target for Cardiac Allograft Vasculopathy.Adenosine A2B receptor and hyaluronan modulate pulmonary hypertension associated with chronic obstructive pulmonary disease Accuracy of Doppler-echocardiographic mean pulmonary artery pressure for diagnosis of pulmonary hypertension.Reactive oxygen species scavengers improve voltage-gated K(+) channel function in pulmonary arteries of newborn pigs with progressive hypoxia-induced pulmonary hypertension.Transient but not genetic loss of miR-451 is protective in the development of pulmonary arterial hypertension Time-dependent PPARγ Modulation of HIF-1α Signaling in Hypoxic Pulmonary Artery Smooth Muscle Cells.Upregulation of steroidogenic acute regulatory protein by hypoxia stimulates aldosterone synthesis in pulmonary artery endothelial cells to promote pulmonary vascular fibrosis.Protective effects of 10-nitro-oleic acid in a hypoxia-induced murine model of pulmonary hypertension Oxidative modulation of voltage-gated potassium channels.Persistence of complex vascular lesions despite prolonged prostacyclin therapy of pulmonary arterial hypertension.Lung ¹⁸F-fluorodeoxyglucose positron emission tomography for diagnosis and monitoring of pulmonary arterial hypertension Therapeutic effects of mesenchymal stem cell-derived microvesicles on pulmonary arterial hypertension in rats Targeting soluble guanylate cyclase for the treatment of pulmonary hypertension.Circulating levels of copeptin predict outcome in patients with pulmonary arterial hypertension.Enhancer of zeste homolog 2 induces pulmonary artery smooth muscle cell proliferation.The Nox4 inhibitor GKT137831 attenuates hypoxia-induced pulmonary vascular cell proliferation TBX4 mutations (small patella syndrome) are associated with childhood-onset pulmonary arterial hypertension Mir-206 regulates pulmonary artery smooth muscle cell proliferation and differentiation Strategic plan for lung vascular research: An NHLBI-ORDR Workshop Report.High-sensitive Troponin T increase after exercise in patients with pulmonary arterial hypertension Clinical relevance of plasma prostaglandin F2α metabolite concentrations in patients with idiopathic pulmonary fibrosis.MicroRNA-124 suppresses the transactivation of nuclear factor of activated T cells by targeting multiple genes and inhibits the proliferation of pulmonary artery smooth muscle cells

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Basic science of pulmonary arterial hypertension for clinicians: new concepts and experimental therapies

http://www.wikidata.org/entity/Q33849207

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2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Basic science of pulmonary art ...... pts and experimental therapies

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Basic science of pulmonary art ...... pts and experimental therapies

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E Kenneth Weir

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Martin R Wilkins

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Stephen L Archer

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Altered growth responses of pulmonary artery smooth muscle cells from patients with primary pulmonary hypertension to transforming growth factor-beta(1) and bone morphogenetic proteins

Apolipoprotein E inhibits platelet-derived growth factor-induced vascular smooth muscle cell migration and proliferation by suppressing signal transduction and preventing cell entry to G1 phase

NO-independent stimulators and activators of soluble guanylate cyclase: discovery and therapeutic potential

Disruption of methylarginine metabolism impairs vascular homeostasis

Interleukin-6 modulates the expression of the bone morphogenic protein receptor type II through a novel STAT3-microRNA cluster 17/92 pathway

Regulation of endothelial nitric oxide synthase by tetrahydrobiopterin in vascular disease

Signal transduction by the TGF-beta superfamily

Function of the serotonin 5-hydroxytryptamine 2B receptor in pulmonary hypertension

Gene expression patterns in the lungs of patients with primary pulmonary hypertension: a gene microarray analysis

A mitochondria-K+ channel axis is suppressed in cancer and its normalization promotes apoptosis and inhibits cancer growth

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