Tumor necrosis factor-α impairs oligodendroglial differentiation through a mitochondria-dependent process.
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New Insights into the Role of Oxidative Stress Mechanisms in the Pathophysiology and Treatment of Multiple SclerosisNudging oligodendrocyte intrinsic signaling to remyelinate and repair: Estrogen receptor ligand effects.Inhibition of Drp1 hyper-activation is protective in animal models of experimental multiple sclerosis.Upregulation of human PINK1 gene expression by NFκB signallingDevelopment of a central nervous system axonal myelination assay for high throughput screening.Demyelination as a rational therapeutic target for ischemic or traumatic brain injury.Intertwining extracellular nucleotides and their receptors with Ca2+ in determining adult neural stem cell survival, proliferation and final fate.Mitochondrial dysfunction in alveolar and white matter developmental failure in premature infants.Oxidative stress, prefrontal cortex hypomyelination and cognitive symptoms in schizophreniaProtandim Protects Oligodendrocytes against an Oxidative Insult.Treatment against human endogenous retrovirus: a possible personalized medicine approach for multiple sclerosis.Metabolic reprogramming during neuronal differentiation.Erythropoietin Increases Myelination in Oligodendrocytes: Gene Expression Profiling Reveals Early Induction of Genes Involved in Lipid Transport and Metabolism.Complex interactions between hypoxia-ischemia and inflammation in preterm brain injury.Grey matter OPCs are less mature and less sensitive to IFNγ than white matter OPCs: consequences for remyelination.Blockade of sustained tumor necrosis factor in a transgenic model of progressive autoimmune encephalomyelitis limits oligodendrocyte apoptosis and promotes oligodendrocyte maturation.Pulsed electromagnetic fields increase osteogenetic commitment of MSCs via the mTOR pathway in TNF-α mediated inflammatory conditions: an in-vitro study.Glia and hemichannels: key mediators of perinatal encephalopathy.Critical Role of Tumor Necrosis Factor Signaling in Mesenchymal Stem Cell-Based Therapy for Autoimmune and Inflammatory Diseases
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P2860
Tumor necrosis factor-α impairs oligodendroglial differentiation through a mitochondria-dependent process.
description
2014 nî lūn-bûn
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2014 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մարտին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
@yue
2014年論文
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2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Tumor necrosis factor-α impair ...... itochondria-dependent process.
@ast
Tumor necrosis factor-α impair ...... itochondria-dependent process.
@en
type
label
Tumor necrosis factor-α impair ...... itochondria-dependent process.
@ast
Tumor necrosis factor-α impair ...... itochondria-dependent process.
@en
prefLabel
Tumor necrosis factor-α impair ...... itochondria-dependent process.
@ast
Tumor necrosis factor-α impair ...... itochondria-dependent process.
@en
P2093
P2860
P50
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P1476
Tumor necrosis factor-α impair ...... itochondria-dependent process.
@en
P2093
E De Marchi
J Duszyński
M R Wieckowski
S Patergnani
P2860
P2888
P304
P356
10.1038/CDD.2014.35
P577
2014-03-21T00:00:00Z
P5875
P6179
1020376710