HDAC inhibition decreases the expression of EGFR in colorectal cancer cells.
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Pathophysiological mechanisms of death resistance in colorectal carcinomaHistone Modifications, Modifiers and Readers in Melanoma Resistance to Targeted and Immune TherapyVorinostat induces apoptosis and differentiation in myeloid malignancies: genetic and molecular mechanismsModulation of epigenetic targets for anticancer therapy: clinicopathological relevance, structural data and drug discovery perspectives3, 3'5 Triiodo L thyronine induces apoptosis in human breast cancer MCF-7 cells, repressing SMP30 expression through negative thyroid response elements.Class I HDAC activity is required for renal protection and regeneration after acute kidney injuryHistone hyperacetylation up-regulates protein kinase Cδ in dopaminergic neurons to induce cell death: relevance to epigenetic mechanisms of neurodegeneration in Parkinson diseaseHistone deacetylase inhibitor SAHA epigenetically regulates miR-17-92 cluster and MCM7 to upregulate MICA expression in hepatoma.Molecular Mechanism of the Cell Death Induced by the Histone Deacetylase Pan Inhibitor LBH589 (Panobinostat) in Wilms Tumor Cells.Induction of c-Cbl contributes to anti-cancer effects of HDAC inhibitor in lung cancer.Activation of Sirtuin-1 Promotes Renal Fibroblast Activation and Aggravates Renal FibrogenesisInhibition of breast cancer progression by a novel histone deacetylase inhibitor, LW479, by down-regulating EGFR expression.Elevated H3K18 acetylation in airway epithelial cells of asthmatic subjects.Induction of cell cycle arrest and apoptosis with downregulation of Hsp90 client proteins and histone modification by 4β-hydroxywithanolide E isolated from Physalis peruviana.Degradation of epidermal growth factor receptor mediates dasatinib-induced apoptosis in head and neck squamous cell carcinoma cells.EGFR promoter exhibits dynamic histone modifications and binding of ASH2L and P300 in human germinal matrix and gliomas.Vorinostat, an HDAC inhibitor attenuates epidermoid squamous cell carcinoma growth by dampening mTOR signaling pathway in a human xenograft murine model.Histone Deacetylase-3/CAGE Axis Targets EGFR Signaling and Regulates the Response to Anti-Cancer DrugsNutrient transporters: the Achilles' heel of anabolism.Exogenous expression of human SGLT1 exhibits aggregations in sodium dodecyl sulfate polyacrylamide gel electrophoresisHDAC inhibitors induce transcriptional repression of high copy number genes in breast cancer through elongation blockadeHistone Deacetylases Positively Regulate Transcription through the Elongation Machinery.Class I histone deacetylase activity is required for proliferation of renal epithelial cells.Dual Targeting of 3-Hydroxy-3-methylglutaryl Coenzyme A Reductase and Histone Deacetylase as a Therapy for Colorectal CancerTrichostatin A suppresses EGFR expression through induction of microRNA-7 in an HDAC-independent manner in lapatinib-treated cells.Epigenetic modulation: a novel therapeutic target for overcoming hormonal therapy resistance.Epigenetics of colorectal cancer.Valproic acid inhibits the proliferation of SHSY5Y neuroblastoma cancer cells by downregulating URG4/URGCP and CCND1 gene expression.Curcumin as a multifaceted compound against human papilloma virus infection and cervical cancers: A review of chemistry, cellular, molecular, and preclinical features.Notch1 signaling is irresponsible to the anti-leukemic effect of HDACis in B-ALL Nalm-6 cells.Hyper-activation of WNT/β-catenin signaling pathway mediates anti-tumor effects of histone deacetylase inhibitors in acute T lymphoblastic leukemia.IRF5 is elevated in childhood-onset SLE and regulated by histone acetyltransferase and histone deacetylase inhibitors.MRGBP as a potential biomarker for the malignancy of pancreatic ductal adenocarcinoma.HDAC-4 regulates claudin-2 expression in EGFR-ERK1/2 dependent manner to regulate colonic epithelial cell differentiation.Trichostatin A modulates intracellular reactive oxygen species through SOD2 and FOXO1 in human bone marrow-mesenchymal stem cells.RUNX2 expression in thyroid and breast cancer requires the cooperation of three non-redundant enhancers under the control of BRD4 and c-JUN.Histone Deacetylase Inhibitors Resensitize EGFR/EGFRvIII-Overexpressing, Erlotinib-Resistant Glioblastoma Cells to Tyrosine Kinase Inhibition.
P2860
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P2860
HDAC inhibition decreases the expression of EGFR in colorectal cancer cells.
description
2011 nî lūn-bûn
@nan
2011 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մարտին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
HDAC inhibition decreases the expression of EGFR in colorectal cancer cells.
@ast
HDAC inhibition decreases the expression of EGFR in colorectal cancer cells.
@en
type
label
HDAC inhibition decreases the expression of EGFR in colorectal cancer cells.
@ast
HDAC inhibition decreases the expression of EGFR in colorectal cancer cells.
@en
prefLabel
HDAC inhibition decreases the expression of EGFR in colorectal cancer cells.
@ast
HDAC inhibition decreases the expression of EGFR in colorectal cancer cells.
@en
P2860
P1433
P1476
HDAC inhibition decreases the expression of EGFR in colorectal cancer cells.
@en
P2093
Chia-Wei Chou
Wei-Chien Huang
P2860
P304
P356
10.1371/JOURNAL.PONE.0018087
P407
P577
2011-03-25T00:00:00Z