Medroxyprogesterone acetate and dexamethasone are competitive inhibitors of different human steroidogenic enzymes.
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Medroxyprogesterone acetate antagonizes the effects of estrogen treatment on social and sexual behavior in female macaques.The molecular biology, biochemistry, and physiology of human steroidogenesis and its disordersIdentification of new biomarker candidates for glucocorticoid induced insulin resistance using literature miningEvidence that androgens are the primary steroids produced by Xenopus laevis ovaries and may signal through the classical androgen receptor to promote oocyte maturation.Cortisol stimulates secretion of dehydroepiandrosterone in human adrenocortical cells through inhibition of 3betaHSD2.Clinical, biochemical, and molecular characterization of macronodular adrenocortical hyperplasia of the zona reticularis: a new syndrome.Mechanisms responsible for progesterone's protection against lordosis-inhibiting effects of restraint I. Role of progesterone receptorsMechanisms responsible for progesterone's protection against lordosis-inhibiting effects of restraint II. Role of progesterone metabolitesProgressive obesity alters ovarian folliculogenesis with impacts on pro-inflammatory and steroidogenic signaling in female mice.Adrenal androgens and androgen precursors-definition, synthesis, regulation and physiologic actions.Long-term replacement of estrogen in combination with medroxyprogesterone acetate improves acquisition of an alternation task in middle-aged female rats.Effects of long-term treatment with estrogen and medroxyprogesterone acetate on synapse number in the medial prefrontal cortex of aged female rats.Fourth-Generation Progestins Inhibit 3β-Hydroxysteroid Dehydrogenase Type 2 and Modulate the Biosynthesis of Endogenous Steroids.The effects of long-term treatment with estradiol and medroxyprogesterone acetate on tyrosine hydroxylase fibers and neuron number in the medial prefrontal cortex of aged female rats.Factors influencing the cognitive and neural effects of hormone treatment during aging in a rodent model.McCune-Albright syndrome and disorders due to activating mutations of GNAS1.Progesterone and medroxyprogesterone acetate differentially regulate alpha4 subunit expression of GABA(A) receptors in the CA1 hippocampus of female rats.Thiazolidinediones but not metformin directly inhibit the steroidogenic enzymes P450c17 and 3beta -hydroxysteroid dehydrogenase.
P2860
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P2860
Medroxyprogesterone acetate and dexamethasone are competitive inhibitors of different human steroidogenic enzymes.
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Medroxyprogesterone Acetate an ...... t Human Steroidogenic Enzymes1
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Medroxyprogesterone acetate an ...... t human steroidogenic enzymes.
@ast
Medroxyprogesterone acetate an ...... t human steroidogenic enzymes.
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type
label
Medroxyprogesterone Acetate an ...... t Human Steroidogenic Enzymes1
@nl
Medroxyprogesterone acetate an ...... t human steroidogenic enzymes.
@ast
Medroxyprogesterone acetate an ...... t human steroidogenic enzymes.
@en
altLabel
Medroxyprogesterone Acetate an ...... t Human Steroidogenic Enzymes1
@en
prefLabel
Medroxyprogesterone Acetate an ...... t Human Steroidogenic Enzymes1
@nl
Medroxyprogesterone acetate an ...... t human steroidogenic enzymes.
@ast
Medroxyprogesterone acetate an ...... t human steroidogenic enzymes.
@en
P2093
P1476
Medroxyprogesterone Acetate an ...... t Human Steroidogenic Enzymes1
@en
Medroxyprogesterone acetate an ...... t human steroidogenic enzymes.
@en
P2093
R J Auchus
Richard J. Auchus
Tim C. Lee
W L Miller
Walter L. Miller
P304
P356
10.1210/JC.84.6.2104
10.1210/JCEM.84.6.5646
P407
P577
1999-06-01T00:00:00Z