The C3H/HeJBir mouse model: a high susceptibility phenotype for colitis.
about
Role of regulatory T cell in the pathogenesis of inflammatory bowel diseaseDevelopment and survival of Th17 cells within the intestines: the influence of microbiome- and diet-derived signalsThe SAMP1/Yit mouse: another step closer to modeling human inflammatory bowel disease.Host susceptibility to the attaching and effacing bacterial pathogen Citrobacter rodentiumMicrobial-induced Th17: superhero or supervillain?The role of T-regulatory cells and Toll-like receptors in the pathogenesis of human inflammatory bowel disease.Strain dependent differences in glucocorticoid-induced bone loss between C57BL/6J and CD-1 mice.The pathogen recognition receptor NOD2 regulates human FOXP3+ T cell survival.Making sense of the cause of Crohn's - a new look at an old disease.Using murine colitis models to analyze probiotics-host interactions.
P2860
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P2860
The C3H/HeJBir mouse model: a high susceptibility phenotype for colitis.
description
2000 nî lūn-bûn
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2000 թուականի Յունուարին հրատարակուած գիտական յօդուած
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2000 թվականի հունվարին հրատարակված գիտական հոդված
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2000年の論文
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2000年学术文章
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name
The C3H/HeJBir mouse model: a high susceptibility phenotype for colitis.
@ast
The C3H/HeJBir mouse model: a high susceptibility phenotype for colitis.
@en
type
label
The C3H/HeJBir mouse model: a high susceptibility phenotype for colitis.
@ast
The C3H/HeJBir mouse model: a high susceptibility phenotype for colitis.
@en
prefLabel
The C3H/HeJBir mouse model: a high susceptibility phenotype for colitis.
@ast
The C3H/HeJBir mouse model: a high susceptibility phenotype for colitis.
@en
P2093
P2860
P1476
The C3H/HeJBir mouse model: a high susceptibility phenotype for colitis.
@en
P2093
P2860
P356
10.3109/08830180009048390
P577
2000-01-01T00:00:00Z