EMT and inflammation: inseparable actors of cancer progression. - Wikidata - awgldk - TriplyDB

EMT and inflammation: inseparable actors of cancer progression.

EMT and inflammation: inseparable actors of cancer progression.

http://www.wikidata.org/entity/Q33869755

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EMT and Treatment Resistance in Pancreatic Cancer.Basic understanding and therapeutic approaches to target toll-like receptors in cancerous microenvironment and metastasis.Novel agents for pancreatic ductal adenocarcinoma: emerging therapeutics and future directions.Autologous cytokine-induced killer cell immunotherapy may improve overall survival in advanced malignant melanoma patients.The A2B Adenosine Receptor Modulates the Epithelial- Mesenchymal Transition through the Balance of cAMP/PKA and MAPK/ERK Pathway Activation in Human Epithelial Lung Cells.Differential role of Interleukin-1 and Interleukin-6 in K-Ras-driven pancreatic carcinoma undergoing mesenchymal transition Nanomedicines reveal how PBOV1 promotes hepatocellular carcinoma for effective gene therapy LncRNA MALAT1 promotes relapse of breast cancer patients with postoperative fever Increased insulin-like growth factor 1 production by polyploid adipose stem cells promotes growth of breast cancer cells A new meroterpenoid functions as an anti-tumor agent in hepatoma cells by downregulating mTOR activation and inhibiting EMT

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P2860

EMT and inflammation: inseparable actors of cancer progression.

http://www.wikidata.org/entity/Q33869755

description

2017 nî lūn-bûn

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2017 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2017 թվականի հուլիսին հրատարակված գիտական հոդված

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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name

EMT and inflammation: inseparable actors of cancer progression.

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EMT and inflammation: inseparable actors of cancer progression.

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EMT and inflammation: inseparable actors of cancer progression.

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EMT and inflammation: inseparable actors of cancer progression.

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EMT and inflammation: inseparable actors of cancer progression.

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EMT and inflammation: inseparable actors of cancer progression.

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Molecular Oncology

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EMT and inflammation: inseparable actors of cancer progression

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Christine Gilles

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Didier Cataldo

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Julien Lesage

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Creative Commons Attribution

P2860

Identification of human CCR8 as a CCL18 receptor

Mechanisms of TGFβ-Induced Epithelial-Mesenchymal Transition

Signal Transduction Pathways of EMT Induced by TGF-β, SHH, and WNT and Their Crosstalks

TGF-β1-induced EMT promotes targeted migration of breast cancer cells through the lymphatic system by the activation of CCR7/CCL21-mediated chemotaxis

Mesenchymal transition and dissemination of cancer cells is driven by myeloid-derived suppressor cells infiltrating the primary tumor

Role of TGFβ in regulation of the tumor microenvironment and drug delivery (review)

Evidence for a tumoral immune resistance mechanism based on tryptophan degradation by indoleamine 2,3-dioxygenase

Epithelial-to-mesenchymal transition is not required for lung metastasis but contributes to chemoresistance

Interleukin-23 promotes the epithelial-mesenchymal transition of oesophageal carcinoma cells via the Wnt/β-catenin pathway

SNAI1-mediated epithelial-mesenchymal transition confers chemoresistance and cellular plasticity by regulating genes involved in cell death and stem cell maintenance

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Meggy Suarez-Carmona

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