Blockade of GABA(B) receptors completely reverses age-related learning impairment.
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Implications of GABAergic Neurotransmission in Alzheimer's DiseaseNot all brains are created equal: the relevance of individual differences in responsiveness to transcranial electrical stimulationPresynaptic inhibition of olfactory sensory neurons: new mechanisms and potential functionsMolecular aspects of age-related cognitive decline: the role of GABA signalingHippocampal Gαq/₁₁ but not Gαo-coupled receptors are altered in aging.Embryonic GABA(B) receptor blockade alters cell migration, adult hypothalamic structure, and anxiety- and depression-like behaviors sex specifically in mice.Growth hormone and cognitive function.Correlation of cognitive performance and morphological changes in neocortical pyramidal neurons in agingGABA(B) receptor GTP-binding is decreased in the prefrontal cortex but not the hippocampus of aged ratsWillingness to wait and altered encoding of time-discounted reward in the orbitofrontal cortex with normal aging.Reduced gamma frequency in the medial frontal cortex of aged rats during behavior and rest: implications for age-related behavioral slowing.Age-related changes in rostral basal forebrain cholinergic and GABAergic projection neurons: relationship with spatial impairmentRepetitive transcranial magnetic stimulation applications normalized prefrontal dysfunctions and cognitive-related metabolic profiling in aged mice.Prefrontal cortical GABAergic dysfunction contributes to age-related working memory impairment.GABAergic neurotransmission and new strategies of neuromodulation to compensate synaptic dysfunction in early stages of Alzheimer's disease.The space where aging acts: focus on the GABAergic synapseEffects of acute administration of the GABA(B) receptor agonist baclofen on behavioral flexibility in rats.Normal Aging does Not Impair Orbitofrontal-Dependent Reinforcer Devaluation EffectsIn vitro screening of major neurotransmitter systems possibly involved in the mechanism of action of antibodies to S100 protein in released-active form.GABAergic inhibitory neurons as therapeutic targets for cognitive impairment in schizophrenia.Frontal Gamma-Aminobutyric Acid Concentrations Are Associated With Cognitive Performance in Older Adults
P2860
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P2860
Blockade of GABA(B) receptors completely reverses age-related learning impairment.
description
2009 nî lūn-bûn
@nan
2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Blockade of GABA(B) receptors completely reverses age-related learning impairment.
@ast
Blockade of GABA(B) receptors completely reverses age-related learning impairment.
@en
type
label
Blockade of GABA(B) receptors completely reverses age-related learning impairment.
@ast
Blockade of GABA(B) receptors completely reverses age-related learning impairment.
@en
prefLabel
Blockade of GABA(B) receptors completely reverses age-related learning impairment.
@ast
Blockade of GABA(B) receptors completely reverses age-related learning impairment.
@en
P2093
P2860
P1433
P1476
Blockade of GABA(B) receptors completely reverses age-related learning impairment
@en
P2093
P2860
P304
P356
10.1016/J.NEUROSCIENCE.2009.08.055
P407
P577
2009-08-31T00:00:00Z