Persistence of viral DNA in the epithelial basal layer suggests a model for papillomavirus latency following immune regression. - Wikidata - awgldk - TriplyDB

Persistence of viral DNA in the epithelial basal layer suggests a model for papillomavirus latency following immune regression.

Persistence of viral DNA in the epithelial basal layer suggests a model for papillomavirus latency following immune regression.

http://www.wikidata.org/entity/Q33873015

about

Human Papillomaviruses; Epithelial Tropisms, and the Development of Neoplasia Shared mechanisms in stemness and carcinogenesis: lessons from oncogenic viruses Role of innate immunity against human papillomavirus (HPV) infections and effect of adjuvants in promoting specific immune response Bovine and human papillomaviruses: a comparative review Strain-specific properties and T cells regulate the susceptibility to papilloma induction by Mus musculus papillomavirus 1 The Role of the DNA Damage Response throughout the Papillomavirus Life Cycle HPV clearance and the neglected role of stochasticity Molecular archaeology of Flaviviridae untranslated regions: duplicated RNA structures in the replication enhancer of flaviviruses and pestiviruses emerged via convergent evolution Identification of protein biomarkers for cervical cancer using human cervicovaginal fluid Papillomaviruses use recombination-dependent replication to vegetatively amplify their genomes in differentiated cells Recent Insights into the Control of Human Papillomavirus (HPV) Genome Stability, Loss, and Degradation.The known unknowns of HPV natural history.Immunologic Control of Mus musculus Papillomavirus Type 1.The E1 protein of human papillomavirus type 16 is dispensable for maintenance replication of the viral genome.Exploring the potential of laser capture microdissection technology in integrated oral biosciences.Modeling the Mechanisms by Which HIV-Associated Immunosuppression Influences HPV Persistence at the Oral Mucosa.Contributions of recent and past sexual partnerships on incident human papillomavirus detection: acquisition and reactivation in older women Pathophysiology of ocular surface squamous neoplasia The immune response to papillomavirus during infection persistence and regression.The biology of papillomavirus latency.Evidence and impact of human papillomavirus latency.Patterns of persistent genital human papillomavirus infection among women worldwide: a literature review and meta-analysis.Differential proteins among normal cervix cells and cervical cancer cells with HPV-16 infection, through mass spectrometry-based Proteomics (2D-DIGE) in women from Southern México.Human papillomavirus molecular biology and disease association.Immunosuppression facilitates the reactivation of latent papillomavirus infections.HPV disease transmission protection and control.The role of ubiquitin and ubiquitin-like modification systems in papillomavirus biology.The HPV16 oncogenes cause aberrant stem cell mobilization.Human papillomavirus in the HIV-infected host: epidemiology and pathogenesis in the antiretroviral era.Model systems of human papillomavirus-associated disease.Mechanistic mathematical models: An underused platform for HPV research.Modulation of the DNA damage response during the life cycle of human papillomaviruses.Recent advances in preclinical model systems for papillomaviruses.Human papillomavirus molecular biology.Oncogenic human papillomaviruses.HPV-positive women with normal cytology remain at increased risk of CIN3 after a negative repeat HPV test.Changing Stem Cell Dynamics during Papillomavirus Infection: Potential Roles for Cellular Plasticity in the Viral Lifecycle and Disease.Felis catus papillomavirus type 2 DNA loads on kittens are transient and do not reflect their susceptibility to infection.HPV31 utilizes the ATR-Chk1 pathway to maintain elevated RRM2 levels and a replication-competent environment in differentiating Keratinocytes.Re-detection vs. new acquisition of high-risk human papillomavirus in mid-adult women.

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P2860

Persistence of viral DNA in the epithelial basal layer suggests a model for papillomavirus latency following immune regression.

http://www.wikidata.org/entity/Q33873015

description

2011 nî lūn-bûn

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2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2011 թվականի ապրիլին հրատարակված գիտական հոդված

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2011年の論文

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J.VIROL.2011.03.019

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Gareth Adam Maglennon

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John Doorbar

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Pauline McIntosh

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P2860

Latent infection induced with cottontail rabbit papillomavirus. A model for human papillomavirus latency

Detection of human papillomavirus infection in diseased and nondiseased sites of the respiratory tract in recurrent respiratory papillomatosis patients by DNA hybridization.

Laser capture microdissection of cervical human papillomavirus infections: copy number of the virus in cancerous and normal tissue and heterogeneous DNA methylation.

Common threads in persistent viral infections

Human papillomavirus life cycle: active and latent phases.

Protective immunity to rabbit oral and cutaneous papillomaviruses by immunization with short peptides of L2, the minor capsid protein.

Life cycle heterogeneity in animal models of human papillomavirus-associated disease

European course on HPV associated pathology: guidelines for primary care physicians for the diagnosis and management of anogenital warts.

Human papillomavirus type 31 oncoproteins E6 and E7 are required for the maintenance of episomes during the viral life cycle in normal human keratinocytes.

Regression of papillomas induced by cottontail rabbit papillomavirus is associated with infiltration of CD8+ cells and persistence of viral DNA after regression.

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10.1016/J.VIROL.2011.03.019

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2

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21492895

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viral persistence

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3101335

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