Amelioration of sepsis by inhibiting sialidase-mediated disruption of the CD24-SiglecG interaction. - Wikidata - awgldk - TriplyDB

Amelioration of sepsis by inhibiting sialidase-mediated disruption of the CD24-SiglecG interaction.

Amelioration of sepsis by inhibiting sialidase-mediated disruption of the CD24-SiglecG interaction.

http://www.wikidata.org/entity/Q33874812

about

Sterile post-traumatic immunosuppression Siglecs as sensors of self in innate and adaptive immune responses Siglecs and immune regulation Synthesis and structural characterisation of selective non-carbohydrate-based inhibitors of bacterial sialidases Tuning mechanism-based inactivators of neuraminidases: mechanistic and structural insights Danger Signals and Graft-versus-host Disease: Current Understanding and Future Perspectives Adipocyte progenitor cells initiate monocyte chemoattractant protein-1-mediated macrophage accumulation in visceral adipose tissue.Siglec-G-CD24 axis controls the severity of graft-versus-host disease in mice.Siglec-G/10 in self-nonself discrimination of innate and adaptive immunity.Broad and direct interaction between TLR and Siglec families of pattern recognition receptors and its regulation by Neu1.The interplay between Siglecs and sialylated pathogens Siglec-mediated regulation of immune cell function in disease.Group B Streptococcus engages an inhibitory Siglec through sialic acid mimicry to blunt innate immune and inflammatory responses in vivo Since there are PAMPs and DAMPs, there must be SAMPs? Glycan “self-associated molecular patterns” dampen innate immunity, but pathogens can mimic them.Leukocyte inflammatory responses provoked by pneumococcal sialidase.Structural characterization of the carbohydrate-binding module of NanA sialidase, a pneumococcal virulence factor Desialylation of Spermatozoa and Epithelial Cell Glycocalyx Is a Consequence of Bacterial Infection of the Epididymis.A hypermorphic SP1-binding CD24 variant associates with risk and progression of multiple sclerosis.Steering Siglec-Sialic Acid Interactions on Living Cells using Bioorthogonal Chemistry.Sialic acid metabolism and sialyltransferases: natural functions and applications.Life after death: targeting high mobility group box 1 in emergent cancer therapies.Sialic acids and autoimmune disease.Induction of Siglec-1 by Endotoxin Tolerance Suppresses the Innate Immune Response by Promoting TGF-β1 Production.Copresentation of antigen and ligands of Siglec-G induces B cell tolerance independent of CD22.CD24 enrichment protects while its loss increases susceptibility of juvenile chondrocytes towards inflammation Sepsis-induced potentiation of peritoneal macrophage migration is mitigated by programmed cell death receptor-1 gene deficiency.CD33-related siglecs as potential modulators of inflammatory responses.CD22 and Siglec-G regulate inhibition of B-cell signaling by sialic acid ligand binding and control B-cell tolerance.Siglec-G is a B-1 cell inhibitory receptor and also controls B cell tolerance.Siglec-G represses DAMP-mediated effects on T cells.Altered homeostatic regulation of innate and adaptive immunity in lower gastrointestinal tract GVHD pathogenesis.Gut mucosal DAMPs in IBD: from mechanisms to therapeutic implications.Pneumococcal Neuraminidase Substrates Identified through Comparative Proteomics Enabled by Chemoselective Labeling Antipneumococcal activity of neuraminidase inhibiting artocarpin.Metabolic sialic acid blockade lowers the activation threshold of moDCs for TLR stimulation.CD24 activates the NLRP3 inflammasome through c-Src kinase activity in a model of the lining epithelium of inflamed periodontal tissues.Sialic Acid-Binding Immunoglobulin-like Lectin G Promotes Atherosclerosis and Liver Inflammation by Suppressing the Protective Functions of B-1 Cells.The NEU1-selective sialidase inhibitor, C9-butyl-amide-DANA, blocks sialidase activity and NEU1-mediated bioactivities in human lung in vitro and murine lung in vivo.CD24: A Rheostat That Modulates Cell Surface Receptor Signaling of Diverse Receptors Siglec-E Negatively Regulates the Activation of TLR4 by Controlling Its Endocytosis.

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Amelioration of sepsis by inhibiting sialidase-mediated disruption of the CD24-SiglecG interaction.

http://www.wikidata.org/entity/Q33874812

description

2011 nî lūn-bûn

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2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2011 թվականի ապրիլին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Amelioration of sepsis by inhi ...... the CD24-SiglecG interaction.

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Amelioration of sepsis by inhi ...... the CD24-SiglecG interaction.

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Amelioration of sepsis by inhi ...... the CD24-SiglecG interaction.

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Amelioration of sepsis by inhi ...... the CD24-SiglecG interaction.

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Nature Biotechnology

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Amelioration of sepsis by inhi ...... f the CD24-SiglecG interaction

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Chong Chen

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Cory M Hogaboam

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Dexing Fang

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Guo-Yun Chen

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Hai Yu

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Jiansong Cheng

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Jin-Qing Liu

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Jingyao Qu

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Karen A Cavassani

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Lei Sun

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P2860

Capnocytophaga canimorsus: a human pathogen feeding at the surface of epithelial cells and phagocytes

Complete Genome Sequence of a Virulent Isolate of Streptococcus pneumoniae

The Ashwell receptor mitigates the lethal coagulopathy of sepsis

Silica crystals and aluminum salts activate the NALP3 inflammasome through phagosomal destabilization

CD24-Siglec G/10 discriminates danger- from pathogen-associated molecular patterns

Innate immune activation through Nalp3 inflammasome sensing of asbestos and silica

Critical loss of the balance between Th17 and T regulatory cell populations in pathogenic SIV infection

A human homologue of the Drosophila Toll protein signals activation of adaptive immunity

HMG-1 as a late mediator of endotoxin lethality in mice

Burden of disease caused by Streptococcus pneumoniae in children younger than 5 years: global estimates

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10.1038/NBT.1846

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