Group B Streptococcus β-hemolysin/cytolysin breaches maternal-fetal barriers to cause preterm birth and intrauterine fetal demise in vivo - Wikidata - awgldk - TriplyDB

Group B Streptococcus β-hemolysin/cytolysin breaches maternal-fetal barriers to cause preterm birth and intrauterine fetal demise in vivo

Group B Streptococcus β-hemolysin/cytolysin breaches maternal-fetal barriers to cause preterm birth and intrauterine fetal demise in vivo

http://www.wikidata.org/entity/Q33883219

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Inflammasome/IL-1β Responses to Streptococcal Pathogens Group B Streptococcus vaccine development: present status and future considerations, with emphasis on perspectives for low and middle income countries A streptococcal lipid toxin induces membrane permeabilization and pyroptosis leading to fetal injury Interaction of Streptococcus agalactiae and Cellular Innate Immunity in Colonization and Disease Complete Genome Sequence of Streptococcus agalactiae CNCTC 10/84, a Hypervirulent Sequence Type 26 Strain.Modification of the CpsA protein reveals a role in alteration of the Streptococcus agalactiae cell envelope Identification of a gene in Mycoplasma hominis associated with preterm birth and microbial burden in intraamniotic infection.Mast cell degranulation by a hemolytic lipid toxin decreases GBS colonization and infection Characterization of host immunity during persistent vaginal colonization by Group B Streptococcus.Non-haemolytic and non-pigmented group b streptococcus, an infrequent cause of early onset neonatal sepsis.Molecular Characterization of Nonhemolytic and Nonpigmented Group B Streptococci Responsible for Human Invasive Infections.Draft Genome Sequence of an Invasive Streptococcus agalactiae Isolate Lacking Pigmentation.The placental membrane microbiome is altered among subjects with spontaneous preterm birth with and without chorioamnionitis.Bacterial Hyaluronidase Promotes Ascending GBS Infection and Preterm Birth.Group B Streptococcus circumvents neutrophils and neutrophil extracellular traps during amniotic cavity invasion and preterm labor.Group B Streptococcus Induces Neutrophil Recruitment to Gestational Tissues and Elaboration of Extracellular Traps and Nutritional Immunity.Streptococcal toxins: role in pathogenesis and disease.Intrauterine infection, immune system and premature birth.Perinatal Group B Streptococcal Infections: Virulence Factors, Immunity, and Prevention Strategies.A sexually dichotomous, autistic-like phenotype is induced by Group B Streptococcus maternofetal immune activation.Mucosal vaccination promotes clearance of Streptococcus agalactiae vaginal colonization.Neonatal protection and preterm birth reduction following maternal group B streptococcus vaccination in a mouse model.A Murine Model of Group B Streptococcus Vaginal Colonization.Sex-Dependent Influence of Developmental Toxicant Exposure on Group B Streptococcus-Mediated Preterm Birth in a Murine Model.Long-term impact of intrauterine neuroinflammation and treatment with magnesium sulphate and betamethasone: Sex-specific differences in a preterm labor murine model.The Streptococcus agalactiae Stringent Response Enhances Virulence and Persistence in Human Blood.Human cervical mucus plugs exhibit insufficiencies in antimicrobial activity towards Group B Streptococcus.Protective effect of Group B Streptococcus type-III polysaccharide conjugates against maternal colonization, ascending infection and neonatal transmission in rodent models.Vaginal Microbiomes Associated With Aerobic Vaginitis and Bacterial Vaginosis.Group B Streptococcal Maternal Colonization and Neonatal Disease: Molecular Mechanisms and Preventative Approaches.Group B streptococcus exploits vaginal epithelial exfoliation for ascending infection.Parallel detection of lactobacillus and bacterial vaginosis-associated bacterial DNA in the chorioamnion and vagina of pregnant women at term.Exploring the Pregnant Guinea Pig as a Model for Group B Streptococcus Intrauterine Infection Maternal group B Streptococcus recto vaginal colonization increases the odds of stillbirth: evidence from Eastern Ethiopia Ascending Vaginal Infection Using Bioluminescent Bacteria Evokes Intrauterine Inflammation, Preterm Birth, and Neonatal Brain Injury in Pregnant Mice A mouse model reproducing the pathophysiology of neonatal group B streptococcal infection

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P2860

Group B Streptococcus β-hemolysin/cytolysin breaches maternal-fetal barriers to cause preterm birth and intrauterine fetal demise in vivo

http://www.wikidata.org/entity/Q33883219

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2014 nî lūn-bûn

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2014 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2014 թվականի հունվարին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Group B Streptococcus β-hemoly ...... rauterine fetal demise in vivo

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Group B Streptococcus β-hemoly ...... rauterine fetal demise in vivo

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Group B Streptococcus β-hemoly ...... rauterine fetal demise in vivo

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Group B Streptococcus β-hemoly ...... rauterine fetal demise in vivo

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Group B Streptococcus β-hemoly ...... rauterine fetal demise in vivo

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Group B Streptococcus β-hemoly ...... rauterine fetal demise in vivo

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The Journal of Infectious Diseases

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Group B Streptococcus β-hemoly ...... rauterine fetal demise in vivo

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Leah M Byland

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Leor H Akabas

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Lindsay B Walker

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Rosanna G Abellar

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Shari E Gelber

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P2860

Antibiotics in early life alter the murine colonic microbiome and adiposity

A hemolytic pigment of Group B Streptococcus allows bacterial penetration of human placenta

Recent advances in understanding the molecular basis of group B Streptococcus virulence

Revisiting the Need for Vaccine Prevention of Late-Onset Neonatal Group B Streptococcal Disease

Epidemiology of Invasive Group B Streptococcal Disease in the United States, 1999-2005

CovS/CovR of group B streptococcus: a two-component global regulatory system involved in virulence.

The human microbiome: at the interface of health and disease

The cyl genes of Streptococcus agalactiae are involved in the production of pigment.

The molecular pathophysiology of bacterially induced preterm labor: insights from the murine model.

Regulation of virulence by a two-component system in group B streptococcus.

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265-273

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10.1093/INFDIS/JIU067

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2

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210

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Thomas A Hooven

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2014-01-28T00:00:00Z

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259961152

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24474814

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4092248

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