Notch and transforming growth factor-beta (TGFbeta) signaling pathways cooperatively regulate vascular smooth muscle cell differentiation. - Wikidata - awgldk - TriplyDB

Notch and transforming growth factor-beta (TGFbeta) signaling pathways cooperatively regulate vascular smooth muscle cell differentiation.

Notch and transforming growth factor-beta (TGFbeta) signaling pathways cooperatively regulate vascular smooth muscle cell differentiation.

http://www.wikidata.org/entity/Q33885162

about

Cyclic stretch stimulates vascular smooth muscle cell alignment by redox-dependent activation of Notch3 Olfactomedin 2, a novel regulator for transforming growth factor-β-induced smooth muscle differentiation of human embryonic stem cell-derived mesenchymal cells Impact of notch signaling on inflammatory responses in cardiovascular disorders Induced pluripotent stem cell-derived vascular smooth muscle cells: methods and application Biomechanical regulation of vascular smooth muscle cell functions: from in vitro to in vivo understanding Biochemical characterization and cellular effects of CADASIL mutants of NOTCH3 Histone deacetylase activity selectively regulates notch-mediated smooth muscle differentiation in human vascular cells.Cardiac neural crest orchestrates remodeling and functional maturation of mouse semilunar valves.CD248 facilitates tumor growth via its cytoplasmic domain.Potential drug targets for calcific aortic valve disease.Notch signaling change in pulmonary vascular remodeling in rats with pulmonary hypertension and its implication for therapeutic intervention.Notch signaling in descending thoracic aortic aneurysm and dissection Delta-like ligand 4-Notch signaling regulates bone marrow-derived pericyte/vascular smooth muscle cell formation The Notch ligand Delta-like 1 integrates inputs from TGFbeta/Activin and Wnt pathways.Von Willebrand factor inhibits mature smooth muscle gene expression through impairment of Notch signaling.The periodontal pathogen Porphyromonas gingivalis changes the gene expression in vascular smooth muscle cells involving the TGFbeta/Notch signalling pathway and increased cell proliferation Metalloprotease-disintegrin ADAM12 expression is regulated by Notch signaling via microRNA-29.TGF-β prevents phosphate-induced osteogenesis through inhibition of BMP and Wnt/β-catenin pathways.The miR-143/145 cluster is a novel transcriptional target of Jagged-1/Notch signaling in vascular smooth muscle cells.Mechanisms of TGF-β-induced differentiation in human vascular smooth muscle cells Tetraspanin CD9 regulates cell contraction and actin arrangement via RhoA in human vascular smooth muscle cells Transforming growth factor-beta1 (TGF-beta1) utilizes distinct pathways for the transcriptional activation of microRNA 143/145 in human coronary artery smooth muscle cells.The role of Notch signaling in muscle progenitor cell depletion and the rapid onset of histopathology in muscular dystrophy.Elongin C is a mediator of Notch4 activity in human renal tubule cells.Notch pathway regulation of neural crest cell development in vivo.Cell division cycle 7 is a novel regulator of transforming growth factor-β-induced smooth muscle cell differentiation.Transforming growth factor-β and smooth muscle differentiation Vascular smooth muscle progenitor cells: building and repairing blood vessels.Moxifloxacin modifies corneal fibroblast-to-myofibroblast differentiation Collagen represses canonical Notch signaling and binds to Notch ectodomain Mechanisms simultaneously regulate smooth muscle proliferation and differentiation.Contractile protein expression is upregulated by reactive oxygen species in aorta of Goto-Kakizaki rat Endothelial signaling and the molecular basis of arteriovenous malformation.Molecular pathways of notch signaling in vascular smooth muscle cells.Molecular controls of arterial morphogenesis.Smooth Muscle Cell Differentiation: Model Systems, Regulatory Mechanisms, and Vascular Diseases.Notch: A multi-functional integrating system of microenvironmental signals.Notch Signaling in Vascular Smooth Muscle Cells.Differential Regulation of NOTCH2 and NOTCH3 Contribute to Their Unique Functions in Vascular Smooth Muscle Cells.Phospholipase Cγ1 Mediates Intima Formation Through Akt-Notch1 Signaling Independent of the Phospholipase Activity.

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Notch and transforming growth factor-beta (TGFbeta) signaling pathways cooperatively regulate vascular smooth muscle cell differentiation.

http://www.wikidata.org/entity/Q33885162

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Notch and transforming growth ...... h muscle cell differentiation.

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Notch and transforming growth ...... h muscle cell differentiation.

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Notch and transforming growth ...... h muscle cell differentiation.

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Notch and transforming growth ...... h muscle cell differentiation.

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Notch and transforming growth ...... h muscle cell differentiation.

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Notch and transforming growth ...... h muscle cell differentiation.

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JBC.M109.076414

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Journal of Biological Chemistry

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Notch and transforming growth ...... h muscle cell differentiation.

@en

P2093

Calvin P H Vary

http://www.w3.org/2001/XMLSchema#string

Deepak Venkatesh

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Douglas B Spicer

http://www.w3.org/2001/XMLSchema#string

Joshua Boucher

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Lucy Liaw

http://www.w3.org/2001/XMLSchema#string

Sumithra Urs

http://www.w3.org/2001/XMLSchema#string

Tyler Bernaiche

http://www.w3.org/2001/XMLSchema#string

Yuefeng Tang

http://www.w3.org/2001/XMLSchema#string

P2860

Members of the Jagged/Notch gene families are expressed in injured arteries and regulate cell phenotype via alterations in cell matrix and cell-cell interaction

Activin receptor-like kinase (ALK)1 is an antagonistic mediator of lateral TGFbeta/ALK5 signaling

Hairy-related transcription factors inhibit Notch-induced smooth muscle alpha-actin expression by interfering with Notch intracellular domain/CBF-1 complex interaction with the CBF-1-binding site

Novel crosstalk to BMP signalling: cGMP-dependent kinase I modulates BMP receptor and Smad activity

Cross-talk between the Notch and TGF-beta signaling pathways mediated by interaction of the Notch intracellular domain with Smad3

Members of the HRT family of basic helix-loop-helix proteins act as transcriptional repressors downstream of Notch signaling

Endothelial expression of the Notch ligand Jagged1 is required for vascular smooth muscle development

HERP1 inhibits myocardin-induced vascular smooth muscle cell differentiation by interfering with SRF binding to CArG box

TGF-beta activates Erk MAP kinase signalling through direct phosphorylation of ShcA

Notch 1 and 3 receptor signaling modulates vascular smooth muscle cell growth, apoptosis, and migration via a CBF-1/RBP-Jk dependent pathway

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10.1074/JBC.M109.076414

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23

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285

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20368328

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2878520

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