Reduction of microRNA 122 expression in IFNL3 CT/TT carriers and during progression of fibrosis in patients with chronic hepatitis C - Wikidata - awgldk - TriplyDB

Reduction of microRNA 122 expression in IFNL3 CT/TT carriers and during progression of fibrosis in patients with chronic hepatitis C

Reduction of microRNA 122 expression in IFNL3 CT/TT carriers and during progression of fibrosis in patients with chronic hepatitis C

http://www.wikidata.org/entity/Q33887760

about

Direct-acting antivirals for the treatment of hepatitis C virus infection: optimizing current IFN-free treatment and future perspectives.Interferon lambda: opportunities, risks, and uncertainties in the fight against HCV.IFI35, mir-99a and HCV genotype to predict sustained virological response to pegylated-interferon plus ribavirin in chronic hepatitis C.A microrna screen to identify regulators of peritoneal fibrosis in a rat model of peritoneal dialysis.MicroRNAs: Role in Hepatitis C Virus pathogenesis MicroRNA-122 Inhibits the Production of Inflammatory Cytokines by Targeting the PKR Activator PACT in Human Hepatic Stellate Cells Association with Spontaneous Hepatitis C Viral Clearance and Genetic Differentiation of IL28B/IFNL4 Haplotypes in Populations from Mexico Acute Hepatitis C Virus Infection Induces Consistent Changes in Circulating MicroRNAs That Are Associated with Nonlytic Hepatocyte Release.Parallel expression profiling of hepatic and serum microRNA-122 associated with clinical features and treatment responses in chronic hepatitis C patients.The Liver MicroRNA Expression Profiles Associated With Chronic Hepatitis C Virus (HCV) Genotype-4 Infection: A Preliminary Study.MicroRNA-based diagnostic tools for advanced fibrosis and cirrhosis in patients with chronic hepatitis B and C.Circulating microRNAs as Potential Biomarkers of Infectious Disease.Optimal IFN-free therapy in treatment-naïve patients with HCV genotype 1 infection.Immune regulation in chronic hepatitis C virus infection.Diagnostic and Prognostic Value of MicroRNA in Viral Diseases.Protection of CD4+ T cells from hepatitis C virus infection-associated senescence via ΔNp63-miR-181a-Sirt1 pathway.Decline of miR-124 in myeloid cells promotes regulatory T-cell development in hepatitis C virus infection.Hepatitis C virus-induced reduction in miR-181a impairs CD4(+) T-cell responses through overexpression of DUSP6.MicroRNA-Related Polymorphisms in Infectious Diseases-Tiny Changes With a Huge Impact on Viral Infections and Potential Clinical Applications.

P2860

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P2860

Reduction of microRNA 122 expression in IFNL3 CT/TT carriers and during progression of fibrosis in patients with chronic hepatitis C

http://www.wikidata.org/entity/Q33887760

description

2014 nî lūn-bûn

@nan

2014 թուականի Մարտին հրատարակուած գիտական յօդուած

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2014 թվականի մարտին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Reduction of microRNA 122 expr ...... ients with chronic hepatitis C

@ast

Reduction of microRNA 122 expr ...... ients with chronic hepatitis C

@en

type

label

Reduction of microRNA 122 expr ...... ients with chronic hepatitis C

@ast

Reduction of microRNA 122 expr ...... ients with chronic hepatitis C

@en

prefLabel

Reduction of microRNA 122 expr ...... ients with chronic hepatitis C

@ast

Reduction of microRNA 122 expr ...... ients with chronic hepatitis C

@en

P1433

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P1433

Journal of Virology

P1476

Reduction of microRNA 122 expr ...... ients with chronic hepatitis C

@en

P2093

Emilie Estrabaud

http://www.w3.org/2001/XMLSchema#string

Kevin Appourchaux

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Martine Lapalus

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Michel Vidaud

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Michelle Martinot-Peignoux

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Olivier Lada

http://www.w3.org/2001/XMLSchema#string

Patrick Marcellin

http://www.w3.org/2001/XMLSchema#string

Simon De Muynck

http://www.w3.org/2001/XMLSchema#string

Tarik Asselah

http://www.w3.org/2001/XMLSchema#string

P2860

A variant upstream of IFNL3 (IL28B) creating a new interferon gene IFNL4 is associated with impaired clearance of hepatitis C virus

Antagonism of microRNA-122 in mice by systemically administered LNA-antimiR leads to up-regulation of a large set of predicted target mRNAs in the liver

Interferon modulation of cellular microRNAs as an antiviral mechanism

MicroRNAs: genomics, biogenesis, mechanism, and function

Modulation of hepatitis C virus RNA abundance by a liver-specific MicroRNA

Genetic variation in IL28B predicts hepatitis C treatment-induced viral clearance

Interleukin-28B polymorphism improves viral kinetics and is the strongest pretreatment predictor of sustained virologic response in genotype 1 hepatitis C virus

Mechanisms of gene silencing by double-stranded RNA

miR-122 regulation of lipid metabolism revealed by in vivo antisense targeting

Treatment of HCV infection by targeting microRNA

P304

6394-6402

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scholarly article

P356

10.1128/JVI.00016-14

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P433

11

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P478

88

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P50

Dominique C Valla

P577

2014-03-26T00:00:00Z

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P698

24672032

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P921

P932

4093870

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