A mitochondrial ketogenic enzyme regulates its gene expression by association with the nuclear hormone receptor PPARalpha.
about
Host cell factor-1 interacts with and antagonizes transactivation by the cell cycle regulatory factor Miz-1Mitochondrial 3-hydroxy-3-methylglutaryl-CoA synthase: a control enzyme in ketogenesisHuman HMGCS2 regulates mitochondrial fatty acid oxidation and FGF21 expression in HepG2 cell lineRegulation of Ketone Body Metabolism and the Role of PPARαDifferential recruitment of coactivator RIP140 by Ah and estrogen receptors. Absence of a role for LXXLL motifsNuclear localization of L-type glutaminase in mammalian brainPeroxisomal bifunctional enzyme binds and activates the activation function-1 region of the peroxisome proliferator-activated receptor alphaIdentification of palmitoylated mitochondrial proteins using a bio-orthogonal azido-palmitate analogueHMGCS2 enhances invasion and metastasis via direct interaction with PPARα to activate Src signaling in colorectal cancer and oral cancer.Proteomics analysis reveals diabetic kidney as a ketogenic organ in type 2 diabetes.Peroxisome proliferator-activated receptors in atherosclerosis and inflammation--an update.Thioredoxin-mediated negative autoregulation of peroxisome proliferator-activated receptor alpha transcriptional activity.Ketogenesis prevents diet-induced fatty liver injury and hyperglycemiaGenetic-dependency of peroxisomal cell functions - emerging aspects.Keratin 8 absence down-regulates colonocyte HMGCS2 and modulates colonic ketogenesis and energy metabolism.Ketone body metabolism and cardiovascular diseaseThe SIN3 deacetylase complex represses genes encoding mitochondrial proteins: implications for the regulation of energy metabolism.Level of dietary energy and 2,4-thiazolidinedione alter molecular and systemic biomarkers of inflammation and liver function in Holstein cows.Multi-dimensional Roles of Ketone Bodies in Fuel Metabolism, Signaling, and Therapeutics.Palmitoylation of ketogenic enzyme HMGCS2 enhances its interaction with PPARalpha and transcription at the Hmgcs2 PPRE.Fenofibrate Induces Ketone Body Production in Melanoma and Glioblastoma Cells.The mitochondrial fatty acid synthesis (mtFASII) pathway is capable of mediating nuclear-mitochondrial cross talk through the PPAR system of transcriptional activationGlutaminases.Non-canonical functions of enzymes facilitate cross-talk between cell metabolic and regulatory pathways.
P2860
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P2860
A mitochondrial ketogenic enzyme regulates its gene expression by association with the nuclear hormone receptor PPARalpha.
description
1998 nî lūn-bûn
@nan
1998 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
A mitochondrial ketogenic enzy ...... ar hormone receptor PPARalpha.
@ast
A mitochondrial ketogenic enzy ...... ar hormone receptor PPARalpha.
@en
type
label
A mitochondrial ketogenic enzy ...... ar hormone receptor PPARalpha.
@ast
A mitochondrial ketogenic enzy ...... ar hormone receptor PPARalpha.
@en
prefLabel
A mitochondrial ketogenic enzy ...... ar hormone receptor PPARalpha.
@ast
A mitochondrial ketogenic enzy ...... ar hormone receptor PPARalpha.
@en
P2093
P2860
P356
P1433
P1476
A mitochondrial ketogenic enzy ...... ar hormone receptor PPARalpha.
@en
P2093
Cechetto JD
Meertens LM
Rachubinski RA
P2860
P304
P356
10.1093/EMBOJ/17.23.6972
P407
P577
1998-12-01T00:00:00Z