Natural killer cell function and dysfunction in hepatitis C virus infection.
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IFNL4 and IFNL3 associated polymorphisms strongly influence the spontaneous IFN-alpha receptor-1 expression in HCV-infected patients.Activins and Follistatin in Chronic Hepatitis C and Its Treatment with Pegylated-Interferon-α Based Therapy.Immune Evasion Strategies during Chronic Hepatitis B and C Virus InfectionA multifaceted computational report on the variants effect on KIR2DL3 and IFNL3 candidate gene in HCV clearance.NK cells lacking FcεRIγ are associated with reduced liver damage in chronic hepatitis C virus infection.Innate Cell-Mediated Cytotoxic Activity of European Sea Bass Leucocytes Against Nodavirus-Infected Cells: A Functional and RNA-seq Study.
P2860
Natural killer cell function and dysfunction in hepatitis C virus infection.
description
2014 nî lūn-bûn
@nan
2014 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Natural killer cell function and dysfunction in hepatitis C virus infection.
@ast
Natural killer cell function and dysfunction in hepatitis C virus infection.
@en
type
label
Natural killer cell function and dysfunction in hepatitis C virus infection.
@ast
Natural killer cell function and dysfunction in hepatitis C virus infection.
@en
prefLabel
Natural killer cell function and dysfunction in hepatitis C virus infection.
@ast
Natural killer cell function and dysfunction in hepatitis C virus infection.
@en
P2093
P2860
P921
P356
P1476
Natural killer cell function and dysfunction in hepatitis C virus infection.
@en
P2093
Kayla A Holder
Michael D Grant
Rodney S Russell
P2860
P304
P356
10.1155/2014/903764
P407
P5008
P577
2014-06-25T00:00:00Z