Tumor necrosis factor-alpha blockade prevents neutrophil CD18 receptor upregulation and attenuates acute lung injury in porcine sepsis without inhibition of neutrophil oxygen radical generation.
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Adult respiratory distress syndrome - an update.Ron receptor tyrosine kinase negatively regulates TNFalpha production in alveolar macrophages by inhibiting NF-kappaB activity and Adam17 productionRon receptor deficient alveolar myeloid cells exacerbate LPS-induced acute lung injury in the murine lungBacterial pathogens induce abscess formation by CD4(+) T-cell activation via the CD28-B7-2 costimulatory pathwayModulation of the host response in periodontal therapy.Alveolar macrophages are required for protective pulmonary defenses in murine Klebsiella pneumonia: elimination of alveolar macrophages increases neutrophil recruitment but decreases bacterial clearance and survivalThe LPS-induced neutrophil recruitment into rat air pouches is mediated by TNFalpha: likely macrophage origin.Acute lung injury/acute respiratory distress syndrome (ALI/ARDS): the mechanism, present strategies and future perspectives of therapies.Pharmacotherapy of acute lung injury and acute respiratory distress syndromeMolecular mechanisms of endothelial hyperpermeability: implications in inflammation.Biological therapies in the acute respiratory distress syndrome.Emerging drugs for acute lung injury.Differences in allergic inflammatory responses in murine lungs: comparison of PM2.5 and coarse PM collected during the hazy events in a Chinese city.Dietary Fusarium moniliforme culture material induces in vitro tumor necrosis factor-alpha like activity in the sera of swine.Effects of etanercept, a tumour necrosis factor-alpha antagonist, in an experimental model of periodontitis in rats.Tumor necrosis factor-alpha activation of the c-Jun N-terminal kinase pathway in human neutrophils. Integrin involvement in a pathway leading from cytoplasmic tyrosine kinases apoptosis.Modulation of acute lung injury by integrins.Protective role of synthetic sialylated oligosaccharide in sepsis-induced acute lung injury.Inhibition of CD18 or CD11b attenuates acute lung injury after acid instillation in rabbits.Neutrophils in the lung: "the first responders".Differential effects of anti-cytokine treatment on bronchoalveolar hemostasis in endotoxemic chimpanzees.Bronchoalveolar lavage fluid characteristics of early intermediate and late phases of ARDS. Alterations in leukocytes, proteins, PAF and surfactant components.Silica-carrying particulate matter enhances Bjerkandera adusta-induced murine lung eosinophilia.Tumor necrosis factor activity of pancreatic islets.
P2860
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P2860
Tumor necrosis factor-alpha blockade prevents neutrophil CD18 receptor upregulation and attenuates acute lung injury in porcine sepsis without inhibition of neutrophil oxygen radical generation.
description
1993 nî lūn-bûn
@nan
1993 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1993 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
name
Tumor necrosis factor-alpha bl ...... hil oxygen radical generation.
@ast
Tumor necrosis factor-alpha bl ...... hil oxygen radical generation.
@en
type
label
Tumor necrosis factor-alpha bl ...... hil oxygen radical generation.
@ast
Tumor necrosis factor-alpha bl ...... hil oxygen radical generation.
@en
prefLabel
Tumor necrosis factor-alpha bl ...... hil oxygen radical generation.
@ast
Tumor necrosis factor-alpha bl ...... hil oxygen radical generation.
@en
P2093
P2860
P356
P1476
Tumor necrosis factor-alpha bl ...... hil oxygen radical generation.
@en
P2093
Blocher CR
Fowler AA 3rd
Leeper-Woodford SK
Sugerman HJ
Windsor AC
P2860
P304
P356
10.1172/JCI116351
P407
P577
1993-04-01T00:00:00Z