Fidelity of histone gene regulation is obligatory for genome replication and stability. - Wikidata - awgldk - TriplyDB

Fidelity of histone gene regulation is obligatory for genome replication and stability.

Fidelity of histone gene regulation is obligatory for genome replication and stability.

http://www.wikidata.org/entity/Q33899830

about

Cell cycle gene expression networks discovered using systems biology: Significance in carcinogenesis p53 checkpoint ablation exacerbates the phenotype of Hinfp dependent histone H4 deficiency.The HIST1 Locus Escapes Reprogramming in Cloned Bovine Embryos.Regulation of Replication Fork Advance and Stability by Nucleosome Assembly.Promotion of Cell Viability and Histone Gene Expression by the Acetyltransferase Gcn5 and the Protein Phosphatase PP2A in Saccharomyces cerevisiae.Histone availability as a strategy to control gene expression DDB1 and CUL4 associated factor 11 (DCAF11) mediates degradation of Stem-loop binding protein at the end of S phase Intranuclear and higher-order chromatin organization of the major histone gene cluster in breast cancer.Coordinating cell cycle-regulated histone gene expression through assembly and function of the Histone Locus Body.Maternal expression and early induction of histone gene transcription factor Hinfp sustains development in pre-implantation embryos.dCas9-targeted locus-specific protein isolation method identifies histone gene regulators.Chromatin conformation regulates the coordination between DNA replication and transcription.

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P2860

Fidelity of histone gene regulation is obligatory for genome replication and stability.

http://www.wikidata.org/entity/Q33899830

description

2014 nî lūn-bûn

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2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հուլիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

@zh-hk

2014年論文

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2014年論文

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2014年论文

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name

Fidelity of histone gene regulation is obligatory for genome replication and stability.

@ast

Fidelity of histone gene regulation is obligatory for genome replication and stability.

@en

type

label

Fidelity of histone gene regulation is obligatory for genome replication and stability.

@ast

Fidelity of histone gene regulation is obligatory for genome replication and stability.

@en

prefLabel

Fidelity of histone gene regulation is obligatory for genome replication and stability.

@ast

Fidelity of histone gene regulation is obligatory for genome replication and stability.

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Molecular and Cellular Biology

P1476

Fidelity of histone gene regulation is obligatory for genome replication and stability.

@en

P2093

Andre J van Wijnen

http://www.w3.org/2001/XMLSchema#string

Gary S Stein

http://www.w3.org/2001/XMLSchema#string

Jane B Lian

http://www.w3.org/2001/XMLSchema#string

Janet L Stein

http://www.w3.org/2001/XMLSchema#string

Jennifer L Colby

http://www.w3.org/2001/XMLSchema#string

Prachi N Ghule

http://www.w3.org/2001/XMLSchema#string

Ricardo Medina

http://www.w3.org/2001/XMLSchema#string

Rong-Lin Xie

http://www.w3.org/2001/XMLSchema#string

Stephen N Jones

http://www.w3.org/2001/XMLSchema#string

P2860

Cell cycle-regulated phosphorylation of p220(NPAT) by cyclin E/Cdk2 in Cajal bodies promotes histone gene transcription

Identification of HiNF-P, a key activator of cell cycle-controlled histone H4 genes at the onset of S phase

HiNF-P directly links the cyclin E/CDK2/p220NPAT pathway to histone H4 gene regulation at the G1/S phase cell cycle transition.

The histone gene cell cycle regulator HiNF-P is a unique zinc finger transcription factor with a novel conserved auxiliary DNA-binding motif

A biomarker that identifies senescent human cells in culture and in aging skin in vivo

NPAT links cyclin E-Cdk2 to the regulation of replication-dependent histone gene transcription

Expression of NPAT, a novel substrate of cyclin E-CDK2, promotes S-phase entry

The cyclin E/Cdk2 substrate p220(NPAT) is required for S-phase entry, histone gene expression, and Cajal body maintenance in human somatic cells

NPAT expression is regulated by E2F and is essential for cell cycle progression

Cdk2 knockout mice are viable

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2650-2659

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scholarly article

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10.1128/MCB.01567-13

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14

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34

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2014-07-01T00:00:00Z

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262074202

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24797072

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4097655

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