Deep sequencing reveals direct targets of gammaherpesvirus-induced mRNA decay and suggests that multiple mechanisms govern cellular transcript escape.
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A Tale of Two RNAs during Viral Infection: How Viruses Antagonize mRNAs and Small Non-Coding RNAs in The Host CellShutoff of Host Gene Expression in Influenza A Virus and Herpesviruses: Similar Mechanisms and Common ThemesNext-Generation Sequencing in the Understanding of Kaposi's Sarcoma-Associated Herpesvirus (KSHV) BiologyA ribonucleoprotein complex protects the interleukin-6 mRNA from degradation by distinct herpesviral endonucleasesCoordinated destruction of cellular messages in translation complexes by the gammaherpesvirus host shutoff factor and the mammalian exonuclease Xrn1.Transcriptome-Wide Cleavage Site Mapping on Cellular mRNAs Reveals Features Underlying Sequence-Specific Cleavage by the Viral Ribonuclease SOX.Selective Degradation of Host RNA Polymerase II Transcripts by Influenza A Virus PA-X Host Shutoff Protein.The "Bridge" in the Epstein-Barr virus alkaline exonuclease protein BGLF5 contributes to shutoff activity during productive infection.Integrated systems biology analysis of KSHV latent infection reveals viral induction and reliance on peroxisome mediated lipid metabolism.Epstein-barr virus infected gastric adenocarcinoma expresses latent and lytic viral transcripts and has a distinct human gene expression profileMurine Gammaherpesvirus 68 LANA and SOX Homologs Counteract ATM-Driven p53 Activity during Lytic Viral ReplicationComplexities of gammaherpesvirus transcription revealed by microarrays and RNAseq.Gammaherpesviral gene expression and virion composition are broadly controlled by accelerated mRNA degradation.Application of next-generation sequencing technologies in virology.Recent advances in the study of Kaposi's sarcoma-associated herpesvirus replication and pathogenesis.An RNA element in human interleukin 6 confers escape from degradation by the gammaherpesvirus SOX protein.Nuclease escape elements protect messenger RNA against cleavage by multiple viral endonucleases.Kaposi's sarcoma-associated herpesvirus G-protein-coupled receptor prevents AU-rich-element-mediated mRNA decay.KSHV mRNA accumulation in nuclear foci is influenced by viral DNA replication and the viral noncoding PAN RNA.
P2860
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P2860
Deep sequencing reveals direct targets of gammaherpesvirus-induced mRNA decay and suggests that multiple mechanisms govern cellular transcript escape.
description
2011 nî lūn-bûn
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2011 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Deep sequencing reveals direct ...... rn cellular transcript escape.
@ast
Deep sequencing reveals direct ...... rn cellular transcript escape.
@en
type
label
Deep sequencing reveals direct ...... rn cellular transcript escape.
@ast
Deep sequencing reveals direct ...... rn cellular transcript escape.
@en
prefLabel
Deep sequencing reveals direct ...... rn cellular transcript escape.
@ast
Deep sequencing reveals direct ...... rn cellular transcript escape.
@en
P2860
P1433
P1476
Deep sequencing reveals direct ...... rn cellular transcript escape.
@en
P2093
Britt A Glaunsinger
Karen Clyde
P2860
P304
P356
10.1371/JOURNAL.PONE.0019655
P407
P577
2011-05-09T00:00:00Z