Corticotropin releasing factor-induced amygdala gamma-aminobutyric Acid release plays a key role in alcohol dependence. - Wikidata - awgldk - TriplyDB

Corticotropin releasing factor-induced amygdala gamma-aminobutyric Acid release plays a key role in alcohol dependence.

Corticotropin releasing factor-induced amygdala gamma-aminobutyric Acid release plays a key role in alcohol dependence.

http://www.wikidata.org/entity/Q33904853

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Chronic alcohol neuroadaptation and stress contribute to susceptibility for alcohol craving and relapse Adolescent Alcohol Exposure: Burden of Epigenetic Reprogramming, Synaptic Remodeling, and Adult Psychopathology Effects of anabolic-androgens on brain reward function Effects of alcohol dependence and withdrawal on stress responsiveness and alcohol consumption Neuropeptide modulation of central amygdala neuroplasticity is a key mediator of alcohol dependence.Behavioral, biological, and chemical perspectives on targeting CRF(1) receptor antagonists to treat alcoholism.Neuropeptide Y (NPY) in the extended amygdala is recruited during the transition to alcohol dependence The Sturm und Drang of anabolic steroid use: angst, anxiety, and aggression An Update on CRF Mechanisms Underlying Alcohol Use Disorders and Dependence Preclinical studies of alcohol binge drinking A Phase 2, Double-Blind, Placebo-Controlled Randomized Trial Assessing the Efficacy of ABT-436, a Novel V1b Receptor Antagonist, for Alcohol Dependence.Mad men, women and steroid cocktails: a review of the impact of sex and other factors on anabolic androgenic steroids effects on affective behaviors Effects of chronic ethanol exposure on neuronal function in the prefrontal cortex and extended amygdala.The central amygdala as an integrative hub for anxiety and alcohol use disorders CRF-amplified neuronal TLR4/MCP-1 signaling regulates alcohol self-administration.Synaptic effects induced by alcohol.Polymorphism in the corticotropin-releasing factor receptor 1 (CRF1-R) gene plays a role in shaping the high anxious phenotype of Marchigian Sardinian alcohol-preferring (msP) rats.Addictions Neuroclinical Assessment: A reverse translational approach.Predator odor stress alters corticotropin-releasing factor-1 receptor (CRF1R)-dependent behaviors in rats.Central effects of ethanol interact with endogenous mu-opioid activity to control isolation-induced analgesia in maternally separated infant rats.Corticotropin-releasing factor modulation of forebrain GABAergic transmission has a pivotal role in the expression of anabolic steroid-induced anxiety in the female mouse.Nicotine dependence produces hyperalgesia: role of corticotropin-releasing factor-1 receptors (CRF1Rs) in the central amygdala (CeA).A stress steroid triggers anxiety via increased expression of α4βδ GABAA receptors in methamphetamine dependence.Chronic anabolic androgenic steroid exposure alters corticotropin releasing factor expression and anxiety-like behaviors in the female mouse The Buzz about anabolic androgenic steroids: electrophysiological effects in excitable tissues.Role of corticotropin-releasing factor in drug addiction: potential for pharmacological intervention.Effects of stress on alcohol drinking: a review of animal studies.SEB-3, a CRF receptor-like GPCR, regulates locomotor activity states, stress responses and ethanol tolerance in Caenorhabditis elegans VTA CRF neurons mediate the aversive effects of nicotine withdrawal and promote intake escalation.Defining the role of corticotropin releasing factor binding protein in alcohol consumption.Operant alcohol self-administration in dependent rats: focus on the vapor model Further characterization of the GlyT-1 inhibitor Org25935: anti-alcohol, neurobehavioral, and gene expression effects.Neurobiological mechanisms of addiction: focus on corticotropin-releasing factor.Urocortin-1 within the centrally-projecting Edinger-Westphal nucleus is critical for ethanol preference.Adolescent binge drinking leads to changes in alcohol drinking, anxiety, and amygdalar corticotropin releasing factor cells in adulthood in male rats.Amygdala 14-3-3ζ as a novel modulator of escalating alcohol intake in mice.Corticotropin releasing factor: a key role in the neurobiology of addiction Ethanol-enhanced GABA release: a focus on G protein-coupled receptors.Preventive Effects of Forced Exercise against Alcohol-induced Physical Dependency and Reduction of Pain Perception Threshold Ethanol-induced alterations of amino acids measured by in vivo microdialysis in rats: a meta-analysis.

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Corticotropin releasing factor-induced amygdala gamma-aminobutyric Acid release plays a key role in alcohol dependence.

http://www.wikidata.org/entity/Q33904853

description

2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Corticotropin releasing factor ...... ey role in alcohol dependence.

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Corticotropin releasing factor ...... ey role in alcohol dependence.

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Corticotropin releasing factor ...... ey role in alcohol dependence.

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Corticotropin releasing factor ...... ey role in alcohol dependence.

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J.BIOPSYCH.2009.11.007

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Biological Psychiatry

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Corticotropin releasing factor ...... key role in alcohol dependence

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David G Stouffer

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Eric P Zorrilla

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George F Koob

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George R Siggins

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Loren H Parsons

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Marisa Roberto

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Maureen T Cruz

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Michal Bajo

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Paul Schweitzer

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Samuel G Madamba

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P2860

A role for brain stress systems in addiction.

The sigma-receptor antagonist BD-1063 decreases ethanol intake and reinforcement in animal models of excessive drinking

Conceptual framework for the etiology of alcoholism: a "kindling"/stress hypothesis.

Corticotropin-releasing factor 1 antagonists selectively reduce ethanol self-administration in ethanol-dependent rats

Dependence-induced increases in ethanol self-administration in mice are blocked by the CRF1 receptor antagonist antalarmin and by CRF1 receptor knockout.

The intrinsic organization of the central extended amygdala.

Excessive ethanol drinking following a history of dependence: animal model of allostasis.

The role of stress in alcohol use, alcoholism treatment, and relapse.

The neurobiology of stress: from serendipity to clinical relevance.

Ethanol augments GABAergic transmission in the central amygdala via CRF1 receptors.

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831-839

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scholarly article

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10.1016/J.BIOPSYCH.2009.11.007

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9

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67

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Nicholas W Gilpin

Valentina Sabino

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2010-01-08T00:00:00Z

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40897853

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20060104

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2883449

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