Indo-1 fluorescence signals elicited by membrane depolarization in enzymatically isolated mouse skeletal muscle fibers.
about
Junctophilin 1 and 2 proteins interact with the L-type Ca2+ channel dihydropyridine receptors (DHPRs) in skeletal muscleSuperoxide flashes in mouse skeletal muscle are produced by discrete arrays of active mitochondria operating coherentlyKeratitis-ichthyosis-deafness syndrome-associated Cx26 mutants produce nonfunctional gap junctions but hyperactive hemichannels when co-expressed with wild type Cx43.Alternating bipolar field stimulation identifies muscle fibers with defective excitability but maintained local Ca(2+) signals and contraction.Altered inactivation of Ca2+ current and Ca2+ release in mouse muscle fibers deficient in the DHP receptor gamma1 subunitDisrupted membrane structure and intracellular Ca²⁺ signaling in adult skeletal muscle with acute knockdown of Bin1.Depression of voltage-activated Ca2+ release in skeletal muscle by activation of a voltage-sensing phosphatase.Rapid Ca2+ flux through the transverse tubular membrane, activated by individual action potentials in mammalian skeletal muscle.Measurements of intracellular Mg2+ concentration in mouse skeletal muscle fibers with the fluorescent indicator mag-indo-1Contribution of ryanodine receptor type 3 to Ca(2+) sparks in embryonic mouse skeletal musclePatch-clamp recording of charge movement, Ca2+ current, and Ca2+ transients in adult skeletal muscle fibersSustained release of calcium elicited by membrane depolarization in ryanodine-injected mouse skeletal muscle fibersIntramembrane charge movement and L-type calcium current in skeletal muscle fibers isolated from control and mdx mice.In vivo expression of G-protein beta1gamma2 dimer in adult mouse skeletal muscle alters L-type calcium current and excitation-contraction coupling.Ca2+ release in muscle fibers expressing R4892W and G4896V type 1 ryanodine receptor disease mutantsTransient loss of voltage control of Ca2+ release in the presence of maurocalcine in skeletal muscle.Muscle aging is associated with compromised Ca2+ spark signaling and segregated intracellular Ca2+ releaseEffects of dantrolene on steps of excitation-contraction coupling in mammalian skeletal muscle fibers.Type 1 inositol (1,4,5)-trisphosphate receptor activates ryanodine receptor 1 to mediate calcium spark signaling in adult mammalian skeletal muscleConfocal imaging of transmembrane voltage by SEER of di-8-ANEPPSRespective contribution of mitochondrial superoxide and pH to mitochondria-targeted circularly permuted yellow fluorescent protein (mt-cpYFP) flash activity.Deficiency of MIP/MTMR14 phosphatase induces a muscle disorder by disrupting Ca(2+) homeostasis.Electrically silent divalent cation entries in resting and active voltage-controlled muscle fibersT-tubule disorganization and defective excitation-contraction coupling in muscle fibers lacking myotubularin lipid phosphataseVoltage-activated elementary calcium release events in isolated mouse skeletal muscle fibers.Phosphatidylinositol 3-kinase inhibition restores Ca2+ release defects and prolongs survival in myotubularin-deficient mice.Targeted gene inactivation reveals a functional role of calpain-1 in platelet spreading.Advances in two photon scanning and scanless microscopy technologies for functional neural circuit imaging.Impaired calcium signaling in muscle fibers from intercostal and foot skeletal muscle in a cigarette smoke-induced mouse model of COPD.Voltage-controlled Ca2+ release and entry flux in isolated adult muscle fibres of the mouse.Design of a disulfide-less, pharmacologically inert, and chemically competent analog of maurocalcine for the efficient transport of impermeant compounds into cells.Control of intracellular calcium in the presence of nitric oxide donors in isolated skeletal muscle fibres from mouse.Nitric oxide synthase inhibition affects sarcoplasmic reticulum Ca2+ release in skeletal muscle fibres from mouse.Spontaneous and voltage-activated Ca2+ release in adult mouse skeletal muscle fibres expressing the type 3 ryanodine receptor.Impaired excitation-contraction coupling in muscle fibres from the dynamin2R465W mouse model of centronuclear myopathy.Defects in Ca2+ release associated with local expression of pathological ryanodine receptors in mouse muscle fibres.Activation of Ca2+-activated K+ channels by an increase in intracellular Ca2+ induced by depolarization of mouse skeletal muscle fibres.Evolution and modulation of intracellular calcium release during long-lasting, depleting depolarization in mouse muscle.Phosphoinositide substrates of myotubularin affect voltage-activated Ca²⁺ release in skeletal muscle.
P2860
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P2860
Indo-1 fluorescence signals elicited by membrane depolarization in enzymatically isolated mouse skeletal muscle fibers.
description
1997 nî lūn-bûn
@nan
1997 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
name
Indo-1 fluorescence signals el ...... mouse skeletal muscle fibers.
@ast
Indo-1 fluorescence signals el ...... mouse skeletal muscle fibers.
@en
type
label
Indo-1 fluorescence signals el ...... mouse skeletal muscle fibers.
@ast
Indo-1 fluorescence signals el ...... mouse skeletal muscle fibers.
@en
prefLabel
Indo-1 fluorescence signals el ...... mouse skeletal muscle fibers.
@ast
Indo-1 fluorescence signals el ...... mouse skeletal muscle fibers.
@en
P2860
P1433
P1476
Indo-1 fluorescence signals el ...... mouse skeletal muscle fibers.
@en
P2093
V Jacquemond
P2860
P304
P356
10.1016/S0006-3495(97)78124-4
P407
P577
1997-08-01T00:00:00Z